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You searched for +publisher:"Dalhousie University" +contributor:("Jana Sawynok"). Showing records 1 – 12 of 12 total matches.

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Dalhousie University

1. Sardinha, Joel. CANNABINOID 2 RECEPTOR MODULATION IN EXPERIMENTAL MODELS OF SEPSIS.

Degree: MS, Department of Pharmacology, 2014, Dalhousie University

 Sepsis is a critical disease where a dysregulated immune response causes multi-organ dysfunction, leading to organ failure and eventual mortality. Early in the course of… (more)

Subjects/Keywords: Cannabinoids; Microcirculation; Sepsis

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APA (6th Edition):

Sardinha, J. (2014). CANNABINOID 2 RECEPTOR MODULATION IN EXPERIMENTAL MODELS OF SEPSIS. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/54017

Chicago Manual of Style (16th Edition):

Sardinha, Joel. “CANNABINOID 2 RECEPTOR MODULATION IN EXPERIMENTAL MODELS OF SEPSIS.” 2014. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/54017.

MLA Handbook (7th Edition):

Sardinha, Joel. “CANNABINOID 2 RECEPTOR MODULATION IN EXPERIMENTAL MODELS OF SEPSIS.” 2014. Web. 25 Feb 2021.

Vancouver:

Sardinha J. CANNABINOID 2 RECEPTOR MODULATION IN EXPERIMENTAL MODELS OF SEPSIS. [Internet] [Masters thesis]. Dalhousie University; 2014. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/54017.

Council of Science Editors:

Sardinha J. CANNABINOID 2 RECEPTOR MODULATION IN EXPERIMENTAL MODELS OF SEPSIS. [Masters Thesis]. Dalhousie University; 2014. Available from: http://hdl.handle.net/10222/54017


Dalhousie University

2. Ernst, Matthew. CHEMERIN REGULATES ADIPOSITY AND ENERGY HOMEOSTASIS.

Degree: PhD, Department of Pharmacology, 2011, Dalhousie University

 Obesity, characterized by an excess of adipose tissue, is an established risk factor for cardiovascular disease and type II diabetes. Different mechanisms linking obesity with… (more)

Subjects/Keywords: Chemerin; Adipokine; Obesity; Diabetes; Glucose tolerance; insulin resistance

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APA (6th Edition):

Ernst, M. (2011). CHEMERIN REGULATES ADIPOSITY AND ENERGY HOMEOSTASIS. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14305

Chicago Manual of Style (16th Edition):

Ernst, Matthew. “CHEMERIN REGULATES ADIPOSITY AND ENERGY HOMEOSTASIS.” 2011. Doctoral Dissertation, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/14305.

MLA Handbook (7th Edition):

Ernst, Matthew. “CHEMERIN REGULATES ADIPOSITY AND ENERGY HOMEOSTASIS.” 2011. Web. 25 Feb 2021.

Vancouver:

Ernst M. CHEMERIN REGULATES ADIPOSITY AND ENERGY HOMEOSTASIS. [Internet] [Doctoral dissertation]. Dalhousie University; 2011. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/14305.

Council of Science Editors:

Ernst M. CHEMERIN REGULATES ADIPOSITY AND ENERGY HOMEOSTASIS. [Doctoral Dissertation]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14305


Dalhousie University

3. Tweel, Kristin. Competing Influences Of The Tumor Microenvironment On CD26 And The Cancer Phenotype Of Colorectal Carcinoma Cells.

Degree: PhD, Department of Pharmacology, 2013, Dalhousie University

 In Canada, colorectal cancer is the second leading cause of cancer death for both men and women. There are many different factors that contribute to… (more)

Subjects/Keywords: Colorectal Cancer; Metastasis; CD26; Adenosine; MMP-13; CXCL12; Tumor Microenvironment

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APA (6th Edition):

Tweel, K. (2013). Competing Influences Of The Tumor Microenvironment On CD26 And The Cancer Phenotype Of Colorectal Carcinoma Cells. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/21904

Chicago Manual of Style (16th Edition):

Tweel, Kristin. “Competing Influences Of The Tumor Microenvironment On CD26 And The Cancer Phenotype Of Colorectal Carcinoma Cells.” 2013. Doctoral Dissertation, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/21904.

MLA Handbook (7th Edition):

Tweel, Kristin. “Competing Influences Of The Tumor Microenvironment On CD26 And The Cancer Phenotype Of Colorectal Carcinoma Cells.” 2013. Web. 25 Feb 2021.

Vancouver:

Tweel K. Competing Influences Of The Tumor Microenvironment On CD26 And The Cancer Phenotype Of Colorectal Carcinoma Cells. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/21904.

Council of Science Editors:

Tweel K. Competing Influences Of The Tumor Microenvironment On CD26 And The Cancer Phenotype Of Colorectal Carcinoma Cells. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/21904


Dalhousie University

4. Liu, Jean. Further Studies in Adenosinergic and Monoaminergic Mechanisms of Analgesia by Amitriptyline.

Degree: MS, Department of Pharmacology with Neuroscience, 2012, Dalhousie University

 In this thesis, rodent models of chronic pain were used to explore analgesic mechanisms that may potentially be engaged in spinal and peripheral compartments by… (more)

Subjects/Keywords: amitriptyline; persistent post-surgical pain; adenosine A1 receptor; serotonin 5-HT7 receptor; formalin test; antinociception; spared nerve injury; preventive analgesia; antidepressants; pain

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APA (6th Edition):

Liu, J. (2012). Further Studies in Adenosinergic and Monoaminergic Mechanisms of Analgesia by Amitriptyline. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15329

Chicago Manual of Style (16th Edition):

Liu, Jean. “Further Studies in Adenosinergic and Monoaminergic Mechanisms of Analgesia by Amitriptyline.” 2012. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/15329.

MLA Handbook (7th Edition):

Liu, Jean. “Further Studies in Adenosinergic and Monoaminergic Mechanisms of Analgesia by Amitriptyline.” 2012. Web. 25 Feb 2021.

Vancouver:

Liu J. Further Studies in Adenosinergic and Monoaminergic Mechanisms of Analgesia by Amitriptyline. [Internet] [Masters thesis]. Dalhousie University; 2012. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/15329.

Council of Science Editors:

Liu J. Further Studies in Adenosinergic and Monoaminergic Mechanisms of Analgesia by Amitriptyline. [Masters Thesis]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15329


Dalhousie University

5. Gillies, Kelsie. An examination of how Rab GTPases and molecular chaperones influence plasma membrane expression of chemokine receptor dimers.

Degree: MS, Department of Pharmacology, 2013, Dalhousie University

 Signal termination processes of GPCRs are well established, unlike processes that regulate the assembly and intracellular trafficking of these signaling complexes. Bimolecular fluorescence complementation was… (more)

Subjects/Keywords: GPCR; CXCR4; CCR2; Rab GTPase; Anterograde trafficking; Prostate cancer; CD4; CCR5; Molecular chaperone; HIV

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APA (6th Edition):

Gillies, K. (2013). An examination of how Rab GTPases and molecular chaperones influence plasma membrane expression of chemokine receptor dimers. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/39732

Chicago Manual of Style (16th Edition):

Gillies, Kelsie. “An examination of how Rab GTPases and molecular chaperones influence plasma membrane expression of chemokine receptor dimers.” 2013. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/39732.

MLA Handbook (7th Edition):

Gillies, Kelsie. “An examination of how Rab GTPases and molecular chaperones influence plasma membrane expression of chemokine receptor dimers.” 2013. Web. 25 Feb 2021.

Vancouver:

Gillies K. An examination of how Rab GTPases and molecular chaperones influence plasma membrane expression of chemokine receptor dimers. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/39732.

Council of Science Editors:

Gillies K. An examination of how Rab GTPases and molecular chaperones influence plasma membrane expression of chemokine receptor dimers. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/39732


Dalhousie University

6. Wertman, Jaime. An investigation of the role of cytosolic superoxide dismutase (SOD1) on the C-X-C chemokine receptor type 4 (CXCR4)-mediated signal transduction in prostate cancer cells.

Degree: MS, Department of Pharmacology, 2014, Dalhousie University

 Prostate cancer (PCa) is associated with high rates of cancer spread, or metastasis, the process that accounts for up to 90% of cancer-related deaths. CXCR4,… (more)

Subjects/Keywords: Subject Not Available

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APA (6th Edition):

Wertman, J. (2014). An investigation of the role of cytosolic superoxide dismutase (SOD1) on the C-X-C chemokine receptor type 4 (CXCR4)-mediated signal transduction in prostate cancer cells. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/54087

Chicago Manual of Style (16th Edition):

Wertman, Jaime. “An investigation of the role of cytosolic superoxide dismutase (SOD1) on the C-X-C chemokine receptor type 4 (CXCR4)-mediated signal transduction in prostate cancer cells.” 2014. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/54087.

MLA Handbook (7th Edition):

Wertman, Jaime. “An investigation of the role of cytosolic superoxide dismutase (SOD1) on the C-X-C chemokine receptor type 4 (CXCR4)-mediated signal transduction in prostate cancer cells.” 2014. Web. 25 Feb 2021.

Vancouver:

Wertman J. An investigation of the role of cytosolic superoxide dismutase (SOD1) on the C-X-C chemokine receptor type 4 (CXCR4)-mediated signal transduction in prostate cancer cells. [Internet] [Masters thesis]. Dalhousie University; 2014. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/54087.

Council of Science Editors:

Wertman J. An investigation of the role of cytosolic superoxide dismutase (SOD1) on the C-X-C chemokine receptor type 4 (CXCR4)-mediated signal transduction in prostate cancer cells. [Masters Thesis]. Dalhousie University; 2014. Available from: http://hdl.handle.net/10222/54087


Dalhousie University

7. Sun, Michael. Impact of frailty on cardiac contractile function in an aging mouse model.

Degree: MS, Department of Pharmacology, 2014, Dalhousie University

 This study quantified frailty in a longitudinal study in the mouse model and investigated whether frailty was a better predictor of changes in cardiac morphology… (more)

Subjects/Keywords: Cardiovascular; Contraction; Calcium; Echocardiography; Ventricular Myocytes

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APA (6th Edition):

Sun, M. (2014). Impact of frailty on cardiac contractile function in an aging mouse model. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/53991

Chicago Manual of Style (16th Edition):

Sun, Michael. “Impact of frailty on cardiac contractile function in an aging mouse model.” 2014. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/53991.

MLA Handbook (7th Edition):

Sun, Michael. “Impact of frailty on cardiac contractile function in an aging mouse model.” 2014. Web. 25 Feb 2021.

Vancouver:

Sun M. Impact of frailty on cardiac contractile function in an aging mouse model. [Internet] [Masters thesis]. Dalhousie University; 2014. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/53991.

Council of Science Editors:

Sun M. Impact of frailty on cardiac contractile function in an aging mouse model. [Masters Thesis]. Dalhousie University; 2014. Available from: http://hdl.handle.net/10222/53991

8. Robinson, Jessica. Characterizing the Role of Acetylcholinesterase in Mouse Cardiomyoctyte Proliferation and Differentiation.

Degree: MS, Department of Pharmacology, 2013, Dalhousie University

 There is scarce information on the fate of cardiac progenitor cells (CPC) in the embryonic heart after chamber specification. Furthermore, the role of acetylcholinesterase (AChE)… (more)

Subjects/Keywords: Acetylcholinesterase; AChE; AChE-R; readthrough; cardiac conduction; VCS; cardiac cell proliferation; cardiac cell differentiation; cardiac development; conduction cell development; galantamine

Dalhousie University. As a whole, the faculty, staff and students have all assisted me in various… 

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APA (6th Edition):

Robinson, J. (2013). Characterizing the Role of Acetylcholinesterase in Mouse Cardiomyoctyte Proliferation and Differentiation. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/42718

Chicago Manual of Style (16th Edition):

Robinson, Jessica. “Characterizing the Role of Acetylcholinesterase in Mouse Cardiomyoctyte Proliferation and Differentiation.” 2013. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/42718.

MLA Handbook (7th Edition):

Robinson, Jessica. “Characterizing the Role of Acetylcholinesterase in Mouse Cardiomyoctyte Proliferation and Differentiation.” 2013. Web. 25 Feb 2021.

Vancouver:

Robinson J. Characterizing the Role of Acetylcholinesterase in Mouse Cardiomyoctyte Proliferation and Differentiation. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/42718.

Council of Science Editors:

Robinson J. Characterizing the Role of Acetylcholinesterase in Mouse Cardiomyoctyte Proliferation and Differentiation. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/42718

9. Dunlop, Kate Elizabeth. Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury.

Degree: MS, Department of Pharmacology, 2012, Dalhousie University

 The neuroprotective effects of AF4, a flavonoid-enriched extract derived from the peel of Northern Spy apples (containing quercetin-3-O-glucoside, quercetin-3-O-galactoside, quercetin-3-O-rhamnoside, quercetin-3-O-rutinoside, epicatechin, and cyanidin-3-O-galactoside) were… (more)

Subjects/Keywords: Flavonoids; Stroke; Cytoprotective; Anti-inflammatory; Anti-oxidative

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APA (6th Edition):

Dunlop, K. E. (2012). Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15722

Chicago Manual of Style (16th Edition):

Dunlop, Kate Elizabeth. “Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury.” 2012. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/15722.

MLA Handbook (7th Edition):

Dunlop, Kate Elizabeth. “Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury.” 2012. Web. 25 Feb 2021.

Vancouver:

Dunlop KE. Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury. [Internet] [Masters thesis]. Dalhousie University; 2012. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/15722.

Council of Science Editors:

Dunlop KE. Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury. [Masters Thesis]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15722

10. Linton, Patrick. Involvement of Lysophosphatidic Acid in Peripheral Neuropathy and Osteoarthritic Pain in Rats.

Degree: MS, Department of Pharmacology, 2013, Dalhousie University

 A recent study discovered elevated levels of lysophosphatidic acid (LPA) in the synovial fluid of OA patients (Eli Lilly, unpublished). LPA is required for the… (more)

Subjects/Keywords: Subject Not Available

…committee Dr. Jana Sawynok, Dr. Melanie Kelly, and Dr. Eileen Denovan-Wright for all of their… …support and guidance during my studies here at Dalhousie University. I would like to give a… 

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APA (6th Edition):

Linton, P. (2013). Involvement of Lysophosphatidic Acid in Peripheral Neuropathy and Osteoarthritic Pain in Rats. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/35464

Chicago Manual of Style (16th Edition):

Linton, Patrick. “Involvement of Lysophosphatidic Acid in Peripheral Neuropathy and Osteoarthritic Pain in Rats.” 2013. Masters Thesis, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/35464.

MLA Handbook (7th Edition):

Linton, Patrick. “Involvement of Lysophosphatidic Acid in Peripheral Neuropathy and Osteoarthritic Pain in Rats.” 2013. Web. 25 Feb 2021.

Vancouver:

Linton P. Involvement of Lysophosphatidic Acid in Peripheral Neuropathy and Osteoarthritic Pain in Rats. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/35464.

Council of Science Editors:

Linton P. Involvement of Lysophosphatidic Acid in Peripheral Neuropathy and Osteoarthritic Pain in Rats. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/35464

11. Hogel, Matthew. INVESTIGATING THE MECHANISM OF PROMOTER-SPECIFIC N-TERMINAL MUTANT HUNTINGTIN-MEDIATED TRANSCRIPTIONAL DYSREGULATION.

Degree: PhD, Department of Pharmacology with Neuroscience, 2012, Dalhousie University

 Huntington’s disease (HD) is a neurodegenerative disorder caused by the inheritance of one mutant copy of the huntingtin gene. Mutant huntingtin protein (mHtt) contains an… (more)

Subjects/Keywords: Huntington's; Transcription; Repression; Huntingtin; Polyglutamine; Transcriptional Dysregulation; in vitro transcription; N548; ST14A; Dual-luciferase assay; Chromatin Immunoprecipitation; Promoter Deletion; Linker Scanning Mutagenesis; Quantitative PCR; Promoter binding assay; TBP; RAP30

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APA (6th Edition):

Hogel, M. (2012). INVESTIGATING THE MECHANISM OF PROMOTER-SPECIFIC N-TERMINAL MUTANT HUNTINGTIN-MEDIATED TRANSCRIPTIONAL DYSREGULATION. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15723

Chicago Manual of Style (16th Edition):

Hogel, Matthew. “INVESTIGATING THE MECHANISM OF PROMOTER-SPECIFIC N-TERMINAL MUTANT HUNTINGTIN-MEDIATED TRANSCRIPTIONAL DYSREGULATION.” 2012. Doctoral Dissertation, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/15723.

MLA Handbook (7th Edition):

Hogel, Matthew. “INVESTIGATING THE MECHANISM OF PROMOTER-SPECIFIC N-TERMINAL MUTANT HUNTINGTIN-MEDIATED TRANSCRIPTIONAL DYSREGULATION.” 2012. Web. 25 Feb 2021.

Vancouver:

Hogel M. INVESTIGATING THE MECHANISM OF PROMOTER-SPECIFIC N-TERMINAL MUTANT HUNTINGTIN-MEDIATED TRANSCRIPTIONAL DYSREGULATION. [Internet] [Doctoral dissertation]. Dalhousie University; 2012. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/15723.

Council of Science Editors:

Hogel M. INVESTIGATING THE MECHANISM OF PROMOTER-SPECIFIC N-TERMINAL MUTANT HUNTINGTIN-MEDIATED TRANSCRIPTIONAL DYSREGULATION. [Doctoral Dissertation]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15723

12. Hotchkiss, Adam, Gordon. The Effects of Calcium Channel Blockade and Atrial Natriuretic Peptide Signalling on Proliferation and Differentiation of Cardiac Progenitor Cells.

Degree: PhD, Department of Pharmacology, 2013, Dalhousie University

 Cardiac progenitor cells (CPCs) are abundant in the embryonic heart and have hallmark features which include a rapid rate of cell division and the ability… (more)

Subjects/Keywords: Cardiac progenitor cell; proliferation; differentiation; cell transplantation; cardiac development

…members of my advisory committee, Dr. Denis Dupré and Dr. Jana Sawynok as well as our graduate… 

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APA (6th Edition):

Hotchkiss, Adam, G. (2013). The Effects of Calcium Channel Blockade and Atrial Natriuretic Peptide Signalling on Proliferation and Differentiation of Cardiac Progenitor Cells. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/36236

Chicago Manual of Style (16th Edition):

Hotchkiss, Adam, Gordon. “The Effects of Calcium Channel Blockade and Atrial Natriuretic Peptide Signalling on Proliferation and Differentiation of Cardiac Progenitor Cells.” 2013. Doctoral Dissertation, Dalhousie University. Accessed February 25, 2021. http://hdl.handle.net/10222/36236.

MLA Handbook (7th Edition):

Hotchkiss, Adam, Gordon. “The Effects of Calcium Channel Blockade and Atrial Natriuretic Peptide Signalling on Proliferation and Differentiation of Cardiac Progenitor Cells.” 2013. Web. 25 Feb 2021.

Vancouver:

Hotchkiss, Adam G. The Effects of Calcium Channel Blockade and Atrial Natriuretic Peptide Signalling on Proliferation and Differentiation of Cardiac Progenitor Cells. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2021 Feb 25]. Available from: http://hdl.handle.net/10222/36236.

Council of Science Editors:

Hotchkiss, Adam G. The Effects of Calcium Channel Blockade and Atrial Natriuretic Peptide Signalling on Proliferation and Differentiation of Cardiac Progenitor Cells. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/36236

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