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You searched for +publisher:"Loma Linda University" +contributor:("Ducsay, Charles"). Showing records 1 – 10 of 10 total matches.

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Loma Linda University

1. Vargas, Vladimir Enrique. Long-Term Hypoxia Regulates Cortisol Biosynthesis in the Ovine Fetal Adrenal Cortex.

Degree: PhD, Basic Sciences, 2010, Loma Linda University

 Previous studies demonstrated enhanced fetal adrenal cortisol biosynthesis following exposure to long-term hypoxia (LTH). The studies presented here were designed to determine the mechanism(s) regulating… (more)

Subjects/Keywords: Medical Physiology; Endocrinology; Endocrine glands;

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APA (6th Edition):

Vargas, V. E. (2010). Long-Term Hypoxia Regulates Cortisol Biosynthesis in the Ovine Fetal Adrenal Cortex. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/11

Chicago Manual of Style (16th Edition):

Vargas, Vladimir Enrique. “Long-Term Hypoxia Regulates Cortisol Biosynthesis in the Ovine Fetal Adrenal Cortex.” 2010. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/11.

MLA Handbook (7th Edition):

Vargas, Vladimir Enrique. “Long-Term Hypoxia Regulates Cortisol Biosynthesis in the Ovine Fetal Adrenal Cortex.” 2010. Web. 19 Aug 2019.

Vancouver:

Vargas VE. Long-Term Hypoxia Regulates Cortisol Biosynthesis in the Ovine Fetal Adrenal Cortex. [Internet] [Doctoral dissertation]. Loma Linda University; 2010. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/11.

Council of Science Editors:

Vargas VE. Long-Term Hypoxia Regulates Cortisol Biosynthesis in the Ovine Fetal Adrenal Cortex. [Doctoral Dissertation]. Loma Linda University; 2010. Available from: https://scholarsrepository.llu.edu/etd/11


Loma Linda University

2. Patterson, Andrew James Grant. Chronic Hypoxia Induces Epigenetic Modifications in the Fetal Rat Heart.

Degree: PhD, Basic Sciences, 2011, Loma Linda University

 Heart disease remains the leading cause of death worldwide. As a result of studies done by Barker and associates, our awareness of the significance of… (more)

Subjects/Keywords: Medical Physiology; Cardiovascular Diseases; Fetal Hypoxia  – physiopathology; Fetal Heart  – physiopathology; Fetal Development; Cardiovascular System  – physiopathology; Protein Kinase C; Endothelium, Vascular; Heart disease; Chronic Hypoxia; Fetal rat hearts; Gene expression patterns; Cardioprotective gene; Vascular Endothelial Growth Factor

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APA (6th Edition):

Patterson, A. J. G. (2011). Chronic Hypoxia Induces Epigenetic Modifications in the Fetal Rat Heart. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/54

Chicago Manual of Style (16th Edition):

Patterson, Andrew James Grant. “Chronic Hypoxia Induces Epigenetic Modifications in the Fetal Rat Heart.” 2011. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/54.

MLA Handbook (7th Edition):

Patterson, Andrew James Grant. “Chronic Hypoxia Induces Epigenetic Modifications in the Fetal Rat Heart.” 2011. Web. 19 Aug 2019.

Vancouver:

Patterson AJG. Chronic Hypoxia Induces Epigenetic Modifications in the Fetal Rat Heart. [Internet] [Doctoral dissertation]. Loma Linda University; 2011. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/54.

Council of Science Editors:

Patterson AJG. Chronic Hypoxia Induces Epigenetic Modifications in the Fetal Rat Heart. [Doctoral Dissertation]. Loma Linda University; 2011. Available from: https://scholarsrepository.llu.edu/etd/54


Loma Linda University

3. Tong, Wenni. Effect of Maternal Hypoxia on the Cerebral and Cardiac Tissue Remodeling.

Degree: PhD, Basic Sciences, 2012, Loma Linda University

 Large epidemiological studies and animal studies have indicated a clear association of adverse intrauterine environment with an increased incidence of low birth weight, perinatal mortality,… (more)

Subjects/Keywords: Medical Pharmacology; Cardiovascular Disease; Hypoxia-Ischemia, Brain; Fetal Hypoxia; Fetal Heart; Fetal Development; Birth weight; Perinatal mortality; Adverse Intrauterine Environment; MMP; TIMP; Matrix Metalloproteinase; Tissue Inhibitors of Metalloproteinase

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APA (6th Edition):

Tong, W. (2012). Effect of Maternal Hypoxia on the Cerebral and Cardiac Tissue Remodeling. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/101

Chicago Manual of Style (16th Edition):

Tong, Wenni. “Effect of Maternal Hypoxia on the Cerebral and Cardiac Tissue Remodeling.” 2012. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/101.

MLA Handbook (7th Edition):

Tong, Wenni. “Effect of Maternal Hypoxia on the Cerebral and Cardiac Tissue Remodeling.” 2012. Web. 19 Aug 2019.

Vancouver:

Tong W. Effect of Maternal Hypoxia on the Cerebral and Cardiac Tissue Remodeling. [Internet] [Doctoral dissertation]. Loma Linda University; 2012. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/101.

Council of Science Editors:

Tong W. Effect of Maternal Hypoxia on the Cerebral and Cardiac Tissue Remodeling. [Doctoral Dissertation]. Loma Linda University; 2012. Available from: https://scholarsrepository.llu.edu/etd/101


Loma Linda University

4. Adeoye, Olayemi Olufikayo. VEGF and Sympathetic Perivascular Nerves Contribute to Hypoxic Remodeling of Ovine Cranial Arteries.

Degree: PhD, Basic Sciences, 2013, Loma Linda University

 Chronic hypoxia complicates many pregnancies and can result in postnatal pathologies that include compromised fetal cardiovascular structure and function. Mechanisms involved remain unclear. Because hypoxia… (more)

Subjects/Keywords: Medical Physiology; Fetal hypoxia; Fetal development; Endothelium, vascular; Cardiovascular system - physiopathology; Fetal heart - physiopathology; Hypoxia-Ischemia, Brain - physiopathology; Ovine cranial arteries; Sympathetic perivascular nerves; VEGF; Hypoxia; Vascular Endothelial Growth factor; Chronic hypoxia; Hypoxic fetal vascular remodeling

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APA (6th Edition):

Adeoye, O. O. (2013). VEGF and Sympathetic Perivascular Nerves Contribute to Hypoxic Remodeling of Ovine Cranial Arteries. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/110

Chicago Manual of Style (16th Edition):

Adeoye, Olayemi Olufikayo. “VEGF and Sympathetic Perivascular Nerves Contribute to Hypoxic Remodeling of Ovine Cranial Arteries.” 2013. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/110.

MLA Handbook (7th Edition):

Adeoye, Olayemi Olufikayo. “VEGF and Sympathetic Perivascular Nerves Contribute to Hypoxic Remodeling of Ovine Cranial Arteries.” 2013. Web. 19 Aug 2019.

Vancouver:

Adeoye OO. VEGF and Sympathetic Perivascular Nerves Contribute to Hypoxic Remodeling of Ovine Cranial Arteries. [Internet] [Doctoral dissertation]. Loma Linda University; 2013. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/110.

Council of Science Editors:

Adeoye OO. VEGF and Sympathetic Perivascular Nerves Contribute to Hypoxic Remodeling of Ovine Cranial Arteries. [Doctoral Dissertation]. Loma Linda University; 2013. Available from: https://scholarsrepository.llu.edu/etd/110


Loma Linda University

5. Paradis, Alexandra. Perinatal Hypoxia Exposure on the Developing Heart: The Role of Endothelin-1.

Degree: PhD, Basic Sciences, 2015, Loma Linda University

  Heart disease is the leading cause of death worldwide. Numerous epidemiological and animal studies have indicated that an adverse intrauterine environment is associated with… (more)

Subjects/Keywords: Pharmacy and Pharmaceutical Sciences; Fetal Anoxia, Perinatology, Child Development, Cardiovascular Disease; Heart Disease; Cardiomyocyte Endowment; Perinatal Hypoxia; Perinatal Development; Endothelin-1

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APA (6th Edition):

Paradis, A. (2015). Perinatal Hypoxia Exposure on the Developing Heart: The Role of Endothelin-1. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/173

Chicago Manual of Style (16th Edition):

Paradis, Alexandra. “Perinatal Hypoxia Exposure on the Developing Heart: The Role of Endothelin-1.” 2015. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/173.

MLA Handbook (7th Edition):

Paradis, Alexandra. “Perinatal Hypoxia Exposure on the Developing Heart: The Role of Endothelin-1.” 2015. Web. 19 Aug 2019.

Vancouver:

Paradis A. Perinatal Hypoxia Exposure on the Developing Heart: The Role of Endothelin-1. [Internet] [Doctoral dissertation]. Loma Linda University; 2015. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/173.

Council of Science Editors:

Paradis A. Perinatal Hypoxia Exposure on the Developing Heart: The Role of Endothelin-1. [Doctoral Dissertation]. Loma Linda University; 2015. Available from: https://scholarsrepository.llu.edu/etd/173


Loma Linda University

6. Zhu, Ronghui. Expression and Function of Ca2+-Activated K+ Channels in Uterine Arteries.

Degree: PhD, Basic Sciences, 2013, Loma Linda University

  Chronic hypoxia during pregnancy is one of the most common insults to the maternal cardiovascular system and fetal development, and is associated with increased… (more)

Subjects/Keywords: Biochemistry, Biophysics, and Structural Biology; Medical Biochemistry; Medicine and Health Sciences; Uterine Artery; Anoxia; Vasodilation; Potassium Channels - Calcium-Activated; Small-Conductance Calcium-Activated Potassium Channel Alpha Subunits; Reactive Oxygen Species; Estradiol; Progesterone; Vascular Resistance; Pregnancy Complications; Fetal Hypoxia; Fetal Development; Fetal Intrauterine Growth Restriction; Uterine Vascular Adaptation; Basal Uterine Vascular Tone

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APA (6th Edition):

Zhu, R. (2013). Expression and Function of Ca2+-Activated K+ Channels in Uterine Arteries. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/299

Chicago Manual of Style (16th Edition):

Zhu, Ronghui. “Expression and Function of Ca2+-Activated K+ Channels in Uterine Arteries.” 2013. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/299.

MLA Handbook (7th Edition):

Zhu, Ronghui. “Expression and Function of Ca2+-Activated K+ Channels in Uterine Arteries.” 2013. Web. 19 Aug 2019.

Vancouver:

Zhu R. Expression and Function of Ca2+-Activated K+ Channels in Uterine Arteries. [Internet] [Doctoral dissertation]. Loma Linda University; 2013. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/299.

Council of Science Editors:

Zhu R. Expression and Function of Ca2+-Activated K+ Channels in Uterine Arteries. [Doctoral Dissertation]. Loma Linda University; 2013. Available from: https://scholarsrepository.llu.edu/etd/299


Loma Linda University

7. Gonzalez-Rodriguez, Pablo J. Effect of Maternal Hypoxia on Fetal Development: Programming of HIE Phenotype in Neonatal Rat Brain.

Degree: PhD, Basic Sciences, 2014, Loma Linda University

  Developmental programming of health and disease correlates the adverse intrauterine environment and the risk of disease later in life as cardiovascular disease, metabolic diseases,… (more)

Subjects/Keywords: Biochemistry; Life Sciences; Medicine and Health Sciences; Brain - Growth & Development; Fetal Hypoxia; Hypoxia-Ischemia, Brain; Gene Expression Regulation - Developmental; DNA Methylation; Receptors - Glucocorticoid; Dexamethasone;

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APA (6th Edition):

Gonzalez-Rodriguez, P. J. (2014). Effect of Maternal Hypoxia on Fetal Development: Programming of HIE Phenotype in Neonatal Rat Brain. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/204

Chicago Manual of Style (16th Edition):

Gonzalez-Rodriguez, Pablo J. “Effect of Maternal Hypoxia on Fetal Development: Programming of HIE Phenotype in Neonatal Rat Brain.” 2014. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/204.

MLA Handbook (7th Edition):

Gonzalez-Rodriguez, Pablo J. “Effect of Maternal Hypoxia on Fetal Development: Programming of HIE Phenotype in Neonatal Rat Brain.” 2014. Web. 19 Aug 2019.

Vancouver:

Gonzalez-Rodriguez PJ. Effect of Maternal Hypoxia on Fetal Development: Programming of HIE Phenotype in Neonatal Rat Brain. [Internet] [Doctoral dissertation]. Loma Linda University; 2014. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/204.

Council of Science Editors:

Gonzalez-Rodriguez PJ. Effect of Maternal Hypoxia on Fetal Development: Programming of HIE Phenotype in Neonatal Rat Brain. [Doctoral Dissertation]. Loma Linda University; 2014. Available from: https://scholarsrepository.llu.edu/etd/204


Loma Linda University

8. Newby, Elizabeth Anne. Long-Term Hypoxia Alters Ovine Fetal Adrenal eNOS and Cortisol Biosynthesis.

Degree: PhD, Basic Sciences, 2015, Loma Linda University

  Maintaining normal levels of cortisol in response to chronic stress, while retaining the ability to respond to acute stress, is important for ensuring normal… (more)

Subjects/Keywords: Medical Physiology; Medical Sciences; Medicine and Health Sciences; Fetal Hypoxia; Adrenal Cortex  – physiopathology; Sheep  – Metabolism; Hydrocortisone  – Biosynthesis; Cortisol Biosynthesis; Chronic Stress; Fetal Growth; Fetal Hypothalamo-pituitary-adrenal Axis; Endothelial Nitric Oxide Synthase

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APA (6th Edition):

Newby, E. A. (2015). Long-Term Hypoxia Alters Ovine Fetal Adrenal eNOS and Cortisol Biosynthesis. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/360

Chicago Manual of Style (16th Edition):

Newby, Elizabeth Anne. “Long-Term Hypoxia Alters Ovine Fetal Adrenal eNOS and Cortisol Biosynthesis.” 2015. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/360.

MLA Handbook (7th Edition):

Newby, Elizabeth Anne. “Long-Term Hypoxia Alters Ovine Fetal Adrenal eNOS and Cortisol Biosynthesis.” 2015. Web. 19 Aug 2019.

Vancouver:

Newby EA. Long-Term Hypoxia Alters Ovine Fetal Adrenal eNOS and Cortisol Biosynthesis. [Internet] [Doctoral dissertation]. Loma Linda University; 2015. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/360.

Council of Science Editors:

Newby EA. Long-Term Hypoxia Alters Ovine Fetal Adrenal eNOS and Cortisol Biosynthesis. [Doctoral Dissertation]. Loma Linda University; 2015. Available from: https://scholarsrepository.llu.edu/etd/360


Loma Linda University

9. Gay, Maresha. Effects of Neonatal Dexamethasone on the Maturation and Endowment of Cardiomyocytes.

Degree: PhD, Basic Sciences, 2016, Loma Linda University

  Each year 1 in 9 infants are born preterm in the United States. Preterm infants have a host of complications associated with prematurity, including… (more)

Subjects/Keywords: Medical Pharmacology; Medical Sciences; Medicine and Health Sciences; Dexamethasone; Neonatalogy; Respiratory Distress Syndrome; Heart  – Growth and Development; Glucocorticoids; Organ maturation; Surfactant Production; Cardiomyocytes; Terminal differentiation; Epigenetic Modifications

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APA (6th Edition):

Gay, M. (2016). Effects of Neonatal Dexamethasone on the Maturation and Endowment of Cardiomyocytes. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/373

Chicago Manual of Style (16th Edition):

Gay, Maresha. “Effects of Neonatal Dexamethasone on the Maturation and Endowment of Cardiomyocytes.” 2016. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/373.

MLA Handbook (7th Edition):

Gay, Maresha. “Effects of Neonatal Dexamethasone on the Maturation and Endowment of Cardiomyocytes.” 2016. Web. 19 Aug 2019.

Vancouver:

Gay M. Effects of Neonatal Dexamethasone on the Maturation and Endowment of Cardiomyocytes. [Internet] [Doctoral dissertation]. Loma Linda University; 2016. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/373.

Council of Science Editors:

Gay M. Effects of Neonatal Dexamethasone on the Maturation and Endowment of Cardiomyocytes. [Doctoral Dissertation]. Loma Linda University; 2016. Available from: https://scholarsrepository.llu.edu/etd/373


Loma Linda University

10. Li, Yong. Perinatal Nicotine Exposure and Programming of HIE Sensitive Phenotype in Neonatal Rat Brains.

Degree: PhD, Basic Sciences, 2013, Loma Linda University

 Large epidemiological and animal studies have revealed a clear association of adverse intrauterine environment with the increased risk of metabolic, cardiovascular and neurological diseases. Maternal… (more)

Subjects/Keywords: Medical Pharmacology; Cardiovascular Diseases; Neurological Diseases; Metabolic Diseases; Fetal Development; Nicotine;

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APA (6th Edition):

Li, Y. (2013). Perinatal Nicotine Exposure and Programming of HIE Sensitive Phenotype in Neonatal Rat Brains. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/134

Chicago Manual of Style (16th Edition):

Li, Yong. “Perinatal Nicotine Exposure and Programming of HIE Sensitive Phenotype in Neonatal Rat Brains.” 2013. Doctoral Dissertation, Loma Linda University. Accessed August 19, 2019. https://scholarsrepository.llu.edu/etd/134.

MLA Handbook (7th Edition):

Li, Yong. “Perinatal Nicotine Exposure and Programming of HIE Sensitive Phenotype in Neonatal Rat Brains.” 2013. Web. 19 Aug 2019.

Vancouver:

Li Y. Perinatal Nicotine Exposure and Programming of HIE Sensitive Phenotype in Neonatal Rat Brains. [Internet] [Doctoral dissertation]. Loma Linda University; 2013. [cited 2019 Aug 19]. Available from: https://scholarsrepository.llu.edu/etd/134.

Council of Science Editors:

Li Y. Perinatal Nicotine Exposure and Programming of HIE Sensitive Phenotype in Neonatal Rat Brains. [Doctoral Dissertation]. Loma Linda University; 2013. Available from: https://scholarsrepository.llu.edu/etd/134

.