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You searched for +publisher:"Georgia State University" +contributor:("Hang Shi"). Showing records 1 – 9 of 9 total matches.

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Georgia State University

1. Rajan, Anubama. Histone acetylation modulates uncoupling protein1 expression in brown adipocytes.

Degree: MS, Biology, 2014, Georgia State University

  Uncoupling protein 1 (UCP1) is a classical feature of brown adipocytes and understanding its regulatory mechanism will help in the development of a pharmacological… (more)

Subjects/Keywords: Uncoupling protein 1; Brown fat; Epigenetics; Histone deacetylase (HDAC); HDAC inhibitors; Obesity

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APA (6th Edition):

Rajan, A. (2014). Histone acetylation modulates uncoupling protein1 expression in brown adipocytes. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/56

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rajan, Anubama. “Histone acetylation modulates uncoupling protein1 expression in brown adipocytes.” 2014. Thesis, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_theses/56.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rajan, Anubama. “Histone acetylation modulates uncoupling protein1 expression in brown adipocytes.” 2014. Web. 26 Oct 2020.

Vancouver:

Rajan A. Histone acetylation modulates uncoupling protein1 expression in brown adipocytes. [Internet] [Thesis]. Georgia State University; 2014. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_theses/56.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rajan A. Histone acetylation modulates uncoupling protein1 expression in brown adipocytes. [Thesis]. Georgia State University; 2014. Available from: https://scholarworks.gsu.edu/biology_theses/56

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

2. Chen, Yii-Shyuan. Role of DNA Methylation in Adipogenesis.

Degree: MS, Biology, 2014, Georgia State University

  The increase in the prevalence of obesity and obesity-related diseases has caused greater attention to be placed on the molecular mechanisms controlling adipogenesis. In… (more)

Subjects/Keywords: Adipogenesis; DNA Methylation; Osteoblastogenesis; ST2 Cells; Wnt10a; Wnt signaling inhibitor; 3T3-L1 Cells

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APA (6th Edition):

Chen, Y. (2014). Role of DNA Methylation in Adipogenesis. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/57

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Yii-Shyuan. “Role of DNA Methylation in Adipogenesis.” 2014. Thesis, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_theses/57.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Yii-Shyuan. “Role of DNA Methylation in Adipogenesis.” 2014. Web. 26 Oct 2020.

Vancouver:

Chen Y. Role of DNA Methylation in Adipogenesis. [Internet] [Thesis]. Georgia State University; 2014. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_theses/57.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Y. Role of DNA Methylation in Adipogenesis. [Thesis]. Georgia State University; 2014. Available from: https://scholarworks.gsu.edu/biology_theses/57

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

3. Fu, Lizhi. Investigating the Synergistic effect of Leucine-Metformin-Sildenafil on Nonalcoholic Steatohepatitis.

Degree: MS, Biology, 2015, Georgia State University

  Nonalcoholic steatohepatitis (NASH) is a highly prevalent liver disease in the world. About 15% to 25% of patients with NASH may progress to cirrhosis… (more)

Subjects/Keywords: NASH; Leucine-Metformin-Sildenafil; PDE5i and Therapy

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APA (6th Edition):

Fu, L. (2015). Investigating the Synergistic effect of Leucine-Metformin-Sildenafil on Nonalcoholic Steatohepatitis. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/67

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fu, Lizhi. “Investigating the Synergistic effect of Leucine-Metformin-Sildenafil on Nonalcoholic Steatohepatitis.” 2015. Thesis, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_theses/67.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fu, Lizhi. “Investigating the Synergistic effect of Leucine-Metformin-Sildenafil on Nonalcoholic Steatohepatitis.” 2015. Web. 26 Oct 2020.

Vancouver:

Fu L. Investigating the Synergistic effect of Leucine-Metformin-Sildenafil on Nonalcoholic Steatohepatitis. [Internet] [Thesis]. Georgia State University; 2015. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_theses/67.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fu L. Investigating the Synergistic effect of Leucine-Metformin-Sildenafil on Nonalcoholic Steatohepatitis. [Thesis]. Georgia State University; 2015. Available from: https://scholarworks.gsu.edu/biology_theses/67

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

4. Miles-Brown, Jennifer. Microbiota Metabolism of Soluble Fiber Protects Against Low Grade Inflammation and Metabolic Syndrome.

Degree: PhD, Biology, 2016, Georgia State University

  Metabolic syndrome (MetS) is a group of obesity-related metabolic abnormalities that predisposes to type II diabetes mellitus (T2DM) and cardiovascular disease. The dramatic increase… (more)

Subjects/Keywords: Metabolic Syndrome; Obesity; Type 2 Diabetes; Microbiota; Inulin; Cellulose

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APA (6th Edition):

Miles-Brown, J. (2016). Microbiota Metabolism of Soluble Fiber Protects Against Low Grade Inflammation and Metabolic Syndrome. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/184

Chicago Manual of Style (16th Edition):

Miles-Brown, Jennifer. “Microbiota Metabolism of Soluble Fiber Protects Against Low Grade Inflammation and Metabolic Syndrome.” 2016. Doctoral Dissertation, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_diss/184.

MLA Handbook (7th Edition):

Miles-Brown, Jennifer. “Microbiota Metabolism of Soluble Fiber Protects Against Low Grade Inflammation and Metabolic Syndrome.” 2016. Web. 26 Oct 2020.

Vancouver:

Miles-Brown J. Microbiota Metabolism of Soluble Fiber Protects Against Low Grade Inflammation and Metabolic Syndrome. [Internet] [Doctoral dissertation]. Georgia State University; 2016. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_diss/184.

Council of Science Editors:

Miles-Brown J. Microbiota Metabolism of Soluble Fiber Protects Against Low Grade Inflammation and Metabolic Syndrome. [Doctoral Dissertation]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/biology_diss/184


Georgia State University

5. Sharma, Shaligram. EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION.

Degree: MS, Biology, 2017, Georgia State University

  Little is known about the ability of d-δ-tocotrienol to protect against obesity-induced inflammation. These studies were conducted to determine whether d-δ-tocotrienol inhibits highfat diet… (more)

Subjects/Keywords: d-δ-tocotrienol; Anti-inflammatory; Signal Transducer and Activator of Transcription (STAT3)

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APA (6th Edition):

Sharma, S. (2017). EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/79

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharma, Shaligram. “EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION.” 2017. Thesis, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_theses/79.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharma, Shaligram. “EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION.” 2017. Web. 26 Oct 2020.

Vancouver:

Sharma S. EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION. [Internet] [Thesis]. Georgia State University; 2017. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_theses/79.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharma S. EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION. [Thesis]. Georgia State University; 2017. Available from: https://scholarworks.gsu.edu/biology_theses/79

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

6. Schneider, Mary K. Decoding the Sympathetic Nervous System's Regulation of White Adipose Tissue Hyperplasia.

Degree: PhD, Biology, 2017, Georgia State University

  Adipose tissue (AT) expands via both hypertrophy and hyperplasia during the development of obesity. While AT hypertrophy involves the increase in size of existing… (more)

Subjects/Keywords: Proliferation; Adipocyte progenitors; Preadipocytes; Adrenergic receptor; Norepinephrine; Flow cytometry

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APA (6th Edition):

Schneider, M. K. (2017). Decoding the Sympathetic Nervous System's Regulation of White Adipose Tissue Hyperplasia. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/198

Chicago Manual of Style (16th Edition):

Schneider, Mary K. “Decoding the Sympathetic Nervous System's Regulation of White Adipose Tissue Hyperplasia.” 2017. Doctoral Dissertation, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_diss/198.

MLA Handbook (7th Edition):

Schneider, Mary K. “Decoding the Sympathetic Nervous System's Regulation of White Adipose Tissue Hyperplasia.” 2017. Web. 26 Oct 2020.

Vancouver:

Schneider MK. Decoding the Sympathetic Nervous System's Regulation of White Adipose Tissue Hyperplasia. [Internet] [Doctoral dissertation]. Georgia State University; 2017. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_diss/198.

Council of Science Editors:

Schneider MK. Decoding the Sympathetic Nervous System's Regulation of White Adipose Tissue Hyperplasia. [Doctoral Dissertation]. Georgia State University; 2017. Available from: https://scholarworks.gsu.edu/biology_diss/198


Georgia State University

7. Wu, Yang. Cellular And Molecular Insight Into Autonomic Function And Dysfunction.

Degree: PhD, Biology, 2018, Georgia State University

  The autonomic nervous system (ANS) controls several vital functions of the body, especially the autonomic regulation of respiratory and cardiovascular systems. Dysfunction of either… (more)

Subjects/Keywords: Cellular excitation; Rett syndrome; Ventral respiratory column; KATP channels; MicroRNAs; Optogenetics.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, Y. (2018). Cellular And Molecular Insight Into Autonomic Function And Dysfunction. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/206

Chicago Manual of Style (16th Edition):

Wu, Yang. “Cellular And Molecular Insight Into Autonomic Function And Dysfunction.” 2018. Doctoral Dissertation, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_diss/206.

MLA Handbook (7th Edition):

Wu, Yang. “Cellular And Molecular Insight Into Autonomic Function And Dysfunction.” 2018. Web. 26 Oct 2020.

Vancouver:

Wu Y. Cellular And Molecular Insight Into Autonomic Function And Dysfunction. [Internet] [Doctoral dissertation]. Georgia State University; 2018. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_diss/206.

Council of Science Editors:

Wu Y. Cellular And Molecular Insight Into Autonomic Function And Dysfunction. [Doctoral Dissertation]. Georgia State University; 2018. Available from: https://scholarworks.gsu.edu/biology_diss/206


Georgia State University

8. Maganti, Nagini. Role of 26S Proteasome and Regulator of G-Protein Signaling 10 in Regulating Neuroinflammation in the Central Nervous System.

Degree: PhD, Biology, 2015, Georgia State University

  Major histocompatibility complex molecules (MHCII) are cell surface glycoproteins that present extracellular antigens to CD4+ T lymphocytes and initiate adaptive immune responses. Apart from… (more)

Subjects/Keywords: Major Histocompatibility Complex class II; Class II transactivator promoter IV; 26S Proteasome; Ubiquitin Proteasome System; 19S ATPases Sug1; S7; S6a; Regulator of G-protein Signaling 10 (RGS10); Central Nervous System; Neuroinflammation

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APA (6th Edition):

Maganti, N. (2015). Role of 26S Proteasome and Regulator of G-Protein Signaling 10 in Regulating Neuroinflammation in the Central Nervous System. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/162

Chicago Manual of Style (16th Edition):

Maganti, Nagini. “Role of 26S Proteasome and Regulator of G-Protein Signaling 10 in Regulating Neuroinflammation in the Central Nervous System.” 2015. Doctoral Dissertation, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_diss/162.

MLA Handbook (7th Edition):

Maganti, Nagini. “Role of 26S Proteasome and Regulator of G-Protein Signaling 10 in Regulating Neuroinflammation in the Central Nervous System.” 2015. Web. 26 Oct 2020.

Vancouver:

Maganti N. Role of 26S Proteasome and Regulator of G-Protein Signaling 10 in Regulating Neuroinflammation in the Central Nervous System. [Internet] [Doctoral dissertation]. Georgia State University; 2015. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_diss/162.

Council of Science Editors:

Maganti N. Role of 26S Proteasome and Regulator of G-Protein Signaling 10 in Regulating Neuroinflammation in the Central Nervous System. [Doctoral Dissertation]. Georgia State University; 2015. Available from: https://scholarworks.gsu.edu/biology_diss/162

9. Tran, Hao. Microbiota-Immune System Interactions in Diet-Induced Metabolic Syndrome.

Degree: PhD, Biology, 2019, Georgia State University

  Metabolic syndrome (MetS) is the collective term for the interrelated abnormalities associated with obesity. Features of MetS promote a variety of chronic diseases that… (more)

Subjects/Keywords: Intestinal microbiota; Obesity; Immunization; Intestinal Inflammation; Metabolic Syndrome; Fecal metaproteome

…purchased from Jackson Laboratory (Bar Harbor, ME) and maintained at Georgia State… …University, Atlanta, Georgia, USA under institutionally approved protocol under approved protocols… 

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APA (6th Edition):

Tran, H. (2019). Microbiota-Immune System Interactions in Diet-Induced Metabolic Syndrome. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/222

Chicago Manual of Style (16th Edition):

Tran, Hao. “Microbiota-Immune System Interactions in Diet-Induced Metabolic Syndrome.” 2019. Doctoral Dissertation, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_diss/222.

MLA Handbook (7th Edition):

Tran, Hao. “Microbiota-Immune System Interactions in Diet-Induced Metabolic Syndrome.” 2019. Web. 26 Oct 2020.

Vancouver:

Tran H. Microbiota-Immune System Interactions in Diet-Induced Metabolic Syndrome. [Internet] [Doctoral dissertation]. Georgia State University; 2019. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_diss/222.

Council of Science Editors:

Tran H. Microbiota-Immune System Interactions in Diet-Induced Metabolic Syndrome. [Doctoral Dissertation]. Georgia State University; 2019. Available from: https://scholarworks.gsu.edu/biology_diss/222

.