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You searched for +publisher:"IUPUI" +contributor:("Harrington, Maureen A."). Showing records 1 – 30 of 35 total matches.

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IUPUI

1. Hinkle, David T. CORRELATING IRINOTECAN AND CAPECITABINE TREATMENT FOR COLORECTAL CANCER TO GENE EXPRESSION, POLYMORPHISMS, AND CLINICAL OUTCOMES.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Colorectal cancer is the third most common type of cancer and the third most common cause of cancer-related mortality. There… (more)

Subjects/Keywords: Irinotecan, CPT-11, Capecitabine, DPD, B-GUS, TS, TP, TOPO I, UGT1A1*28, colorectal cancer, SN-38; Colon (Anatomy)  – Cancer  – Adjuvant treatment; Rectum  – Cancer  – Adjuvant treatment; Prodrugs; Gene expression

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hinkle, D. T. (2011). CORRELATING IRINOTECAN AND CAPECITABINE TREATMENT FOR COLORECTAL CANCER TO GENE EXPRESSION, POLYMORPHISMS, AND CLINICAL OUTCOMES. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hinkle, David T. “CORRELATING IRINOTECAN AND CAPECITABINE TREATMENT FOR COLORECTAL CANCER TO GENE EXPRESSION, POLYMORPHISMS, AND CLINICAL OUTCOMES.” 2011. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hinkle, David T. “CORRELATING IRINOTECAN AND CAPECITABINE TREATMENT FOR COLORECTAL CANCER TO GENE EXPRESSION, POLYMORPHISMS, AND CLINICAL OUTCOMES.” 2011. Web. 18 Apr 2021.

Vancouver:

Hinkle DT. CORRELATING IRINOTECAN AND CAPECITABINE TREATMENT FOR COLORECTAL CANCER TO GENE EXPRESSION, POLYMORPHISMS, AND CLINICAL OUTCOMES. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hinkle DT. CORRELATING IRINOTECAN AND CAPECITABINE TREATMENT FOR COLORECTAL CANCER TO GENE EXPRESSION, POLYMORPHISMS, AND CLINICAL OUTCOMES. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

2. Cogill, Steven B. PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

SIMPL is a transcriptional co-activator that alters the activity of transcription factor, NF-κB. In response to pathogens, cytokines such as… (more)

Subjects/Keywords: SIMPL; Transcriptional Co-Activator; NF-kB; NF-kappa B (DNA-binding protein); Transcription factors; Cytokines; Immunoglobulins; Immunofluorescence

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APA (6th Edition):

Cogill, S. B. (2011). PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cogill, Steven B. “PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1.” 2011. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cogill, Steven B. “PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1.” 2011. Web. 18 Apr 2021.

Vancouver:

Cogill SB. PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cogill SB. PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

3. Farrington, Daphne L. Estrogen receptor involvement in the response of human keratinocytes to ultraviolet B irradiation.

Degree: 2014, IUPUI

 The signaling mechanisms involved in UVB-induced skin cancer are complex and although the scope of this work is inherently limited in focus, the findings may… (more)

Subjects/Keywords: UVB; Estrogen receptor; Keratinocytes; Skin  – Cancer; Ultraviolet radiation; Solar radiation; Estrogen  – Receptors; Selective estrogen receptor modulators; Cells  – Growth; Cells  – Aging; Cells  – Death; Apoptosis; Keratinocytes

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APA (6th Edition):

Farrington, D. L. (2014). Estrogen receptor involvement in the response of human keratinocytes to ultraviolet B irradiation. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6241

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farrington, Daphne L. “Estrogen receptor involvement in the response of human keratinocytes to ultraviolet B irradiation.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/6241.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farrington, Daphne L. “Estrogen receptor involvement in the response of human keratinocytes to ultraviolet B irradiation.” 2014. Web. 18 Apr 2021.

Vancouver:

Farrington DL. Estrogen receptor involvement in the response of human keratinocytes to ultraviolet B irradiation. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/6241.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farrington DL. Estrogen receptor involvement in the response of human keratinocytes to ultraviolet B irradiation. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6241

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

4. Anuradha, Valiya Kambrath. Testing the reliability and selectivity of different bone-cell-specific Cre- expressing mouse models for studying bone cell metabolism.

Degree: 2015, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The Cre/loxP system is a tool for targeted recombination of DNA. For applying Cre recombinase-mediated genome modifications, there is a… (more)

Subjects/Keywords: DMP1-Cre; Ctsk-Cre; Bones  – Growth; Bones  – Growth  – Molecular aspects; Genetic recombination  – Research; Recombinant DNA; Gene targeting; Osteocytes; Bone cells; Osteoblasts

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APA (6th Edition):

Anuradha, V. K. (2015). Testing the reliability and selectivity of different bone-cell-specific Cre- expressing mouse models for studying bone cell metabolism. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/7942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anuradha, Valiya Kambrath. “Testing the reliability and selectivity of different bone-cell-specific Cre- expressing mouse models for studying bone cell metabolism.” 2015. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/7942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anuradha, Valiya Kambrath. “Testing the reliability and selectivity of different bone-cell-specific Cre- expressing mouse models for studying bone cell metabolism.” 2015. Web. 18 Apr 2021.

Vancouver:

Anuradha VK. Testing the reliability and selectivity of different bone-cell-specific Cre- expressing mouse models for studying bone cell metabolism. [Internet] [Thesis]. IUPUI; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/7942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anuradha VK. Testing the reliability and selectivity of different bone-cell-specific Cre- expressing mouse models for studying bone cell metabolism. [Thesis]. IUPUI; 2015. Available from: http://hdl.handle.net/1805/7942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Qadadri, Brahim. Human papillomavirus type distribution in cervical cancer in Indiana and Botswana.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

In this study we compared the distribution of HPV types in cervical cancer specimens from women living in either Indiana… (more)

Subjects/Keywords: Papillomavirus diseases  – Research  – Epidemiology; Cervix uteri  – Cancer  – Research  – Indiana  – Statistics; Cervix uteri  – Cancer  – Research  – Botswana  – Statistics; Cervix uteri  – Cancer  – Early detection; Cervix uteri  – Cancer  – Prevention; Papillomaviruses  – Pathogenicity; Papillomavirus vaccines  – Research; Gene amplification  – Research; AIDS (Disease)  – Diagnosis; AIDS (Disease)  – Genetic aspects

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APA (6th Edition):

Qadadri, B. (2014). Human papillomavirus type distribution in cervical cancer in Indiana and Botswana. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Qadadri, Brahim. “Human papillomavirus type distribution in cervical cancer in Indiana and Botswana.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/5223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Qadadri, Brahim. “Human papillomavirus type distribution in cervical cancer in Indiana and Botswana.” 2014. Web. 18 Apr 2021.

Vancouver:

Qadadri B. Human papillomavirus type distribution in cervical cancer in Indiana and Botswana. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/5223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Qadadri B. Human papillomavirus type distribution in cervical cancer in Indiana and Botswana. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

6. Pochareddy, Sirisha. Transcriptional regulation of the human alcohol dehydrogenases and alcoholism.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Alcohol dehydrogenase (ADH) genes encode proteins that metabolize ethanol to acetaldehyde. Humans have seven ADH genes in a cluster. The… (more)

Subjects/Keywords: ADH, alcoholism, variations, microarray, exon arrays, differential gene expression, alternative splicing; Alcohol dehydrogenase  – Regulation; Gene expression; Alcoholism  – Research

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APA (6th Edition):

Pochareddy, S. (2011). Transcriptional regulation of the human alcohol dehydrogenases and alcoholism. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pochareddy, Sirisha. “Transcriptional regulation of the human alcohol dehydrogenases and alcoholism.” 2011. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pochareddy, Sirisha. “Transcriptional regulation of the human alcohol dehydrogenases and alcoholism.” 2011. Web. 18 Apr 2021.

Vancouver:

Pochareddy S. Transcriptional regulation of the human alcohol dehydrogenases and alcoholism. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pochareddy S. Transcriptional regulation of the human alcohol dehydrogenases and alcoholism. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

7. Kinzfogl, John M. Neurexophilin1 suppresses the proliferation of hematopoietic progenitor cells.

Degree: 2012, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Neurexin I alpha (NRXN1α) and Dystroglycan (DAG1) are membrane receptors which serve as mutual ligands in the neuronal system. Neurexophilins… (more)

Subjects/Keywords: Neurexin I alpha; Neurexophilin; Dystroglycan; Hematopoiesis; Hematopoietic niche; Hematopoiesis; Hematopoietic stem cells; Proteins

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APA (6th Edition):

Kinzfogl, J. M. (2012). Neurexophilin1 suppresses the proliferation of hematopoietic progenitor cells. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kinzfogl, John M. “Neurexophilin1 suppresses the proliferation of hematopoietic progenitor cells.” 2012. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kinzfogl, John M. “Neurexophilin1 suppresses the proliferation of hematopoietic progenitor cells.” 2012. Web. 18 Apr 2021.

Vancouver:

Kinzfogl JM. Neurexophilin1 suppresses the proliferation of hematopoietic progenitor cells. [Internet] [Thesis]. IUPUI; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kinzfogl JM. Neurexophilin1 suppresses the proliferation of hematopoietic progenitor cells. [Thesis]. IUPUI; 2012. Available from: http://hdl.handle.net/1805/2737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

8. Cao, Liyun. Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis.

Degree: 2012, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Ovarian cancer (OC) is a lethal disease due to metastasis and chemoresistance. Our laboratory previously reported that tissue transglutaminase (TG2)… (more)

Subjects/Keywords: tissue transglutaminase, TGF-beta, ovarian cancer; Ovaries  – Cancer  – Molecular aspects  – Research  – Methodology; Ovaries  – Cancer  – Genetic aspects  – Research  – Methodology; Metastasis; Tumor markers  – Research  – Methodology; Peritoneum  – Cancer; Transglutaminases

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APA (6th Edition):

Cao, L. (2012). Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cao, Liyun. “Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis.” 2012. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cao, Liyun. “Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis.” 2012. Web. 18 Apr 2021.

Vancouver:

Cao L. Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis. [Internet] [Thesis]. IUPUI; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cao L. Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis. [Thesis]. IUPUI; 2012. Available from: http://hdl.handle.net/1805/2817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

9. Adler, Jacob J. The inhibition of mammary epithelial cell growth by the long isoform of Angiomotin.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Mammary ductal epithelial cell growth is controlled by microenvironmental signals in serum under both normal physiological settings and during breast… (more)

Subjects/Keywords: Angiomotin, Breast Cancer, Ubiquitin, Nedd4, YAP, Hippo; Mammary glands  – Molecular aspects  – Research; Breast  – Cancer  – Pathophysiology; Breast  – Cancer  – Genetic aspects  – Research; Protein-tyrosine kinase  – Research; Apoptosis  – Molecular aspects  – Research; Ligases  – Research; Epithelial cells  – Growth  – Inhibitors; Cellular control mechanisms  – Research  – Evaluation; Ubiquitin  – Research; Proteins  – Research; Cancer  – Molecular aspects  – Research; Cellular signal transduction  – Research; Cancer cells  – Growth  – Regulation; Pathology, Molecular; Transcription factors  – Research; Cell migration  – Inhibitors  – Research; Proto-oncogenes  – Research; Cell proliferation  – Molecular aspects  – Research

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APA (6th Edition):

Adler, J. J. (2014). The inhibition of mammary epithelial cell growth by the long isoform of Angiomotin. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/4600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adler, Jacob J. “The inhibition of mammary epithelial cell growth by the long isoform of Angiomotin.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/4600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adler, Jacob J. “The inhibition of mammary epithelial cell growth by the long isoform of Angiomotin.” 2014. Web. 18 Apr 2021.

Vancouver:

Adler JJ. The inhibition of mammary epithelial cell growth by the long isoform of Angiomotin. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/4600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adler JJ. The inhibition of mammary epithelial cell growth by the long isoform of Angiomotin. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/4600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

10. Gendron, Jaimie M. The role of PTEN in human cancer.

Degree: 2015, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Phosphatase and tensin homolog, PTEN, is a key tumor suppressor. Mutation of PTEN is associated with both sporadic cancers and… (more)

Subjects/Keywords: PTEN; Gain of function; Cell cycle; Protein-tyrosine phosphatase  – Mutation.; Phosphatases  – Research; Cells  – Growth

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APA (6th Edition):

Gendron, J. M. (2015). The role of PTEN in human cancer. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/7934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gendron, Jaimie M. “The role of PTEN in human cancer.” 2015. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/7934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gendron, Jaimie M. “The role of PTEN in human cancer.” 2015. Web. 18 Apr 2021.

Vancouver:

Gendron JM. The role of PTEN in human cancer. [Internet] [Thesis]. IUPUI; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/7934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gendron JM. The role of PTEN in human cancer. [Thesis]. IUPUI; 2015. Available from: http://hdl.handle.net/1805/7934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

11. Stritesky, Gretta L. THE DEVELOPMENT AND COMMITMENT OF T HELPER SUBSETS.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

T helper cells play a crucial role in providing protection against a wide variety of pathogens. The differentiation and effector… (more)

Subjects/Keywords: Differentiation; Th2; STAT3; Th2 cells; Phenotype; Cytokines; T cells  – Differentiation

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APA (6th Edition):

Stritesky, G. L. (2011). THE DEVELOPMENT AND COMMITMENT OF T HELPER SUBSETS. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stritesky, Gretta L. “THE DEVELOPMENT AND COMMITMENT OF T HELPER SUBSETS.” 2011. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stritesky, Gretta L. “THE DEVELOPMENT AND COMMITMENT OF T HELPER SUBSETS.” 2011. Web. 18 Apr 2021.

Vancouver:

Stritesky GL. THE DEVELOPMENT AND COMMITMENT OF T HELPER SUBSETS. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stritesky GL. THE DEVELOPMENT AND COMMITMENT OF T HELPER SUBSETS. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

12. Lin, Lin. Gentamicin Induced Intracellular Toxicity in Saccharomyces cerevisiae.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

At the present time, gentamicin is used in the treatment of both Gram-negative and Gram-positive bacterial infections. However, the poorly… (more)

Subjects/Keywords: gentamicin, Arf protein trafficking pathway; Gentamicin; Bacterial diseases  – Treatment; Nephrotoxicology

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APA (6th Edition):

Lin, L. (2011). Gentamicin Induced Intracellular Toxicity in Saccharomyces cerevisiae. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Lin. “Gentamicin Induced Intracellular Toxicity in Saccharomyces cerevisiae.” 2011. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Lin. “Gentamicin Induced Intracellular Toxicity in Saccharomyces cerevisiae.” 2011. Web. 18 Apr 2021.

Vancouver:

Lin L. Gentamicin Induced Intracellular Toxicity in Saccharomyces cerevisiae. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin L. Gentamicin Induced Intracellular Toxicity in Saccharomyces cerevisiae. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

13. Staser, Karl W. Erk1 and Erk2 in hematopoiesis, mast cell function, and the management of Nf1-associated leukemia and tumors.

Degree: 2012, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Neurofibromatosis type 1 is a genetic disease that results from either heritable or spontaneous autosomal dominant mutations in the NF1… (more)

Subjects/Keywords: NF1, Erk1, Erk2, mast cells, hematopoiesis, neurofibroma, leukemia; Neurofibromatosis; Nervous system  – Diseases; Medical genetics; Mast cells; Hematopoiesis; Neurofibroma; Leukemia; Cytokines  – Research  – Methodology; Tumors  – Genetic aspects

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Staser, K. W. (2012). Erk1 and Erk2 in hematopoiesis, mast cell function, and the management of Nf1-associated leukemia and tumors. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Staser, Karl W. “Erk1 and Erk2 in hematopoiesis, mast cell function, and the management of Nf1-associated leukemia and tumors.” 2012. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Staser, Karl W. “Erk1 and Erk2 in hematopoiesis, mast cell function, and the management of Nf1-associated leukemia and tumors.” 2012. Web. 18 Apr 2021.

Vancouver:

Staser KW. Erk1 and Erk2 in hematopoiesis, mast cell function, and the management of Nf1-associated leukemia and tumors. [Internet] [Thesis]. IUPUI; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2892.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Staser KW. Erk1 and Erk2 in hematopoiesis, mast cell function, and the management of Nf1-associated leukemia and tumors. [Thesis]. IUPUI; 2012. Available from: http://hdl.handle.net/1805/2892

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

14. Nguyen, Hoa Bich. The tumor suppressing roles of tissue structure in cervical cancer development.

Degree: 2013, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Cervical cancer is caused by the persistent infection of human papilloma virus (HPV) in the cervix epithelium. Although effective preventative… (more)

Subjects/Keywords: Cervical cancer; Yap; LKB1; HPV-E6; NF2; tissue structure; Papillomaviruses  – Research; Cervix uteri  – Cancer; Tumor suppressor proteins; Cells  – Growth  – Regulation; Cell proliferation  – Research; Papillomavirus vaccines; Phosphorylation; Transcription factors; Dysplasia; Keratinocytes  – Differentiation; Cancer cells  – Research

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APA (6th Edition):

Nguyen, H. B. (2013). The tumor suppressing roles of tissue structure in cervical cancer development. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Hoa Bich. “The tumor suppressing roles of tissue structure in cervical cancer development.” 2013. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/3627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Hoa Bich. “The tumor suppressing roles of tissue structure in cervical cancer development.” 2013. Web. 18 Apr 2021.

Vancouver:

Nguyen HB. The tumor suppressing roles of tissue structure in cervical cancer development. [Internet] [Thesis]. IUPUI; 2013. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/3627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen HB. The tumor suppressing roles of tissue structure in cervical cancer development. [Thesis]. IUPUI; 2013. Available from: http://hdl.handle.net/1805/3627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

15. Park, Soyoung. In vivo analysis of human LHX3 enhancer regulation.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The LHX3 transcription factor is essential for pituitary gland and nervous system development in mammals. In humans, mutations in the… (more)

Subjects/Keywords: LHX3; Enhancer; Pituitary gland  – Pathophysiology; Transcription factors  – Pathophysiology; Genetic regulation  – Research; Adenohypophysis; Pituitary gland  – Diseases  – Research; Mutation (Biology); Zinc proteins  – Metabolism; Human molecular genetics  – Research; Gonadotropin; Cell differentiation; Molecular association  – Research  – Analysis; Biochemistry  – Technique

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APA (6th Edition):

Park, S. (2014). In vivo analysis of human LHX3 enhancer regulation. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Park, Soyoung. “In vivo analysis of human LHX3 enhancer regulation.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/3807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Park, Soyoung. “In vivo analysis of human LHX3 enhancer regulation.” 2014. Web. 18 Apr 2021.

Vancouver:

Park S. In vivo analysis of human LHX3 enhancer regulation. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/3807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Park S. In vivo analysis of human LHX3 enhancer regulation. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/3807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

16. Glosson, Nicole L. Development and stability of IL-17-secreting T cells.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

IL-17-producing T cells are critical to the development of pathogen and tumor immunity, but also contribute to the pathology of… (more)

Subjects/Keywords: T cell, IL-17, Th17, Tc17, STAT4, Allergic Airway Disease; Autoimmune diseases  – Research  – Evaluation  – Analysis; Anti-inflammatory agents; Inflammation  – Immunological aspects; Respiratory allergy; T cells  – Immunology; Interleukins  – Immunology; Asthma  – Pathophysiology; Th1 cells  – Research; Th2 cells  – Research; Cellular immunity; Immune response  – Regulation; Cellular control mechanisms

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APA (6th Edition):

Glosson, N. L. (2014). Development and stability of IL-17-secreting T cells. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Glosson, Nicole L. “Development and stability of IL-17-secreting T cells.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/5902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Glosson, Nicole L. “Development and stability of IL-17-secreting T cells.” 2014. Web. 18 Apr 2021.

Vancouver:

Glosson NL. Development and stability of IL-17-secreting T cells. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/5902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Glosson NL. Development and stability of IL-17-secreting T cells. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

17. Templin, Andrew Thomas. Mechanisms of translational regulation in the pancreatic β cell stress response.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The islet beta cell is unique in its ability to synthesize and secrete insulin for use in the body. A… (more)

Subjects/Keywords: Diabetes, Islet, ER stress, Pdx1; Diabetes  – Research  – Analysis  – Evaluation  – Methodology; Insulin  – Mechanism of action  – Research; Pancreatic beta cells  – Research; Endoplasmic reticulum  – Pathophysiology; Stress (Physiology); Cellular signal transduction; Islands of Langerhans  – Research; Small interfering RNA  – Research; Protein folding  – Research; Proteins  – Conformation; Apoptosis; Pancreas  – Growth  – Molecular aspects; Transcription factors; Cellular control mechanisms

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APA (6th Edition):

Templin, A. T. (2014). Mechanisms of translational regulation in the pancreatic β cell stress response. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Templin, Andrew Thomas. “Mechanisms of translational regulation in the pancreatic β cell stress response.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/6162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Templin, Andrew Thomas. “Mechanisms of translational regulation in the pancreatic β cell stress response.” 2014. Web. 18 Apr 2021.

Vancouver:

Templin AT. Mechanisms of translational regulation in the pancreatic β cell stress response. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/6162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Templin AT. Mechanisms of translational regulation in the pancreatic β cell stress response. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

18. Garyali, Punitee. Lafora Disease: Mechanisms Involved in Pathogenesis.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Lafora disease is a neurodegenerative disorder caused by mutations in either the EPM2A or the EPM2B gene that encode a… (more)

Subjects/Keywords: Lafora Disease, Laforin, Malin, Ubiquitination, diGly proteomics, Protein Quality Control; Myoclonus  – Molecular aspects  – Research  – Methodology; Myoclonus  – Genetic aspects  – Research  – Methodology; Glycogen  – Metabolism; Ubiquitin  – Metabolism; Ligases  – Research; Autophagic vacuoles; Gene targeting; Nervous system  – Degeneration  – Pathophysiology; Genetic transcription  – Regulation; Phosphorylation; Proteomics  – Regulation; Glycopeptides; Proteins  – Metabolism; Homeostasis; Mice as laboratory animals

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Garyali, P. (2014). Lafora Disease: Mechanisms Involved in Pathogenesis. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Garyali, Punitee. “Lafora Disease: Mechanisms Involved in Pathogenesis.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/5903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Garyali, Punitee. “Lafora Disease: Mechanisms Involved in Pathogenesis.” 2014. Web. 18 Apr 2021.

Vancouver:

Garyali P. Lafora Disease: Mechanisms Involved in Pathogenesis. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/5903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Garyali P. Lafora Disease: Mechanisms Involved in Pathogenesis. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

19. Morgan, Cynthia A. Identification and characterization of small-molecule inhibitors of aldehyde dehydrogenase 1A1.

Degree: 2015, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The human genome encodes 19 members of the aldehyde dehydrogenase (ALDH) superfamily, critical enzymes involved in the metabolism of aldehyde… (more)

Subjects/Keywords: Enzyme kinetics; Small molecule inhibitors; X-ray crystallography; Aldehyde dehydrogenase; Human genome; Enzyme kinetics; Oxidation; Molecule-molecule collisions; Enzyme inhibitors; X-ray crystallography - Technique

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APA (6th Edition):

Morgan, C. A. (2015). Identification and characterization of small-molecule inhibitors of aldehyde dehydrogenase 1A1. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morgan, Cynthia A. “Identification and characterization of small-molecule inhibitors of aldehyde dehydrogenase 1A1.” 2015. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/6593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morgan, Cynthia A. “Identification and characterization of small-molecule inhibitors of aldehyde dehydrogenase 1A1.” 2015. Web. 18 Apr 2021.

Vancouver:

Morgan CA. Identification and characterization of small-molecule inhibitors of aldehyde dehydrogenase 1A1. [Internet] [Thesis]. IUPUI; 2015. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/6593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morgan CA. Identification and characterization of small-molecule inhibitors of aldehyde dehydrogenase 1A1. [Thesis]. IUPUI; 2015. Available from: http://hdl.handle.net/1805/6593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Malik, Raleigh Elizabeth. The role of DNA methylation in regulating LHX3 gene expression.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

LIM homeodomain 3 (LHX3) is an important regulator of pituitary and nervous system development. To date, twelve LHX3 gene mutations… (more)

Subjects/Keywords: LHX3; DNA Methylation; DNA  – Methylation  – Research  – Methodology; Developmental neurophysiology; Histone deacetylase; Chromatin  – Research; Nucleotide sequence; Pituitary hormones  – Diseases  – Etiology; Genetic regulation  – Research; Adenohypophysis; Acetylation; Transcription factors  – Research  – Analysis; Gene targeting  – Methodology  – Technique

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APA (6th Edition):

Malik, R. E. (2014). The role of DNA methylation in regulating LHX3 gene expression. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/4039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Malik, Raleigh Elizabeth. “The role of DNA methylation in regulating LHX3 gene expression.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/4039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Malik, Raleigh Elizabeth. “The role of DNA methylation in regulating LHX3 gene expression.” 2014. Web. 18 Apr 2021.

Vancouver:

Malik RE. The role of DNA methylation in regulating LHX3 gene expression. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/4039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Malik RE. The role of DNA methylation in regulating LHX3 gene expression. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/4039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Speth, Jennifer M. Defining the mechanism of prostaglandin E₂-enhanced hematopoietic stem and progenitor cell homing.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Hematopoietic stem cell (HSC) transplantation is a lifesaving therapy for a number of hematological disorders. However, to be effective, transplanted… (more)

Subjects/Keywords: Prostaglandin, Homing, HIF1-alpha, hematopoietic stem cell; Hematopoietic stem cells  – Transplantation; Prostaglandins E  – Research  – Methodology; Regeneration (Biology); Bone marrow  – Transplantation; Transcription factors  – Research  – Methodology; Immune response  – Regulation; Hematology  – Research; Gene expression; Sodium nitroferricyanide; Imaging systems in medicine; Cells  – Motility; Cell migration  – Research; Cellular signal transduction

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APA (6th Edition):

Speth, J. M. (2014). Defining the mechanism of prostaglandin E₂-enhanced hematopoietic stem and progenitor cell homing. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/4206

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Speth, Jennifer M. “Defining the mechanism of prostaglandin E₂-enhanced hematopoietic stem and progenitor cell homing.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/4206.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Speth, Jennifer M. “Defining the mechanism of prostaglandin E₂-enhanced hematopoietic stem and progenitor cell homing.” 2014. Web. 18 Apr 2021.

Vancouver:

Speth JM. Defining the mechanism of prostaglandin E₂-enhanced hematopoietic stem and progenitor cell homing. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/4206.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Speth JM. Defining the mechanism of prostaglandin E₂-enhanced hematopoietic stem and progenitor cell homing. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/4206

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Ha, Sung Pil. CD4+ T cell mediated tumor immunity following transplantation of TRP-1 TCR gene modified hematopoietic stem cells.

Degree: 2013, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Immunotherapy for cancer has held much promise as a potent modality of cancer treatment. The ability to selectively destroy diseased… (more)

Subjects/Keywords: Melanoma; Lentivirus; Hematopoietic Stem Cells; Immunology; Tumor Immunology; CD4 T Cells; Gene Therapy; Tumor Antigens; T Cell Receptor; Bone Marrow Transplantation; Tumor Immunity; Tyrosine Relate Protein; Melanocyte Differentiation Antigen; Lentiviral Vectors; Melanoma  – Research; Lentiviruses; Hematopoietic stem cells  – Transplantation; Cancer  – Immunology  – Genetic aspects; Tumors  – Immunological aspects; T cells  – Receptors; Cancer  – Gene therapy; Tumor antigens; Bone marrow  – Transplantation; Tyrosine; MSH (Hormone); Genetic vectors  – Research

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ha, S. P. (2013). CD4+ T cell mediated tumor immunity following transplantation of TRP-1 TCR gene modified hematopoietic stem cells. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ha, Sung Pil. “CD4+ T cell mediated tumor immunity following transplantation of TRP-1 TCR gene modified hematopoietic stem cells.” 2013. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/3739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ha, Sung Pil. “CD4+ T cell mediated tumor immunity following transplantation of TRP-1 TCR gene modified hematopoietic stem cells.” 2013. Web. 18 Apr 2021.

Vancouver:

Ha SP. CD4+ T cell mediated tumor immunity following transplantation of TRP-1 TCR gene modified hematopoietic stem cells. [Internet] [Thesis]. IUPUI; 2013. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/3739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ha SP. CD4+ T cell mediated tumor immunity following transplantation of TRP-1 TCR gene modified hematopoietic stem cells. [Thesis]. IUPUI; 2013. Available from: http://hdl.handle.net/1805/3739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Fan, Li. mTOR regulates Aurora A via enhancing protein stability.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Mammalian target of rapamycin (mTOR) is a key regulator of protein synthesis. Dysregulation of mTOR signaling occurs in many human… (more)

Subjects/Keywords: mTOR, Aurora A, protein stability; Proteins  – Stability; Proteins  – Conformation; Proteins  – Research  – Methodology; Cell cycle  – Regulation; Mitosis  – Regulation; Phosphoprotein phosphatases  – Inhibitors; Serine proteinases  – Inhibitors; Rho GTPases; Neuroglia; Enzymes  – Regulation; G proteins; Cell division; Proteins  – Metabolism  – Regulation; Proteins  – Synthesis; Rapamycin; Transcription factors  – Research; Cellular signal transduction; Messenger RNA  – Research

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APA (6th Edition):

Fan, L. (2014). mTOR regulates Aurora A via enhancing protein stability. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/4648

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fan, Li. “mTOR regulates Aurora A via enhancing protein stability.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/4648.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fan, Li. “mTOR regulates Aurora A via enhancing protein stability.” 2014. Web. 18 Apr 2021.

Vancouver:

Fan L. mTOR regulates Aurora A via enhancing protein stability. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/4648.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fan L. mTOR regulates Aurora A via enhancing protein stability. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/4648

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Lajiness, Jacquelyn D. Shp2 deletion in post-migratory neural crest cells results in impaired cardiac sympathetic innervation.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Autonomic innervation of the heart begins in utero and continues during the neonatal phase of life. A balance between the… (more)

Subjects/Keywords: Cardiac development; Sympathetic innervation; Shp2; pERK; Bradycardia; Heart  – Innervation  – Research  – Analysis; Heart  – Diseases; Autonomic nervous system  – Physiology; Heart  – Growth; Sympathetic nervous system; Protein-tyrosine phosphatase; Heart conduction system; Heart  – Diseases  – Treatment; Protein kinases; Developmental neurobiology  – Research; Heart rate monitoring; Neural crest  – Research; Cell differentiation  – Molecular aspects; Bradycardia  – Etiology; Mice  – Diseases  – Molecular aspects

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APA (6th Edition):

Lajiness, J. D. (2014). Shp2 deletion in post-migratory neural crest cells results in impaired cardiac sympathetic innervation. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lajiness, Jacquelyn D. “Shp2 deletion in post-migratory neural crest cells results in impaired cardiac sympathetic innervation.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/5495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lajiness, Jacquelyn D. “Shp2 deletion in post-migratory neural crest cells results in impaired cardiac sympathetic innervation.” 2014. Web. 18 Apr 2021.

Vancouver:

Lajiness JD. Shp2 deletion in post-migratory neural crest cells results in impaired cardiac sympathetic innervation. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/5495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lajiness JD. Shp2 deletion in post-migratory neural crest cells results in impaired cardiac sympathetic innervation. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Hollister, Kristin N. Elucidating the role of BCL6 in helper T cell activation, proliferation, and differentiation.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The transcriptional repressor BCL6 has been shown to be essential for the differentiation of germinal center (GC) B cells and… (more)

Subjects/Keywords: T cells; follicular T helper cells; BCL6; germinal center; HIV vaccine; T cells  – Regulation  – Research  – Analysis; T cells  – Differentiation; Th2 cells  – Regulation  – Research  – Analysis; Th1 cells  – Regulation  – Research  – Analysis; Cell transformation  – Regulation; Repressors, Genetic  – Research; HIV (Viruses)  – Vaccination; AIDS vaccines  – Research; Inflammation  – Immunological aspects; B cells  – Research  – Analysis; Germinal centers; Immune system; Autoimmune diseases; Mice  – Diseases  – Molecular aspects; Animal models in research

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hollister, K. N. (2014). Elucidating the role of BCL6 in helper T cell activation, proliferation, and differentiation. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hollister, Kristin N. “Elucidating the role of BCL6 in helper T cell activation, proliferation, and differentiation.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/5930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hollister, Kristin N. “Elucidating the role of BCL6 in helper T cell activation, proliferation, and differentiation.” 2014. Web. 18 Apr 2021.

Vancouver:

Hollister KN. Elucidating the role of BCL6 in helper T cell activation, proliferation, and differentiation. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/5930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hollister KN. Elucidating the role of BCL6 in helper T cell activation, proliferation, and differentiation. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Woods, Derek S. The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

In mammalian cells DNA double strand breaks (DSBs) are highly variable with respect to sequence and structure all of which… (more)

Subjects/Keywords: DNA Repair,; NHEJ; DNA-PK; Ku70/80; Ku80 Carboxy Terminus; DNA  – Research  – Methodology; DNA-protein interactions; Protein-protein interactions; DNA  – Synthesis; Enzymes  – Analysis; Protein kinases  – Research  – Evaluation; DNA repair; DNA damage; DNA-binding proteins; Cellular control mechanisms; Nucleotide sequence; Mutagenesis; Biochemical genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Woods, D. S. (2014). The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/4665

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Woods, Derek S. “The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors.” 2014. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/4665.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Woods, Derek S. “The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors.” 2014. Web. 18 Apr 2021.

Vancouver:

Woods DS. The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/4665.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Woods DS. The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/4665

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Head, Sally K. The Effect of Omega-3 Fatty Acids on Airway Inflammation, Hyperpnea-Induced Bronchoconstriction, and Airway Smooth Muscle Contractility in Asthma.

Degree: 2012, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Asthma, a chronic inflammatory disease of the airways, affects nearly 25 million Americans. The vast majority of these patients suffer… (more)

Subjects/Keywords: Asthma; Bronchoconstriction; Fish Oil; Asthma  – Treatment  – Research; Fish oils  – Therapeutic use; Smooth muscle  – Contraction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Head, S. K. (2012). The Effect of Omega-3 Fatty Acids on Airway Inflammation, Hyperpnea-Induced Bronchoconstriction, and Airway Smooth Muscle Contractility in Asthma. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Head, Sally K. “The Effect of Omega-3 Fatty Acids on Airway Inflammation, Hyperpnea-Induced Bronchoconstriction, and Airway Smooth Muscle Contractility in Asthma.” 2012. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Head, Sally K. “The Effect of Omega-3 Fatty Acids on Airway Inflammation, Hyperpnea-Induced Bronchoconstriction, and Airway Smooth Muscle Contractility in Asthma.” 2012. Web. 18 Apr 2021.

Vancouver:

Head SK. The Effect of Omega-3 Fatty Acids on Airway Inflammation, Hyperpnea-Induced Bronchoconstriction, and Airway Smooth Muscle Contractility in Asthma. [Internet] [Thesis]. IUPUI; 2012. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Head SK. The Effect of Omega-3 Fatty Acids on Airway Inflammation, Hyperpnea-Induced Bronchoconstriction, and Airway Smooth Muscle Contractility in Asthma. [Thesis]. IUPUI; 2012. Available from: http://hdl.handle.net/1805/2752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

28. Benson, Eric Ashley. Loss of SIMPL increases TNFα sensitivity during hematopoiesis.

Degree: 2008, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The innate and adaptive immune responses are critical for host survival. The TNFα/NF-κB signaling pathway is a major regulator of… (more)

Subjects/Keywords: TNFalpha; Hematopoiesis; p65/p50; NF-kappaB; hematopoietic progenitor; CFU-GM; SIMPL; colony assay; hematopoietic stem cell; SIMPL; Hematopoiesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Benson, E. A. (2008). Loss of SIMPL increases TNFα sensitivity during hematopoiesis. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/1851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Benson, Eric Ashley. “Loss of SIMPL increases TNFα sensitivity during hematopoiesis.” 2008. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/1851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Benson, Eric Ashley. “Loss of SIMPL increases TNFα sensitivity during hematopoiesis.” 2008. Web. 18 Apr 2021.

Vancouver:

Benson EA. Loss of SIMPL increases TNFα sensitivity during hematopoiesis. [Internet] [Thesis]. IUPUI; 2008. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/1851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Benson EA. Loss of SIMPL increases TNFα sensitivity during hematopoiesis. [Thesis]. IUPUI; 2008. Available from: http://hdl.handle.net/1805/1851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

29. McNeil, Sara M. ELUCIDATING THE ROLE OF REDOX EFFECTS AND THE KU80 C-TERMINAL REGION IN THE REGULATION OF THE HUMAN DNA REPAIR PROTEIN KU.

Degree: 2010, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

DNA double strand breaks (DSB) are among the most lethal forms of DNA damage and can occur as a result… (more)

Subjects/Keywords: DSB repair; DNA-PKcs regulation; Ku; NHEJ; DNA repair; DNA-binding proteins; Oxidation-reduction reaction; Biochemical genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McNeil, S. M. (2010). ELUCIDATING THE ROLE OF REDOX EFFECTS AND THE KU80 C-TERMINAL REGION IN THE REGULATION OF THE HUMAN DNA REPAIR PROTEIN KU. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McNeil, Sara M. “ELUCIDATING THE ROLE OF REDOX EFFECTS AND THE KU80 C-TERMINAL REGION IN THE REGULATION OF THE HUMAN DNA REPAIR PROTEIN KU.” 2010. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/2202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McNeil, Sara M. “ELUCIDATING THE ROLE OF REDOX EFFECTS AND THE KU80 C-TERMINAL REGION IN THE REGULATION OF THE HUMAN DNA REPAIR PROTEIN KU.” 2010. Web. 18 Apr 2021.

Vancouver:

McNeil SM. ELUCIDATING THE ROLE OF REDOX EFFECTS AND THE KU80 C-TERMINAL REGION IN THE REGULATION OF THE HUMAN DNA REPAIR PROTEIN KU. [Internet] [Thesis]. IUPUI; 2010. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/2202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McNeil SM. ELUCIDATING THE ROLE OF REDOX EFFECTS AND THE KU80 C-TERMINAL REGION IN THE REGULATION OF THE HUMAN DNA REPAIR PROTEIN KU. [Thesis]. IUPUI; 2010. Available from: http://hdl.handle.net/1805/2202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

30. Jimenez, Juan Antonio. ANTI-TUMOR AND RADIO-SENSITIZING PROPERTIES OF AD-IU2, A PROSTATE-SPECIFIC REPLICATION-COMPETENT ADENOVIRUS ARMED WITH TRAIL.

Degree: 2008, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

In this thesis, I investigated the preclinical utility and antitumor efficacy of TRAIL delivered by Ad-IU2, a prostate-specific replication-competent adenovirus… (more)

Subjects/Keywords: TRAIL; Prostate Cancer; PSES; Gene Therapy; Ad-IU2; Adenovirus; Adenoviruses; Prostate  – Cancer; Cancer  – Gene therapy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jimenez, J. A. (2008). ANTI-TUMOR AND RADIO-SENSITIZING PROPERTIES OF AD-IU2, A PROSTATE-SPECIFIC REPLICATION-COMPETENT ADENOVIRUS ARMED WITH TRAIL. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/1871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jimenez, Juan Antonio. “ANTI-TUMOR AND RADIO-SENSITIZING PROPERTIES OF AD-IU2, A PROSTATE-SPECIFIC REPLICATION-COMPETENT ADENOVIRUS ARMED WITH TRAIL.” 2008. Thesis, IUPUI. Accessed April 18, 2021. http://hdl.handle.net/1805/1871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jimenez, Juan Antonio. “ANTI-TUMOR AND RADIO-SENSITIZING PROPERTIES OF AD-IU2, A PROSTATE-SPECIFIC REPLICATION-COMPETENT ADENOVIRUS ARMED WITH TRAIL.” 2008. Web. 18 Apr 2021.

Vancouver:

Jimenez JA. ANTI-TUMOR AND RADIO-SENSITIZING PROPERTIES OF AD-IU2, A PROSTATE-SPECIFIC REPLICATION-COMPETENT ADENOVIRUS ARMED WITH TRAIL. [Internet] [Thesis]. IUPUI; 2008. [cited 2021 Apr 18]. Available from: http://hdl.handle.net/1805/1871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jimenez JA. ANTI-TUMOR AND RADIO-SENSITIZING PROPERTIES OF AD-IU2, A PROSTATE-SPECIFIC REPLICATION-COMPETENT ADENOVIRUS ARMED WITH TRAIL. [Thesis]. IUPUI; 2008. Available from: http://hdl.handle.net/1805/1871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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