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You searched for +publisher:"Georgia Tech" +contributor:("LaPlaca, Michelle C."). Showing records 1 – 4 of 4 total matches.

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Georgia Tech

1. Shoemaker, James Thomas. Development of an in vitro model of neuroinflammation for studying secondary injury mechanisms in traumatic brain injury.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2015, Georgia Tech

 A novel cell culture system was designed to serve as a model of neuroinflammation. Neurons, astrocytes, and microglia derived from embryonic and perinatal rat cortical… (more)

Subjects/Keywords: Cell culture; Three-dimensional; Microglia; Traumatic brain injury; Inflammation; Neuroinflammation

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APA (6th Edition):

Shoemaker, J. T. (2015). Development of an in vitro model of neuroinflammation for studying secondary injury mechanisms in traumatic brain injury. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/53878

Chicago Manual of Style (16th Edition):

Shoemaker, James Thomas. “Development of an in vitro model of neuroinflammation for studying secondary injury mechanisms in traumatic brain injury.” 2015. Doctoral Dissertation, Georgia Tech. Accessed October 24, 2020. http://hdl.handle.net/1853/53878.

MLA Handbook (7th Edition):

Shoemaker, James Thomas. “Development of an in vitro model of neuroinflammation for studying secondary injury mechanisms in traumatic brain injury.” 2015. Web. 24 Oct 2020.

Vancouver:

Shoemaker JT. Development of an in vitro model of neuroinflammation for studying secondary injury mechanisms in traumatic brain injury. [Internet] [Doctoral dissertation]. Georgia Tech; 2015. [cited 2020 Oct 24]. Available from: http://hdl.handle.net/1853/53878.

Council of Science Editors:

Shoemaker JT. Development of an in vitro model of neuroinflammation for studying secondary injury mechanisms in traumatic brain injury. [Doctoral Dissertation]. Georgia Tech; 2015. Available from: http://hdl.handle.net/1853/53878

2. Killian, Nathaniel J. Bioelectrical dynamics of the entorhinal cortex.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2013, Georgia Tech

 The entorhinal cortex (EC) in the medial temporal lobe plays a critical role in memory formation and is implicated in several neurological diseases including temporal… (more)

Subjects/Keywords: Spatial representation; Primate; Medial temporal lob; Entorhinal cortex; Hippocampus; Grid cell; Border cell; Memory; Saccade; Fixation; Visual; Stimulus; Saccade-direction cell; Encoding; Recognition; Macaque; Monkey; MTL; Perfusion; Perforated microelectrode array; Neurons; Brain slice; Three-dimensional culture; MEA

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APA (6th Edition):

Killian, N. J. (2013). Bioelectrical dynamics of the entorhinal cortex. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/52148

Chicago Manual of Style (16th Edition):

Killian, Nathaniel J. “Bioelectrical dynamics of the entorhinal cortex.” 2013. Doctoral Dissertation, Georgia Tech. Accessed October 24, 2020. http://hdl.handle.net/1853/52148.

MLA Handbook (7th Edition):

Killian, Nathaniel J. “Bioelectrical dynamics of the entorhinal cortex.” 2013. Web. 24 Oct 2020.

Vancouver:

Killian NJ. Bioelectrical dynamics of the entorhinal cortex. [Internet] [Doctoral dissertation]. Georgia Tech; 2013. [cited 2020 Oct 24]. Available from: http://hdl.handle.net/1853/52148.

Council of Science Editors:

Killian NJ. Bioelectrical dynamics of the entorhinal cortex. [Doctoral Dissertation]. Georgia Tech; 2013. Available from: http://hdl.handle.net/1853/52148


Georgia Tech

3. Michael, Kristin E. FAK Modulates Cell Adhesion Strengthening Via Two Distinct Mechanisms: Integrin Binding and Vinculin Localization.

Degree: PhD, Bioengineering, 2006, Georgia Tech

 Cell adhesion to the extracellular matrix (ECM) provides tissue structure and integrity as well as triggers signals that regulate complex biological processes such as cell… (more)

Subjects/Keywords: Focal adhesion kinase; Extracellular matrix; Integrins; Cell adhesion; Cell mechanics; Force; Migration; Fibronectin; Immobilized proteins; Integrins; Cell adhesion; Extracellular matrix; Fibronectins; Focal adhesion kinase

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APA (6th Edition):

Michael, K. E. (2006). FAK Modulates Cell Adhesion Strengthening Via Two Distinct Mechanisms: Integrin Binding and Vinculin Localization. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/14136

Chicago Manual of Style (16th Edition):

Michael, Kristin E. “FAK Modulates Cell Adhesion Strengthening Via Two Distinct Mechanisms: Integrin Binding and Vinculin Localization.” 2006. Doctoral Dissertation, Georgia Tech. Accessed October 24, 2020. http://hdl.handle.net/1853/14136.

MLA Handbook (7th Edition):

Michael, Kristin E. “FAK Modulates Cell Adhesion Strengthening Via Two Distinct Mechanisms: Integrin Binding and Vinculin Localization.” 2006. Web. 24 Oct 2020.

Vancouver:

Michael KE. FAK Modulates Cell Adhesion Strengthening Via Two Distinct Mechanisms: Integrin Binding and Vinculin Localization. [Internet] [Doctoral dissertation]. Georgia Tech; 2006. [cited 2020 Oct 24]. Available from: http://hdl.handle.net/1853/14136.

Council of Science Editors:

Michael KE. FAK Modulates Cell Adhesion Strengthening Via Two Distinct Mechanisms: Integrin Binding and Vinculin Localization. [Doctoral Dissertation]. Georgia Tech; 2006. Available from: http://hdl.handle.net/1853/14136


Georgia Tech

4. Jain, Anjana. Delivery of Cdc42, Rac1, and Brain-derived Neurotrophic Factor to Promote Axonal Outgrowth After Spinal Cord Injury.

Degree: PhD, Biomedical Engineering, 2007, Georgia Tech

 Injury severs the axons in the spinal cord causing permanent functional loss. After injury, a series of events occur around the lesion site, including the… (more)

Subjects/Keywords: Spinal cord injury; CNS scaffolds; Rho GTPases; BDNF; Nerve regeneration; CNS drug delivery

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jain, A. (2007). Delivery of Cdc42, Rac1, and Brain-derived Neurotrophic Factor to Promote Axonal Outgrowth After Spinal Cord Injury. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/16210

Chicago Manual of Style (16th Edition):

Jain, Anjana. “Delivery of Cdc42, Rac1, and Brain-derived Neurotrophic Factor to Promote Axonal Outgrowth After Spinal Cord Injury.” 2007. Doctoral Dissertation, Georgia Tech. Accessed October 24, 2020. http://hdl.handle.net/1853/16210.

MLA Handbook (7th Edition):

Jain, Anjana. “Delivery of Cdc42, Rac1, and Brain-derived Neurotrophic Factor to Promote Axonal Outgrowth After Spinal Cord Injury.” 2007. Web. 24 Oct 2020.

Vancouver:

Jain A. Delivery of Cdc42, Rac1, and Brain-derived Neurotrophic Factor to Promote Axonal Outgrowth After Spinal Cord Injury. [Internet] [Doctoral dissertation]. Georgia Tech; 2007. [cited 2020 Oct 24]. Available from: http://hdl.handle.net/1853/16210.

Council of Science Editors:

Jain A. Delivery of Cdc42, Rac1, and Brain-derived Neurotrophic Factor to Promote Axonal Outgrowth After Spinal Cord Injury. [Doctoral Dissertation]. Georgia Tech; 2007. Available from: http://hdl.handle.net/1853/16210

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