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You searched for +publisher:"Georgia Tech" +contributor:("Jo, Hanjoong"). Showing records 1 – 30 of 36 total matches.

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1. Salim, Md Tausif Tausif. Role of microRNA 214 in cyclic stretch induced aortic valve pathogenesis.

Degree: MS, Chemical and Biomolecular Engineering, 2018, Georgia Tech

 Calcific aortic valve disease (CAVD) is one of the most prevalent valvular diseases among the elderly population. Unfortunately, there are no therapeutic drugs available to… (more)

Subjects/Keywords: Aortic valve; miR-214; Cyclic stretch

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APA (6th Edition):

Salim, M. T. T. (2018). Role of microRNA 214 in cyclic stretch induced aortic valve pathogenesis. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/61174

Chicago Manual of Style (16th Edition):

Salim, Md Tausif Tausif. “Role of microRNA 214 in cyclic stretch induced aortic valve pathogenesis.” 2018. Masters Thesis, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/61174.

MLA Handbook (7th Edition):

Salim, Md Tausif Tausif. “Role of microRNA 214 in cyclic stretch induced aortic valve pathogenesis.” 2018. Web. 16 Apr 2021.

Vancouver:

Salim MTT. Role of microRNA 214 in cyclic stretch induced aortic valve pathogenesis. [Internet] [Masters thesis]. Georgia Tech; 2018. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/61174.

Council of Science Editors:

Salim MTT. Role of microRNA 214 in cyclic stretch induced aortic valve pathogenesis. [Masters Thesis]. Georgia Tech; 2018. Available from: http://hdl.handle.net/1853/61174


Georgia Tech

2. Sei, Yoshitaka John. Microfluidics for translating multifunctional nanomaterials.

Degree: PhD, Mechanical Engineering, 2018, Georgia Tech

 Conflicting results between benchtop experiments, animal models, and clinical trials have posed a significant challenge in translational medical research, motivating the development of technologies with… (more)

Subjects/Keywords: Microfluidics; Nanomaterials

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APA (6th Edition):

Sei, Y. J. (2018). Microfluidics for translating multifunctional nanomaterials. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/62187

Chicago Manual of Style (16th Edition):

Sei, Yoshitaka John. “Microfluidics for translating multifunctional nanomaterials.” 2018. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/62187.

MLA Handbook (7th Edition):

Sei, Yoshitaka John. “Microfluidics for translating multifunctional nanomaterials.” 2018. Web. 16 Apr 2021.

Vancouver:

Sei YJ. Microfluidics for translating multifunctional nanomaterials. [Internet] [Doctoral dissertation]. Georgia Tech; 2018. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/62187.

Council of Science Editors:

Sei YJ. Microfluidics for translating multifunctional nanomaterials. [Doctoral Dissertation]. Georgia Tech; 2018. Available from: http://hdl.handle.net/1853/62187


Georgia Tech

3. Gray, Warren Dale. Development of therapeutic systems to treat the infarcted heart.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2014, Georgia Tech

 Cardiovascular disease is the leading cause of morbidity and mortality in developed nations, and heart disease is predicted to remain the leading killer for the… (more)

Subjects/Keywords: Myocardial infarction; Cardiac protection; Cardiac regeneration; Angiogenesis; Therapeutic system; Nanoparticle; Dendrimer; Exosome; Hypoxia; N-acetylglucosamine

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APA (6th Edition):

Gray, W. D. (2014). Development of therapeutic systems to treat the infarcted heart. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/53429

Chicago Manual of Style (16th Edition):

Gray, Warren Dale. “Development of therapeutic systems to treat the infarcted heart.” 2014. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/53429.

MLA Handbook (7th Edition):

Gray, Warren Dale. “Development of therapeutic systems to treat the infarcted heart.” 2014. Web. 16 Apr 2021.

Vancouver:

Gray WD. Development of therapeutic systems to treat the infarcted heart. [Internet] [Doctoral dissertation]. Georgia Tech; 2014. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/53429.

Council of Science Editors:

Gray WD. Development of therapeutic systems to treat the infarcted heart. [Doctoral Dissertation]. Georgia Tech; 2014. Available from: http://hdl.handle.net/1853/53429


Georgia Tech

4. Rathan, Swetha. Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification.

Degree: PhD, Chemical and Biomolecular Engineering, 2016, Georgia Tech

 Calcific aortic valve (AV) disease is a strong risk factor for cardiovascular related deaths and is a significant source of mortality worldwide, with the number… (more)

Subjects/Keywords: Aortic valve; Hemodynamics; Mechanobiology; MicroRNA; Calcification

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APA (6th Edition):

Rathan, S. (2016). Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/58144

Chicago Manual of Style (16th Edition):

Rathan, Swetha. “Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification.” 2016. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/58144.

MLA Handbook (7th Edition):

Rathan, Swetha. “Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification.” 2016. Web. 16 Apr 2021.

Vancouver:

Rathan S. Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification. [Internet] [Doctoral dissertation]. Georgia Tech; 2016. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/58144.

Council of Science Editors:

Rathan S. Aortic valve mechanobiology- role of altered hemodynamics in mediating aortic valve inflammation and calcification. [Doctoral Dissertation]. Georgia Tech; 2016. Available from: http://hdl.handle.net/1853/58144


Georgia Tech

5. Caesar, Christa. The role and regulation of osteopontin in hypertension related remodeling and inflammation of the aorta.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2016, Georgia Tech

 The arterial wall experiences elevated mechanical strain in the setting of hypertension, which leads to vascular inflammation and atherosclerosis. However, many of the underlying molecular… (more)

Subjects/Keywords: Osteopontin; Hypertension; Aorta; Mechanical strain; Hydrogen peroxide

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APA (6th Edition):

Caesar, C. (2016). The role and regulation of osteopontin in hypertension related remodeling and inflammation of the aorta. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/58157

Chicago Manual of Style (16th Edition):

Caesar, Christa. “The role and regulation of osteopontin in hypertension related remodeling and inflammation of the aorta.” 2016. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/58157.

MLA Handbook (7th Edition):

Caesar, Christa. “The role and regulation of osteopontin in hypertension related remodeling and inflammation of the aorta.” 2016. Web. 16 Apr 2021.

Vancouver:

Caesar C. The role and regulation of osteopontin in hypertension related remodeling and inflammation of the aorta. [Internet] [Doctoral dissertation]. Georgia Tech; 2016. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/58157.

Council of Science Editors:

Caesar C. The role and regulation of osteopontin in hypertension related remodeling and inflammation of the aorta. [Doctoral Dissertation]. Georgia Tech; 2016. Available from: http://hdl.handle.net/1853/58157


Georgia Tech

6. Wen, Mary Mei. New strategies for tagging quantum dots for dynamic cellular imaging.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2013, Georgia Tech

 In recent years, semiconductor quantum dots (QDs) have arisen as a new class of fluorescent probes that possess unique optical and electronic properties well-suited for… (more)

Subjects/Keywords: Quantum dots; Live cell imaging; Single molecule imaging; HaloTag; Tagging strategies; Nanotechnology; Bionanotechnology

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APA (6th Edition):

Wen, M. M. (2013). New strategies for tagging quantum dots for dynamic cellular imaging. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/52150

Chicago Manual of Style (16th Edition):

Wen, Mary Mei. “New strategies for tagging quantum dots for dynamic cellular imaging.” 2013. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/52150.

MLA Handbook (7th Edition):

Wen, Mary Mei. “New strategies for tagging quantum dots for dynamic cellular imaging.” 2013. Web. 16 Apr 2021.

Vancouver:

Wen MM. New strategies for tagging quantum dots for dynamic cellular imaging. [Internet] [Doctoral dissertation]. Georgia Tech; 2013. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/52150.

Council of Science Editors:

Wen MM. New strategies for tagging quantum dots for dynamic cellular imaging. [Doctoral Dissertation]. Georgia Tech; 2013. Available from: http://hdl.handle.net/1853/52150


Georgia Tech

7. Keegan, Philip Michael. Shear stress, hemodynamics, and proteolytic mechanisms underlying large artery remodeling in sickle cell disease.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2014, Georgia Tech

 Sickle cell disease is a genetic disorder that affects 100,000 Americans and millions more worldwide. Although the sickle mutation affects one protein, which is only… (more)

Subjects/Keywords: Sickle cell disease; Arterial remodeling; Cathepsins; Stroke; Hemodynamics; Shear stress

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APA (6th Edition):

Keegan, P. M. (2014). Shear stress, hemodynamics, and proteolytic mechanisms underlying large artery remodeling in sickle cell disease. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/54289

Chicago Manual of Style (16th Edition):

Keegan, Philip Michael. “Shear stress, hemodynamics, and proteolytic mechanisms underlying large artery remodeling in sickle cell disease.” 2014. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/54289.

MLA Handbook (7th Edition):

Keegan, Philip Michael. “Shear stress, hemodynamics, and proteolytic mechanisms underlying large artery remodeling in sickle cell disease.” 2014. Web. 16 Apr 2021.

Vancouver:

Keegan PM. Shear stress, hemodynamics, and proteolytic mechanisms underlying large artery remodeling in sickle cell disease. [Internet] [Doctoral dissertation]. Georgia Tech; 2014. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/54289.

Council of Science Editors:

Keegan PM. Shear stress, hemodynamics, and proteolytic mechanisms underlying large artery remodeling in sickle cell disease. [Doctoral Dissertation]. Georgia Tech; 2014. Available from: http://hdl.handle.net/1853/54289

8. Balderrama, Fanor Alberto. Incorporation of recombinant fibronectin into genetically engineered elastin-based polymers.

Degree: MS, Bioengineering, 2009, Georgia Tech

 Cardiovascular disease is the main cause of death in the United States. Many of these conditions require the grafting or bypassing of compromised blood vessels.… (more)

Subjects/Keywords: Functionalization; Coating; Protein; Crosslinking; Genipin; HUVEC; Stability; Cell proliferation; Vascular graft; Elastin; FNIII7-10; Cell adhesion; Endothelialization; Endothelial; Fibronectins; Globulins; Biomedical materials; Vascular grafts

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APA (6th Edition):

Balderrama, F. A. (2009). Incorporation of recombinant fibronectin into genetically engineered elastin-based polymers. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/31640

Chicago Manual of Style (16th Edition):

Balderrama, Fanor Alberto. “Incorporation of recombinant fibronectin into genetically engineered elastin-based polymers.” 2009. Masters Thesis, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/31640.

MLA Handbook (7th Edition):

Balderrama, Fanor Alberto. “Incorporation of recombinant fibronectin into genetically engineered elastin-based polymers.” 2009. Web. 16 Apr 2021.

Vancouver:

Balderrama FA. Incorporation of recombinant fibronectin into genetically engineered elastin-based polymers. [Internet] [Masters thesis]. Georgia Tech; 2009. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/31640.

Council of Science Editors:

Balderrama FA. Incorporation of recombinant fibronectin into genetically engineered elastin-based polymers. [Masters Thesis]. Georgia Tech; 2009. Available from: http://hdl.handle.net/1853/31640


Georgia Tech

9. Fernandez Esmerats, Joan. The role of flow-sensitive MiRNAs and UBE2C-dependent HIF1α pathway in calcific aortic valve disease.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2018, Georgia Tech

 Calcific Aortic Valve Disease (CAVD), characterized by aortic valve (AV) stenosis and insufficiency (regurgitation), is a major cause of cardiac-related deaths worldwide, especially in the… (more)

Subjects/Keywords: OS; miRNA; HAVECs; LS; TIMP3; UBE2C; HIF1A; pVHL; KLF2; Inflammation; Aortic valve; Calcification

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APA (6th Edition):

Fernandez Esmerats, J. (2018). The role of flow-sensitive MiRNAs and UBE2C-dependent HIF1α pathway in calcific aortic valve disease. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/62206

Chicago Manual of Style (16th Edition):

Fernandez Esmerats, Joan. “The role of flow-sensitive MiRNAs and UBE2C-dependent HIF1α pathway in calcific aortic valve disease.” 2018. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/62206.

MLA Handbook (7th Edition):

Fernandez Esmerats, Joan. “The role of flow-sensitive MiRNAs and UBE2C-dependent HIF1α pathway in calcific aortic valve disease.” 2018. Web. 16 Apr 2021.

Vancouver:

Fernandez Esmerats J. The role of flow-sensitive MiRNAs and UBE2C-dependent HIF1α pathway in calcific aortic valve disease. [Internet] [Doctoral dissertation]. Georgia Tech; 2018. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/62206.

Council of Science Editors:

Fernandez Esmerats J. The role of flow-sensitive MiRNAs and UBE2C-dependent HIF1α pathway in calcific aortic valve disease. [Doctoral Dissertation]. Georgia Tech; 2018. Available from: http://hdl.handle.net/1853/62206


Georgia Tech

10. Doroudi, Maryam. Essential roles of Pdia3/PLAA receptor complex and CaMKII IN 1α,25(OH)₂D₃ and Wnt5a calcium-dependent signaling pathways in osteoblasts and chondrocytes.

Degree: PhD, Biology, 2014, Georgia Tech

 The vitamin D metabolite 1,25-dihydroxyvitamin D3 [1α,25(OH)2D3] plays an important role in the regulation of musculoskeletal growth and differentiation. 1α,25(OH)2D3 mediates its effects on cells,… (more)

Subjects/Keywords: 1,25-dihydroxyvitamin D3; Growth plate

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APA (6th Edition):

Doroudi, M. (2014). Essential roles of Pdia3/PLAA receptor complex and CaMKII IN 1α,25(OH)₂D₃ and Wnt5a calcium-dependent signaling pathways in osteoblasts and chondrocytes. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/53427

Chicago Manual of Style (16th Edition):

Doroudi, Maryam. “Essential roles of Pdia3/PLAA receptor complex and CaMKII IN 1α,25(OH)₂D₃ and Wnt5a calcium-dependent signaling pathways in osteoblasts and chondrocytes.” 2014. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/53427.

MLA Handbook (7th Edition):

Doroudi, Maryam. “Essential roles of Pdia3/PLAA receptor complex and CaMKII IN 1α,25(OH)₂D₃ and Wnt5a calcium-dependent signaling pathways in osteoblasts and chondrocytes.” 2014. Web. 16 Apr 2021.

Vancouver:

Doroudi M. Essential roles of Pdia3/PLAA receptor complex and CaMKII IN 1α,25(OH)₂D₃ and Wnt5a calcium-dependent signaling pathways in osteoblasts and chondrocytes. [Internet] [Doctoral dissertation]. Georgia Tech; 2014. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/53427.

Council of Science Editors:

Doroudi M. Essential roles of Pdia3/PLAA receptor complex and CaMKII IN 1α,25(OH)₂D₃ and Wnt5a calcium-dependent signaling pathways in osteoblasts and chondrocytes. [Doctoral Dissertation]. Georgia Tech; 2014. Available from: http://hdl.handle.net/1853/53427


Georgia Tech

11. Yap, Choon Hwai. The fluid shear stress environment of the normal and congenital bicuspid aortic valve and the implications on valve calcification.

Degree: PhD, Biomedical Engineering, 2011, Georgia Tech

 Calcific aortic valve disease is highly prevalent, especially in the elderly. Currently, the exact mechanism of the calcification process is not completely understood, limiting our… (more)

Subjects/Keywords: Bicuspid aortic valve; Laser doppler velocimetry; Cone and plate bioreactor; Shear stress; Fluid mechanics; Aortic valve; Aortic valve calcification disease; Aortic valve Diseases; Aortic valve insufficiency; Aortic valve Stenosis; Mitral valve

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APA (6th Edition):

Yap, C. H. (2011). The fluid shear stress environment of the normal and congenital bicuspid aortic valve and the implications on valve calcification. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/45742

Chicago Manual of Style (16th Edition):

Yap, Choon Hwai. “The fluid shear stress environment of the normal and congenital bicuspid aortic valve and the implications on valve calcification.” 2011. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/45742.

MLA Handbook (7th Edition):

Yap, Choon Hwai. “The fluid shear stress environment of the normal and congenital bicuspid aortic valve and the implications on valve calcification.” 2011. Web. 16 Apr 2021.

Vancouver:

Yap CH. The fluid shear stress environment of the normal and congenital bicuspid aortic valve and the implications on valve calcification. [Internet] [Doctoral dissertation]. Georgia Tech; 2011. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/45742.

Council of Science Editors:

Yap CH. The fluid shear stress environment of the normal and congenital bicuspid aortic valve and the implications on valve calcification. [Doctoral Dissertation]. Georgia Tech; 2011. Available from: http://hdl.handle.net/1853/45742

12. Yin, Weiwei. The role and regulatory mechanisms of nox1 in vascular systems.

Degree: PhD, Biomedical Engineering, 2012, Georgia Tech

 As an important endogenous source of reactive oxygen species (ROS), NADPH oxidase 1 (Nox1) has received tremendous attention in the past few decades. It has… (more)

Subjects/Keywords: Atherosclerosis; Mechanotransduction; NADPH oxidase 1; Computational model; Biochemical systems theory; System biology; Cardiovascular system; Design princple; Reactive oxygen species; Pathway analysis; Multi-scale system modeling; Signaling pathway modeling; Monte Carlo method; Dynamic simulation; Endothelium; Nitric oxide; Active oxygen

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APA (6th Edition):

Yin, W. (2012). The role and regulatory mechanisms of nox1 in vascular systems. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/44833

Chicago Manual of Style (16th Edition):

Yin, Weiwei. “The role and regulatory mechanisms of nox1 in vascular systems.” 2012. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/44833.

MLA Handbook (7th Edition):

Yin, Weiwei. “The role and regulatory mechanisms of nox1 in vascular systems.” 2012. Web. 16 Apr 2021.

Vancouver:

Yin W. The role and regulatory mechanisms of nox1 in vascular systems. [Internet] [Doctoral dissertation]. Georgia Tech; 2012. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/44833.

Council of Science Editors:

Yin W. The role and regulatory mechanisms of nox1 in vascular systems. [Doctoral Dissertation]. Georgia Tech; 2012. Available from: http://hdl.handle.net/1853/44833

13. Zhong, Ming. Apoptotic signaling pathways in mammalian growth plate chondrocytes.

Degree: PhD, Biomedical Engineering, 2010, Georgia Tech

 The growth plate resting zone consists of hyaline-like chondrocytes disbursed in a proteoglycan rich extracellular matrix. These cells give rise to the columns of the… (more)

Subjects/Keywords: Apoptosis; MAP kinases; Nitric oxide; Phosphates; P53; Estrogen; Growth plate chondrocytes; Endochondral ossification; Growth plate; Bones Growth; Cartilage; Bone; Ossification

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APA (6th Edition):

Zhong, M. (2010). Apoptotic signaling pathways in mammalian growth plate chondrocytes. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/33993

Chicago Manual of Style (16th Edition):

Zhong, Ming. “Apoptotic signaling pathways in mammalian growth plate chondrocytes.” 2010. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/33993.

MLA Handbook (7th Edition):

Zhong, Ming. “Apoptotic signaling pathways in mammalian growth plate chondrocytes.” 2010. Web. 16 Apr 2021.

Vancouver:

Zhong M. Apoptotic signaling pathways in mammalian growth plate chondrocytes. [Internet] [Doctoral dissertation]. Georgia Tech; 2010. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/33993.

Council of Science Editors:

Zhong M. Apoptotic signaling pathways in mammalian growth plate chondrocytes. [Doctoral Dissertation]. Georgia Tech; 2010. Available from: http://hdl.handle.net/1853/33993

14. Sargent, Carolyn Yeago. Effects of hydrodynamic culture on embryonic stem cell differentiation: cardiogenic modulation.

Degree: PhD, Biomedical Engineering, 2010, Georgia Tech

 Stem and progenitor cells are an attractive cell source for the treatment of degenerative diseases due to their potential to differentiate into multiple cell types… (more)

Subjects/Keywords: Embryonic stem cells; Embryoid body; Hydrodynamic; Bioprocessing; Differentiation; Cardiomyocyte; Heart cells; Degeneration (Pathology); Hydrodynamics; Embryonic stem cells Research

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APA (6th Edition):

Sargent, C. Y. (2010). Effects of hydrodynamic culture on embryonic stem cell differentiation: cardiogenic modulation. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/34710

Chicago Manual of Style (16th Edition):

Sargent, Carolyn Yeago. “Effects of hydrodynamic culture on embryonic stem cell differentiation: cardiogenic modulation.” 2010. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/34710.

MLA Handbook (7th Edition):

Sargent, Carolyn Yeago. “Effects of hydrodynamic culture on embryonic stem cell differentiation: cardiogenic modulation.” 2010. Web. 16 Apr 2021.

Vancouver:

Sargent CY. Effects of hydrodynamic culture on embryonic stem cell differentiation: cardiogenic modulation. [Internet] [Doctoral dissertation]. Georgia Tech; 2010. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/34710.

Council of Science Editors:

Sargent CY. Effects of hydrodynamic culture on embryonic stem cell differentiation: cardiogenic modulation. [Doctoral Dissertation]. Georgia Tech; 2010. Available from: http://hdl.handle.net/1853/34710

15. Simmons, Rachel Diane. MIR-744 modulation by disturbed flow and its role in endothelial inflammation and atherosclerosis.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2017, Georgia Tech

 Atherosclerosis is the leading cause of death in developed nations as it is the underlying cause of many cardiovascular diseases (CVD) such as myocardial infarction,… (more)

Subjects/Keywords: miRNAs; Hemodynamics; Atherosclerosis; Endothelial inflammation

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APA (6th Edition):

Simmons, R. D. (2017). MIR-744 modulation by disturbed flow and its role in endothelial inflammation and atherosclerosis. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/60679

Chicago Manual of Style (16th Edition):

Simmons, Rachel Diane. “MIR-744 modulation by disturbed flow and its role in endothelial inflammation and atherosclerosis.” 2017. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/60679.

MLA Handbook (7th Edition):

Simmons, Rachel Diane. “MIR-744 modulation by disturbed flow and its role in endothelial inflammation and atherosclerosis.” 2017. Web. 16 Apr 2021.

Vancouver:

Simmons RD. MIR-744 modulation by disturbed flow and its role in endothelial inflammation and atherosclerosis. [Internet] [Doctoral dissertation]. Georgia Tech; 2017. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/60679.

Council of Science Editors:

Simmons RD. MIR-744 modulation by disturbed flow and its role in endothelial inflammation and atherosclerosis. [Doctoral Dissertation]. Georgia Tech; 2017. Available from: http://hdl.handle.net/1853/60679

16. Holliday, Casey Jane. Discovery of shear- and side-dependent messenger RNAs and microRNAs in aortic valvular endothelium.

Degree: PhD, Biomedical Engineering, 2012, Georgia Tech

 Aortic valve (AV) disease is a major cause of cardiovascular-linked deaths globally. In addition, AV disease is a strong risk factor for additional cardiovascular events;… (more)

Subjects/Keywords: Aortic valve; Endothelium; MicroRNAs; Shear stress; Microarrays; MRNAs; Aortic valve Diseases; Aortic valve Stenosis; Messenger RNA; Vascular endothelium

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APA (6th Edition):

Holliday, C. J. (2012). Discovery of shear- and side-dependent messenger RNAs and microRNAs in aortic valvular endothelium. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/47517

Chicago Manual of Style (16th Edition):

Holliday, Casey Jane. “Discovery of shear- and side-dependent messenger RNAs and microRNAs in aortic valvular endothelium.” 2012. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/47517.

MLA Handbook (7th Edition):

Holliday, Casey Jane. “Discovery of shear- and side-dependent messenger RNAs and microRNAs in aortic valvular endothelium.” 2012. Web. 16 Apr 2021.

Vancouver:

Holliday CJ. Discovery of shear- and side-dependent messenger RNAs and microRNAs in aortic valvular endothelium. [Internet] [Doctoral dissertation]. Georgia Tech; 2012. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/47517.

Council of Science Editors:

Holliday CJ. Discovery of shear- and side-dependent messenger RNAs and microRNAs in aortic valvular endothelium. [Doctoral Dissertation]. Georgia Tech; 2012. Available from: http://hdl.handle.net/1853/47517

17. Boopathy, Archana Vidya. Engineering stem cell responses using oxidative stress and notch ligand containing hydrogels.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2014, Georgia Tech

 Heart failure is the leading cause of death worldwide. In 2013, the American Heart Association estimated that one American will die of cardiovascular disease every… (more)

Subjects/Keywords: Adult stem cells; Notch; Hydrogel; Oxidative stress; Myocardial infarction; Colloids in medicine; Biocolloids; Myocardium Regeneration; Mesenchymal stem cells; Oxidative stress; Notch genes

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APA (6th Edition):

Boopathy, A. V. (2014). Engineering stem cell responses using oxidative stress and notch ligand containing hydrogels. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/51837

Chicago Manual of Style (16th Edition):

Boopathy, Archana Vidya. “Engineering stem cell responses using oxidative stress and notch ligand containing hydrogels.” 2014. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/51837.

MLA Handbook (7th Edition):

Boopathy, Archana Vidya. “Engineering stem cell responses using oxidative stress and notch ligand containing hydrogels.” 2014. Web. 16 Apr 2021.

Vancouver:

Boopathy AV. Engineering stem cell responses using oxidative stress and notch ligand containing hydrogels. [Internet] [Doctoral dissertation]. Georgia Tech; 2014. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/51837.

Council of Science Editors:

Boopathy AV. Engineering stem cell responses using oxidative stress and notch ligand containing hydrogels. [Doctoral Dissertation]. Georgia Tech; 2014. Available from: http://hdl.handle.net/1853/51837

18. Dunn, Jessilyn. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2015, Georgia Tech

 Atherosclerosis is an inflammatory disease of the arterial walls and is the major cause of heart attack and stroke. Atherosclerosis is localized to curves or… (more)

Subjects/Keywords: DNMT; Transcriptome; Shear stress; Disturbed flow; Atherosclerosis; HoxA5; Klf3; Endothelial cells; DNA methylome; Gene expression

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APA (6th Edition):

Dunn, J. (2015). Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/53536

Chicago Manual of Style (16th Edition):

Dunn, Jessilyn. “Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.” 2015. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/53536.

MLA Handbook (7th Edition):

Dunn, Jessilyn. “Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.” 2015. Web. 16 Apr 2021.

Vancouver:

Dunn J. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. [Internet] [Doctoral dissertation]. Georgia Tech; 2015. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/53536.

Council of Science Editors:

Dunn J. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. [Doctoral Dissertation]. Georgia Tech; 2015. Available from: http://hdl.handle.net/1853/53536

19. Prasanphanich, Adam Franklin. Dynamic redox signaling during TGF-beta-induced epithelial-mesenchymal transition.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2015, Georgia Tech

 Individual biological processes operate within larger contexts and can participate in the emergence of complex phenotypes. The morphogen transforming growth factor β (TGFβ) can initiate… (more)

Subjects/Keywords: TGF-beta; Computational modeling; Redox biology

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APA (6th Edition):

Prasanphanich, A. F. (2015). Dynamic redox signaling during TGF-beta-induced epithelial-mesenchymal transition. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/56163

Chicago Manual of Style (16th Edition):

Prasanphanich, Adam Franklin. “Dynamic redox signaling during TGF-beta-induced epithelial-mesenchymal transition.” 2015. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/56163.

MLA Handbook (7th Edition):

Prasanphanich, Adam Franklin. “Dynamic redox signaling during TGF-beta-induced epithelial-mesenchymal transition.” 2015. Web. 16 Apr 2021.

Vancouver:

Prasanphanich AF. Dynamic redox signaling during TGF-beta-induced epithelial-mesenchymal transition. [Internet] [Doctoral dissertation]. Georgia Tech; 2015. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/56163.

Council of Science Editors:

Prasanphanich AF. Dynamic redox signaling during TGF-beta-induced epithelial-mesenchymal transition. [Doctoral Dissertation]. Georgia Tech; 2015. Available from: http://hdl.handle.net/1853/56163

20. Caulk, Alexander Wilson. Biomechanics and modeling methods for quantifying mechanically-mediated disease progression in neglected populations.

Degree: PhD, Mechanical Engineering, 2015, Georgia Tech

 It is well known that biological tissue grows and remodels in response to changes in mechanical loading. Arteries and lymphatic vessels share many similar mechanical… (more)

Subjects/Keywords: Biomechanics; HIV; HAART; lymphedema; growth and remodeling; constitutive modeling

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APA (6th Edition):

Caulk, A. W. (2015). Biomechanics and modeling methods for quantifying mechanically-mediated disease progression in neglected populations. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/55490

Chicago Manual of Style (16th Edition):

Caulk, Alexander Wilson. “Biomechanics and modeling methods for quantifying mechanically-mediated disease progression in neglected populations.” 2015. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/55490.

MLA Handbook (7th Edition):

Caulk, Alexander Wilson. “Biomechanics and modeling methods for quantifying mechanically-mediated disease progression in neglected populations.” 2015. Web. 16 Apr 2021.

Vancouver:

Caulk AW. Biomechanics and modeling methods for quantifying mechanically-mediated disease progression in neglected populations. [Internet] [Doctoral dissertation]. Georgia Tech; 2015. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/55490.

Council of Science Editors:

Caulk AW. Biomechanics and modeling methods for quantifying mechanically-mediated disease progression in neglected populations. [Doctoral Dissertation]. Georgia Tech; 2015. Available from: http://hdl.handle.net/1853/55490

21. Sy, Jay Christopher. Novel strategies for cardiac drug delivery.

Degree: PhD, Biomedical Engineering, 2011, Georgia Tech

 The American Heart Association (AHA) estimates that at least one American will die from a coronary event every minute, costing over $150 billion in 2008… (more)

Subjects/Keywords: Cardiac dysfunction; Myocardial infarction; Necrosis; Hoechst; Nitrilotriacetic acid; Polyketals; Biomaterials; Heart disease; Drug delivery; Drug delivery systems; Guided tissue regeneration

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APA (6th Edition):

Sy, J. C. (2011). Novel strategies for cardiac drug delivery. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/39531

Chicago Manual of Style (16th Edition):

Sy, Jay Christopher. “Novel strategies for cardiac drug delivery.” 2011. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/39531.

MLA Handbook (7th Edition):

Sy, Jay Christopher. “Novel strategies for cardiac drug delivery.” 2011. Web. 16 Apr 2021.

Vancouver:

Sy JC. Novel strategies for cardiac drug delivery. [Internet] [Doctoral dissertation]. Georgia Tech; 2011. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/39531.

Council of Science Editors:

Sy JC. Novel strategies for cardiac drug delivery. [Doctoral Dissertation]. Georgia Tech; 2011. Available from: http://hdl.handle.net/1853/39531


Georgia Tech

22. Bellott, Anne Claire. The Role of Caveolae in the Loss of ERK2 Activation in Stretched Skeletal Myotubes.

Degree: MS, Mechanical Engineering, 2004, Georgia Tech

 Skeletal muscle function is important to the human body for daily activities. Mechanical signals are critical to the maintenance of that function. Muscle diseases, such… (more)

Subjects/Keywords: Skeletal muscle; ERK2; Mechanotransduction; Caveolae; Caveolin-3

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APA (6th Edition):

Bellott, A. C. (2004). The Role of Caveolae in the Loss of ERK2 Activation in Stretched Skeletal Myotubes. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/5075

Chicago Manual of Style (16th Edition):

Bellott, Anne Claire. “The Role of Caveolae in the Loss of ERK2 Activation in Stretched Skeletal Myotubes.” 2004. Masters Thesis, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/5075.

MLA Handbook (7th Edition):

Bellott, Anne Claire. “The Role of Caveolae in the Loss of ERK2 Activation in Stretched Skeletal Myotubes.” 2004. Web. 16 Apr 2021.

Vancouver:

Bellott AC. The Role of Caveolae in the Loss of ERK2 Activation in Stretched Skeletal Myotubes. [Internet] [Masters thesis]. Georgia Tech; 2004. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/5075.

Council of Science Editors:

Bellott AC. The Role of Caveolae in the Loss of ERK2 Activation in Stretched Skeletal Myotubes. [Masters Thesis]. Georgia Tech; 2004. Available from: http://hdl.handle.net/1853/5075


Georgia Tech

23. Wong, Kevin L. Caveolae and Caveolin-1 are important for Vitamin D signalling.

Degree: MS, Biomedical Engineering, 2010, Georgia Tech

 The most active form of Vitamin D, 1alpha,25(OH)2D3, modulates cells via receptor mediated mechanisms. While studies have elucidated the pathway via the classical nuclear Vitamin… (more)

Subjects/Keywords: Chondrocytes; Lipid raft; Cav-1 knockout mice; Matrix vesicles; Protein kinase C; Cartilage cells; Human physiology; Cholecalciferol; Vitamin D

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APA (6th Edition):

Wong, K. L. (2010). Caveolae and Caveolin-1 are important for Vitamin D signalling. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/37086

Chicago Manual of Style (16th Edition):

Wong, Kevin L. “Caveolae and Caveolin-1 are important for Vitamin D signalling.” 2010. Masters Thesis, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/37086.

MLA Handbook (7th Edition):

Wong, Kevin L. “Caveolae and Caveolin-1 are important for Vitamin D signalling.” 2010. Web. 16 Apr 2021.

Vancouver:

Wong KL. Caveolae and Caveolin-1 are important for Vitamin D signalling. [Internet] [Masters thesis]. Georgia Tech; 2010. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/37086.

Council of Science Editors:

Wong KL. Caveolae and Caveolin-1 are important for Vitamin D signalling. [Masters Thesis]. Georgia Tech; 2010. Available from: http://hdl.handle.net/1853/37086


Georgia Tech

24. Tressel, Sarah Lynne. Role of shear stress in angiopoietin-2-dependent neovascularization: implications in occlusive vascular disease and atherosclerosis.

Degree: PhD, Biomedical Engineering, 2008, Georgia Tech

 Neovascularization, or the formation of blood vessels, is important in both normal physiological processes as well as pathophysiological processes. The main players in neovascularization, endothelial… (more)

Subjects/Keywords: Hindlimb ischemia; Endothelial cells; Arteriogenesis; Shear stress; Angiopoietin-2; Angiogenesis; Vascular endothelium; Blood-vessels Growth; Shear flow; Neovascularization

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APA (6th Edition):

Tressel, S. L. (2008). Role of shear stress in angiopoietin-2-dependent neovascularization: implications in occlusive vascular disease and atherosclerosis. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/22646

Chicago Manual of Style (16th Edition):

Tressel, Sarah Lynne. “Role of shear stress in angiopoietin-2-dependent neovascularization: implications in occlusive vascular disease and atherosclerosis.” 2008. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/22646.

MLA Handbook (7th Edition):

Tressel, Sarah Lynne. “Role of shear stress in angiopoietin-2-dependent neovascularization: implications in occlusive vascular disease and atherosclerosis.” 2008. Web. 16 Apr 2021.

Vancouver:

Tressel SL. Role of shear stress in angiopoietin-2-dependent neovascularization: implications in occlusive vascular disease and atherosclerosis. [Internet] [Doctoral dissertation]. Georgia Tech; 2008. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/22646.

Council of Science Editors:

Tressel SL. Role of shear stress in angiopoietin-2-dependent neovascularization: implications in occlusive vascular disease and atherosclerosis. [Doctoral Dissertation]. Georgia Tech; 2008. Available from: http://hdl.handle.net/1853/22646


Georgia Tech

25. Xing, Yun. Effects of Mechanical Forces on the Biological Properties of Porcine Aortic Valve Leaflets.

Degree: PhD, Bioengineering, 2005, Georgia Tech

 Cardiac valves are dynamic, sophisticated structures which interact closely with the surrounding hemodynamic environment. Altered mechanical stresses, including pressure, shear and bending stresses, are believed… (more)

Subjects/Keywords: Heart valves; Mechanical forces; Tissue engineering; Shear flow; Strains and stresses; Heart valves; Hemodynamics

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APA (6th Edition):

Xing, Y. (2005). Effects of Mechanical Forces on the Biological Properties of Porcine Aortic Valve Leaflets. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/6828

Chicago Manual of Style (16th Edition):

Xing, Yun. “Effects of Mechanical Forces on the Biological Properties of Porcine Aortic Valve Leaflets.” 2005. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/6828.

MLA Handbook (7th Edition):

Xing, Yun. “Effects of Mechanical Forces on the Biological Properties of Porcine Aortic Valve Leaflets.” 2005. Web. 16 Apr 2021.

Vancouver:

Xing Y. Effects of Mechanical Forces on the Biological Properties of Porcine Aortic Valve Leaflets. [Internet] [Doctoral dissertation]. Georgia Tech; 2005. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/6828.

Council of Science Editors:

Xing Y. Effects of Mechanical Forces on the Biological Properties of Porcine Aortic Valve Leaflets. [Doctoral Dissertation]. Georgia Tech; 2005. Available from: http://hdl.handle.net/1853/6828


Georgia Tech

26. Sykes, Michelle Christine. Regulation of endothelial gene transcription by shear stress in a manner dependent on p47phox-based NADPH oxidases.

Degree: PhD, Biomedical Engineering, 2008, Georgia Tech

 Atherosclerosis occurs preferentially at branches and curves in arteries exposed to disturbed flow while sparing straight portions of arteries exposed to undisturbed flow. In vivo… (more)

Subjects/Keywords: Endothelial cell; P47phox; NADPH oxidase; Microarray; Shear stress; Atherosclerosis; NAD (Coenzyme); Oxidases; Shear flow; Endothelium

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APA (6th Edition):

Sykes, M. C. (2008). Regulation of endothelial gene transcription by shear stress in a manner dependent on p47phox-based NADPH oxidases. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/24824

Chicago Manual of Style (16th Edition):

Sykes, Michelle Christine. “Regulation of endothelial gene transcription by shear stress in a manner dependent on p47phox-based NADPH oxidases.” 2008. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/24824.

MLA Handbook (7th Edition):

Sykes, Michelle Christine. “Regulation of endothelial gene transcription by shear stress in a manner dependent on p47phox-based NADPH oxidases.” 2008. Web. 16 Apr 2021.

Vancouver:

Sykes MC. Regulation of endothelial gene transcription by shear stress in a manner dependent on p47phox-based NADPH oxidases. [Internet] [Doctoral dissertation]. Georgia Tech; 2008. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/24824.

Council of Science Editors:

Sykes MC. Regulation of endothelial gene transcription by shear stress in a manner dependent on p47phox-based NADPH oxidases. [Doctoral Dissertation]. Georgia Tech; 2008. Available from: http://hdl.handle.net/1853/24824


Georgia Tech

27. Tolentino, Timothy P. The Roles of Membrane Rafts in CD32A Mediated Formation of a Phagocytic Contact Area.

Degree: PhD, Biomedical Engineering, 2007, Georgia Tech

 Membrane rafts are highly dynamic heterogeneous sterol- and sphingolipid-rich micro-domains on cell surfaces. They are generally believed to provide residency for cell surface molecules (e.g.,… (more)

Subjects/Keywords: Receptor-ligand binding; FcγRIIA; Phagocytosis; Signaling; Receptor-ligand complexes; Membrane lipids; Immune response; Cellular signal transduction; Cell membranes

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APA (6th Edition):

Tolentino, T. P. (2007). The Roles of Membrane Rafts in CD32A Mediated Formation of a Phagocytic Contact Area. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/16127

Chicago Manual of Style (16th Edition):

Tolentino, Timothy P. “The Roles of Membrane Rafts in CD32A Mediated Formation of a Phagocytic Contact Area.” 2007. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/16127.

MLA Handbook (7th Edition):

Tolentino, Timothy P. “The Roles of Membrane Rafts in CD32A Mediated Formation of a Phagocytic Contact Area.” 2007. Web. 16 Apr 2021.

Vancouver:

Tolentino TP. The Roles of Membrane Rafts in CD32A Mediated Formation of a Phagocytic Contact Area. [Internet] [Doctoral dissertation]. Georgia Tech; 2007. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/16127.

Council of Science Editors:

Tolentino TP. The Roles of Membrane Rafts in CD32A Mediated Formation of a Phagocytic Contact Area. [Doctoral Dissertation]. Georgia Tech; 2007. Available from: http://hdl.handle.net/1853/16127


Georgia Tech

28. Ni, Chih-Wen. Discovery of mechanosensitive microrna and messenger RNA in mouse arterial endothelium and in cultured endothelial cells.

Degree: PhD, Biomedical Engineering, 2010, Georgia Tech

 Atherosclerosis is a major contributor to cardiovascular disease and accounts for an estimated one third of deaths overall. In order to address the hemodynamic components… (more)

Subjects/Keywords: MiRNA; IMAEC; Atherosclerosis; Shear stress; Endothelial cell; Endothelium; Vascular endothelium; Cardiovascular system Diseases; Cardiovascular system Diseases Diagnosis

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APA (6th Edition):

Ni, C. (2010). Discovery of mechanosensitive microrna and messenger RNA in mouse arterial endothelium and in cultured endothelial cells. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/34674

Chicago Manual of Style (16th Edition):

Ni, Chih-Wen. “Discovery of mechanosensitive microrna and messenger RNA in mouse arterial endothelium and in cultured endothelial cells.” 2010. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/34674.

MLA Handbook (7th Edition):

Ni, Chih-Wen. “Discovery of mechanosensitive microrna and messenger RNA in mouse arterial endothelium and in cultured endothelial cells.” 2010. Web. 16 Apr 2021.

Vancouver:

Ni C. Discovery of mechanosensitive microrna and messenger RNA in mouse arterial endothelium and in cultured endothelial cells. [Internet] [Doctoral dissertation]. Georgia Tech; 2010. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/34674.

Council of Science Editors:

Ni C. Discovery of mechanosensitive microrna and messenger RNA in mouse arterial endothelium and in cultured endothelial cells. [Doctoral Dissertation]. Georgia Tech; 2010. Available from: http://hdl.handle.net/1853/34674


Georgia Tech

29. Huang, Jun. A kinetic study of the T cell recognition mechanism.

Degree: PhD, Biomedical Engineering, 2008, Georgia Tech

 The mechanism of T cell recognition is the central but unsolved puzzle of adaptive immunology. The difficulties come from the multichain structure of TCR/CD3, the… (more)

Subjects/Keywords: Recognition; CD8; TCR; PMHC; T cells; Cellular recognition

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APA (6th Edition):

Huang, J. (2008). A kinetic study of the T cell recognition mechanism. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/26693

Chicago Manual of Style (16th Edition):

Huang, Jun. “A kinetic study of the T cell recognition mechanism.” 2008. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/26693.

MLA Handbook (7th Edition):

Huang, Jun. “A kinetic study of the T cell recognition mechanism.” 2008. Web. 16 Apr 2021.

Vancouver:

Huang J. A kinetic study of the T cell recognition mechanism. [Internet] [Doctoral dissertation]. Georgia Tech; 2008. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/26693.

Council of Science Editors:

Huang J. A kinetic study of the T cell recognition mechanism. [Doctoral Dissertation]. Georgia Tech; 2008. Available from: http://hdl.handle.net/1853/26693


Georgia Tech

30. Butcher, Jonathan Talbot. The Effects of Steady Laminar Shear Stress on Aortic Valve Cell Biology.

Degree: PhD, Mechanical Engineering, 2004, Georgia Tech

 Aortic valve disease (AVD) affects millions of people of all ages around the world. Current treatment for AVD consists of valvular replacement with a non-living… (more)

Subjects/Keywords: Microarray; Interstitial cells; Aortic valve; Shear stress; Endothelial cells

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APA (6th Edition):

Butcher, J. T. (2004). The Effects of Steady Laminar Shear Stress on Aortic Valve Cell Biology. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/4824

Chicago Manual of Style (16th Edition):

Butcher, Jonathan Talbot. “The Effects of Steady Laminar Shear Stress on Aortic Valve Cell Biology.” 2004. Doctoral Dissertation, Georgia Tech. Accessed April 16, 2021. http://hdl.handle.net/1853/4824.

MLA Handbook (7th Edition):

Butcher, Jonathan Talbot. “The Effects of Steady Laminar Shear Stress on Aortic Valve Cell Biology.” 2004. Web. 16 Apr 2021.

Vancouver:

Butcher JT. The Effects of Steady Laminar Shear Stress on Aortic Valve Cell Biology. [Internet] [Doctoral dissertation]. Georgia Tech; 2004. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1853/4824.

Council of Science Editors:

Butcher JT. The Effects of Steady Laminar Shear Stress on Aortic Valve Cell Biology. [Doctoral Dissertation]. Georgia Tech; 2004. Available from: http://hdl.handle.net/1853/4824

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