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You searched for +publisher:"Dalhousie University" +contributor:("Dr. Roy Duncan"). Showing records 1 – 18 of 18 total matches.

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Dalhousie University

1. Furlong, Suzanne Joy. Thy-1 Signaling in T cells is Weaker and Has Delayed Signaling Kinetics, Promotes Delayed Acquisition and Triggering of Cytotoxic Effector Function, and Preferentially Promotes IL-17A and IL-4 Production in Comparison to TcR Signaling.

Degree: PhD, Department of Microbiology & Immunology, 2012, Dalhousie University

 Thy-1 is a glycosylphosphatidylinositol-anchored protein that is expressed on murine T lymphocytes and is involved in T cell-mediated immune responses. In the presence of costimulatory… (more)

Subjects/Keywords: Signal Transduction; T cells; Dendritic Cells; T cell Activation; Cytokine Production; IL-4; IL-17; Cytotoxic Effector Function

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APA (6th Edition):

Furlong, S. J. (2012). Thy-1 Signaling in T cells is Weaker and Has Delayed Signaling Kinetics, Promotes Delayed Acquisition and Triggering of Cytotoxic Effector Function, and Preferentially Promotes IL-17A and IL-4 Production in Comparison to TcR Signaling. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15491

Chicago Manual of Style (16th Edition):

Furlong, Suzanne Joy. “Thy-1 Signaling in T cells is Weaker and Has Delayed Signaling Kinetics, Promotes Delayed Acquisition and Triggering of Cytotoxic Effector Function, and Preferentially Promotes IL-17A and IL-4 Production in Comparison to TcR Signaling.” 2012. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/15491.

MLA Handbook (7th Edition):

Furlong, Suzanne Joy. “Thy-1 Signaling in T cells is Weaker and Has Delayed Signaling Kinetics, Promotes Delayed Acquisition and Triggering of Cytotoxic Effector Function, and Preferentially Promotes IL-17A and IL-4 Production in Comparison to TcR Signaling.” 2012. Web. 22 Jan 2021.

Vancouver:

Furlong SJ. Thy-1 Signaling in T cells is Weaker and Has Delayed Signaling Kinetics, Promotes Delayed Acquisition and Triggering of Cytotoxic Effector Function, and Preferentially Promotes IL-17A and IL-4 Production in Comparison to TcR Signaling. [Internet] [Doctoral dissertation]. Dalhousie University; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/15491.

Council of Science Editors:

Furlong SJ. Thy-1 Signaling in T cells is Weaker and Has Delayed Signaling Kinetics, Promotes Delayed Acquisition and Triggering of Cytotoxic Effector Function, and Preferentially Promotes IL-17A and IL-4 Production in Comparison to TcR Signaling. [Doctoral Dissertation]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15491


Dalhousie University

2. Cyr, David P. KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE.

Degree: MS, Department of Microbiology & Immunology, 2013, Dalhousie University

 Autophagy (literally to ‘self-eat’) is an intracellular, catabolic mechanism to degrade and recycle cytoplasmic contents in response to metabolic, oxidative, and genotoxic stresses. Autophagy plays… (more)

Subjects/Keywords: KSHV; vGPCR; autophagy; senescence

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APA (6th Edition):

Cyr, D. P. (2013). KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/36316

Chicago Manual of Style (16th Edition):

Cyr, David P. “KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE.” 2013. Masters Thesis, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/36316.

MLA Handbook (7th Edition):

Cyr, David P. “KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE.” 2013. Web. 22 Jan 2021.

Vancouver:

Cyr DP. KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/36316.

Council of Science Editors:

Cyr DP. KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/36316


Dalhousie University

3. Sidik, Saima. TOOLS FOR IDENTIFYING FUNCTIONS OF TYPE III SECRETION SYSTEM EFFECTORS FROM SHIGELLA FLEXNERI.

Degree: MS, Department of Microbiology & Immunology, 2013, Dalhousie University

 Shigellae are pathogenic bacteria that cause the disease shigellosis. Two methods for studying secreted effectors encoded by this pathogen’s virulence plasmid are described. First, protein… (more)

Subjects/Keywords: Shigella; bacteriology; Type III Secretion; Genetic Tools; Ubiquitin; IpaHs

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APA (6th Edition):

Sidik, S. (2013). TOOLS FOR IDENTIFYING FUNCTIONS OF TYPE III SECRETION SYSTEM EFFECTORS FROM SHIGELLA FLEXNERI. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/22282

Chicago Manual of Style (16th Edition):

Sidik, Saima. “TOOLS FOR IDENTIFYING FUNCTIONS OF TYPE III SECRETION SYSTEM EFFECTORS FROM SHIGELLA FLEXNERI.” 2013. Masters Thesis, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/22282.

MLA Handbook (7th Edition):

Sidik, Saima. “TOOLS FOR IDENTIFYING FUNCTIONS OF TYPE III SECRETION SYSTEM EFFECTORS FROM SHIGELLA FLEXNERI.” 2013. Web. 22 Jan 2021.

Vancouver:

Sidik S. TOOLS FOR IDENTIFYING FUNCTIONS OF TYPE III SECRETION SYSTEM EFFECTORS FROM SHIGELLA FLEXNERI. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/22282.

Council of Science Editors:

Sidik S. TOOLS FOR IDENTIFYING FUNCTIONS OF TYPE III SECRETION SYSTEM EFFECTORS FROM SHIGELLA FLEXNERI. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/22282


Dalhousie University

4. Tunis, Matthew C. Investigating the Roles of Mast Cells and Innate Activators in Oral Tolerance.

Degree: PhD, Department of Microbiology & Immunology, 2013, Dalhousie University

 Oral tolerance is the state of immunologic non-responsiveness that is established following oral antigen consumption. Failures of oral tolerance can result in food allergy. The… (more)

Subjects/Keywords: mast cell; T cell; Treg; oral tolerance; TLR2; allergy; peanut; OVA

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APA (6th Edition):

Tunis, M. C. (2013). Investigating the Roles of Mast Cells and Innate Activators in Oral Tolerance. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/36318

Chicago Manual of Style (16th Edition):

Tunis, Matthew C. “Investigating the Roles of Mast Cells and Innate Activators in Oral Tolerance.” 2013. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/36318.

MLA Handbook (7th Edition):

Tunis, Matthew C. “Investigating the Roles of Mast Cells and Innate Activators in Oral Tolerance.” 2013. Web. 22 Jan 2021.

Vancouver:

Tunis MC. Investigating the Roles of Mast Cells and Innate Activators in Oral Tolerance. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/36318.

Council of Science Editors:

Tunis MC. Investigating the Roles of Mast Cells and Innate Activators in Oral Tolerance. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/36318


Dalhousie University

5. Liu, Xinwei. The study on oxysterol-binding protein (OSBP) and related protein 9 (ORP9) ligands: sterols and phosphatidylinositol 4-phosphate.

Degree: MS, Department of Biochemistry & Molecular Biology, 2014, Dalhousie University

 The oxysterol-binding protein OSBP-gene family is composed of 12 members with a common C-terminal sterol-binding domain (SBD). OSBP and ORP9 are members of the family… (more)

Subjects/Keywords: Cholesterol; cholestatrienol; phosphatidylinositol 4-phosphate; oxysterol-binding protein (OSBP); oxysterol-binding protein-related protein 9 (ORP9); sterol transport; Frster resonance energy transfer (FRET)

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APA (6th Edition):

Liu, X. (2014). The study on oxysterol-binding protein (OSBP) and related protein 9 (ORP9) ligands: sterols and phosphatidylinositol 4-phosphate. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/54063

Chicago Manual of Style (16th Edition):

Liu, Xinwei. “The study on oxysterol-binding protein (OSBP) and related protein 9 (ORP9) ligands: sterols and phosphatidylinositol 4-phosphate.” 2014. Masters Thesis, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/54063.

MLA Handbook (7th Edition):

Liu, Xinwei. “The study on oxysterol-binding protein (OSBP) and related protein 9 (ORP9) ligands: sterols and phosphatidylinositol 4-phosphate.” 2014. Web. 22 Jan 2021.

Vancouver:

Liu X. The study on oxysterol-binding protein (OSBP) and related protein 9 (ORP9) ligands: sterols and phosphatidylinositol 4-phosphate. [Internet] [Masters thesis]. Dalhousie University; 2014. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/54063.

Council of Science Editors:

Liu X. The study on oxysterol-binding protein (OSBP) and related protein 9 (ORP9) ligands: sterols and phosphatidylinositol 4-phosphate. [Masters Thesis]. Dalhousie University; 2014. Available from: http://hdl.handle.net/10222/54063


Dalhousie University

6. Racine, Trina. THE AVIAN REOVIRUS TRICISTRONIC S1 mRNA: NEW INSIGHTS INTO CONTROL OF TRANSLATION INITIATION.

Degree: PhD, Department of Microbiology & Immunology, 2010, Dalhousie University

 The S1 genome segment of avian reovirus is functionally tricistronic, encoding three independent protein products (named p10, p17 and ?C) from three sequential, partially overlapping… (more)

Subjects/Keywords: Translation Initiation; Avian Reovirus; S1 mRNA

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APA (6th Edition):

Racine, T. (2010). THE AVIAN REOVIRUS TRICISTRONIC S1 mRNA: NEW INSIGHTS INTO CONTROL OF TRANSLATION INITIATION. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/12842

Chicago Manual of Style (16th Edition):

Racine, Trina. “THE AVIAN REOVIRUS TRICISTRONIC S1 mRNA: NEW INSIGHTS INTO CONTROL OF TRANSLATION INITIATION.” 2010. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/12842.

MLA Handbook (7th Edition):

Racine, Trina. “THE AVIAN REOVIRUS TRICISTRONIC S1 mRNA: NEW INSIGHTS INTO CONTROL OF TRANSLATION INITIATION.” 2010. Web. 22 Jan 2021.

Vancouver:

Racine T. THE AVIAN REOVIRUS TRICISTRONIC S1 mRNA: NEW INSIGHTS INTO CONTROL OF TRANSLATION INITIATION. [Internet] [Doctoral dissertation]. Dalhousie University; 2010. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/12842.

Council of Science Editors:

Racine T. THE AVIAN REOVIRUS TRICISTRONIC S1 mRNA: NEW INSIGHTS INTO CONTROL OF TRANSLATION INITIATION. [Doctoral Dissertation]. Dalhousie University; 2010. Available from: http://hdl.handle.net/10222/12842


Dalhousie University

7. LeBlanc, Marissa. Regulation of Lipid Metabolism and Membrane Trafficking by the Oxysterol Binding Protein Superfamily Member Kes1.

Degree: PhD, Department of Biochemistry & Molecular Biology, 2010, Dalhousie University

 The Saccharomyces cerevisiae oxysterol binding protein homologue Kes1/Osh4 is a member of an enigmatic class of proteins found throughout Eukarya. This family of proteins is… (more)

Subjects/Keywords: lipid; vesicular trafficking; yeast genetics

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APA (6th Edition):

LeBlanc, M. (2010). Regulation of Lipid Metabolism and Membrane Trafficking by the Oxysterol Binding Protein Superfamily Member Kes1. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/13033

Chicago Manual of Style (16th Edition):

LeBlanc, Marissa. “Regulation of Lipid Metabolism and Membrane Trafficking by the Oxysterol Binding Protein Superfamily Member Kes1.” 2010. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/13033.

MLA Handbook (7th Edition):

LeBlanc, Marissa. “Regulation of Lipid Metabolism and Membrane Trafficking by the Oxysterol Binding Protein Superfamily Member Kes1.” 2010. Web. 22 Jan 2021.

Vancouver:

LeBlanc M. Regulation of Lipid Metabolism and Membrane Trafficking by the Oxysterol Binding Protein Superfamily Member Kes1. [Internet] [Doctoral dissertation]. Dalhousie University; 2010. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/13033.

Council of Science Editors:

LeBlanc M. Regulation of Lipid Metabolism and Membrane Trafficking by the Oxysterol Binding Protein Superfamily Member Kes1. [Doctoral Dissertation]. Dalhousie University; 2010. Available from: http://hdl.handle.net/10222/13033


Dalhousie University

8. Lin, Liang-Tzung. HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS.

Degree: PhD, Department of Microbiology & Immunology, 2010, Dalhousie University

 Hepatitis C virus (HCV) is a serious global health problem with an estimate of 170 million carriers worldwide. Most individuals exposed to this blood-borne pathogen… (more)

Subjects/Keywords: HCV; host factors; IRF-3; miR-122; mouse model

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APA (6th Edition):

Lin, L. (2010). HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/13159

Chicago Manual of Style (16th Edition):

Lin, Liang-Tzung. “HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS.” 2010. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/13159.

MLA Handbook (7th Edition):

Lin, Liang-Tzung. “HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS.” 2010. Web. 22 Jan 2021.

Vancouver:

Lin L. HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS. [Internet] [Doctoral dissertation]. Dalhousie University; 2010. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/13159.

Council of Science Editors:

Lin L. HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS. [Doctoral Dissertation]. Dalhousie University; 2010. Available from: http://hdl.handle.net/10222/13159


Dalhousie University

9. Al-Afif, Ayham. HUMAN MAST CELL RESPONSES TO RESPIRATORY SYNCYTIAL VIRUS.

Degree: MS, Department of Microbiology & Immunology, 2012, Dalhousie University

 Mast cells reside at tissue sites closely interfacing the environment and play a role in host defense against pathogens. Mast cell responses to respiratory syncytial… (more)

Subjects/Keywords: Virus; Inflammation; Immuology; Microbiology

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APA (6th Edition):

Al-Afif, A. (2012). HUMAN MAST CELL RESPONSES TO RESPIRATORY SYNCYTIAL VIRUS. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15709

Chicago Manual of Style (16th Edition):

Al-Afif, Ayham. “HUMAN MAST CELL RESPONSES TO RESPIRATORY SYNCYTIAL VIRUS.” 2012. Masters Thesis, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/15709.

MLA Handbook (7th Edition):

Al-Afif, Ayham. “HUMAN MAST CELL RESPONSES TO RESPIRATORY SYNCYTIAL VIRUS.” 2012. Web. 22 Jan 2021.

Vancouver:

Al-Afif A. HUMAN MAST CELL RESPONSES TO RESPIRATORY SYNCYTIAL VIRUS. [Internet] [Masters thesis]. Dalhousie University; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/15709.

Council of Science Editors:

Al-Afif A. HUMAN MAST CELL RESPONSES TO RESPIRATORY SYNCYTIAL VIRUS. [Masters Thesis]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15709


Dalhousie University

10. Benjamin, Jeremy. Functions of the Yeast GTPase-Activating Proteins Age1 and Gcs1 for Post-Golgi Vesicular Transport.

Degree: PhD, Department of Microbiology & Immunology, 2011, Dalhousie University

 Organelles within the endomembrane system of all eukaryotic cells exchange membrane lipids and proteins using membrane-bound transport vesicles. This highly conserved vesicular transport process is… (more)

Subjects/Keywords: Yeast; vesicular transport; Arf; GAP; Gcs1; Age1; Arl

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APA (6th Edition):

Benjamin, J. (2011). Functions of the Yeast GTPase-Activating Proteins Age1 and Gcs1 for Post-Golgi Vesicular Transport. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14218

Chicago Manual of Style (16th Edition):

Benjamin, Jeremy. “Functions of the Yeast GTPase-Activating Proteins Age1 and Gcs1 for Post-Golgi Vesicular Transport.” 2011. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/14218.

MLA Handbook (7th Edition):

Benjamin, Jeremy. “Functions of the Yeast GTPase-Activating Proteins Age1 and Gcs1 for Post-Golgi Vesicular Transport.” 2011. Web. 22 Jan 2021.

Vancouver:

Benjamin J. Functions of the Yeast GTPase-Activating Proteins Age1 and Gcs1 for Post-Golgi Vesicular Transport. [Internet] [Doctoral dissertation]. Dalhousie University; 2011. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/14218.

Council of Science Editors:

Benjamin J. Functions of the Yeast GTPase-Activating Proteins Age1 and Gcs1 for Post-Golgi Vesicular Transport. [Doctoral Dissertation]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14218


Dalhousie University

11. Langelaan, David. Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks.

Degree: PhD, Department of Biochemistry & Molecular Biology, 2013, Dalhousie University

 Apelin, the endogenous ligand to the apelin receptor, is a small peptide involved with cardiovascular regulation. Using nuclear magnetic resonance (NMR) spectroscopy, I demonstrate that… (more)

Subjects/Keywords: G-protein coupled receptor; Nuclear magnetic resonance spectroscopy; Apelin

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APA (6th Edition):

Langelaan, D. (2013). Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/22285

Chicago Manual of Style (16th Edition):

Langelaan, David. “Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks.” 2013. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/22285.

MLA Handbook (7th Edition):

Langelaan, David. “Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks.” 2013. Web. 22 Jan 2021.

Vancouver:

Langelaan D. Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/22285.

Council of Science Editors:

Langelaan D. Structural Studies Of Apelin And Its Receptor As Well As The Characteristics And Causes Of Membrane Protein Helix Kinks. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/22285


Dalhousie University

12. Leidal, Andrew Michael. Dynamic Interplay Between Kaposi's Sarcoma-Associated Herpesvirus Latent Proteins in the Control of Oncogene-Induced Senescence.

Degree: PhD, Department of Microbiology & Immunology, 2012, Dalhousie University

 Acute oncogenic stress can activate autophagy and facilitate permanent arrest of the cell cycle through a failsafe mechanism known as oncogene-induced senescence (OIS). Kaposi's sarcoma-associated… (more)

Subjects/Keywords: p53; oncogene-induced senescence; autophagy; Kaposi's sarcoma-associated herpesvirus (KSHV); Kaposi's sarcoma; oncovirus; DNA damage

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APA (6th Edition):

Leidal, A. M. (2012). Dynamic Interplay Between Kaposi's Sarcoma-Associated Herpesvirus Latent Proteins in the Control of Oncogene-Induced Senescence. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14813

Chicago Manual of Style (16th Edition):

Leidal, Andrew Michael. “Dynamic Interplay Between Kaposi's Sarcoma-Associated Herpesvirus Latent Proteins in the Control of Oncogene-Induced Senescence.” 2012. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/14813.

MLA Handbook (7th Edition):

Leidal, Andrew Michael. “Dynamic Interplay Between Kaposi's Sarcoma-Associated Herpesvirus Latent Proteins in the Control of Oncogene-Induced Senescence.” 2012. Web. 22 Jan 2021.

Vancouver:

Leidal AM. Dynamic Interplay Between Kaposi's Sarcoma-Associated Herpesvirus Latent Proteins in the Control of Oncogene-Induced Senescence. [Internet] [Doctoral dissertation]. Dalhousie University; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/14813.

Council of Science Editors:

Leidal AM. Dynamic Interplay Between Kaposi's Sarcoma-Associated Herpesvirus Latent Proteins in the Control of Oncogene-Induced Senescence. [Doctoral Dissertation]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/14813


Dalhousie University

13. Hulbah, Maram. Development of An Antibiotic Marker-Free Gene Delivery System in Streptococcus gordonii.

Degree: MS, Department of Microbiology & Immunology, 2013, Dalhousie University

 Streptococcus gordonii, a commensal oral bacterium, is considered a good candidate to function as a live oral vaccine vector. The introduction of vaccine antigen genes… (more)

Subjects/Keywords: Streptococcus gordonii; Auxotrophic complementation; live oral vaccine; selectable markers; antibiotic resistance genes

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APA (6th Edition):

Hulbah, M. (2013). Development of An Antibiotic Marker-Free Gene Delivery System in Streptococcus gordonii. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/21770

Chicago Manual of Style (16th Edition):

Hulbah, Maram. “Development of An Antibiotic Marker-Free Gene Delivery System in Streptococcus gordonii.” 2013. Masters Thesis, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/21770.

MLA Handbook (7th Edition):

Hulbah, Maram. “Development of An Antibiotic Marker-Free Gene Delivery System in Streptococcus gordonii.” 2013. Web. 22 Jan 2021.

Vancouver:

Hulbah M. Development of An Antibiotic Marker-Free Gene Delivery System in Streptococcus gordonii. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/21770.

Council of Science Editors:

Hulbah M. Development of An Antibiotic Marker-Free Gene Delivery System in Streptococcus gordonii. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/21770


Dalhousie University

14. Garant, Katy. Oncolytic Reovirus Induces Intracellular Redistribution of Ras to Promote Apoptosis and Progeny Virus Release.

Degree: PhD, Department of Microbiology & Immunology, 2014, Dalhousie University

 Ras is a dynamic protein capable of interacting with plasma membrane microdomains and intracellular membranes. Its activity, as controlled by post-translational modifications, is essential in… (more)

Subjects/Keywords: Reovirus; Ras; Cancer Biology; Oncolytic Virus; Palmitoylation; Apoptosis

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APA (6th Edition):

Garant, K. (2014). Oncolytic Reovirus Induces Intracellular Redistribution of Ras to Promote Apoptosis and Progeny Virus Release. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/54002

Chicago Manual of Style (16th Edition):

Garant, Katy. “Oncolytic Reovirus Induces Intracellular Redistribution of Ras to Promote Apoptosis and Progeny Virus Release.” 2014. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/54002.

MLA Handbook (7th Edition):

Garant, Katy. “Oncolytic Reovirus Induces Intracellular Redistribution of Ras to Promote Apoptosis and Progeny Virus Release.” 2014. Web. 22 Jan 2021.

Vancouver:

Garant K. Oncolytic Reovirus Induces Intracellular Redistribution of Ras to Promote Apoptosis and Progeny Virus Release. [Internet] [Doctoral dissertation]. Dalhousie University; 2014. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/54002.

Council of Science Editors:

Garant K. Oncolytic Reovirus Induces Intracellular Redistribution of Ras to Promote Apoptosis and Progeny Virus Release. [Doctoral Dissertation]. Dalhousie University; 2014. Available from: http://hdl.handle.net/10222/54002

15. Pan, Da. Molecular Mechanisms of Reovirus Oncolysis.

Degree: PhD, Department of Microbiology & Immunology, 2013, Dalhousie University

 Mammalian reovirus is a naturally benign virus that preferentially replicates in cancer cells (reovirus oncolysis) and has been tested as a potential cancer therapy in… (more)

Subjects/Keywords: Reovirus; oncolysis; p53; Ras

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pan, D. (2013). Molecular Mechanisms of Reovirus Oncolysis. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15960

Chicago Manual of Style (16th Edition):

Pan, Da. “Molecular Mechanisms of Reovirus Oncolysis.” 2013. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/15960.

MLA Handbook (7th Edition):

Pan, Da. “Molecular Mechanisms of Reovirus Oncolysis.” 2013. Web. 22 Jan 2021.

Vancouver:

Pan D. Molecular Mechanisms of Reovirus Oncolysis. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/15960.

Council of Science Editors:

Pan D. Molecular Mechanisms of Reovirus Oncolysis. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/15960

16. Hilchie, Ashley. Studies on the anticancer properties of pleurocidins: a preclinical evaluation.

Degree: PhD, Department of Microbiology & Immunology, 2012, Dalhousie University

 Cationic antimicrobial peptides (CAPs) are small peptides that constitute an important defence against microbial pathogens. Certain CAPs also possess anticancer properties. NRC-03 and NRC-07 are… (more)

Subjects/Keywords: cationic antimicrobial peptide; breast cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hilchie, A. (2012). Studies on the anticancer properties of pleurocidins: a preclinical evaluation. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15713

Chicago Manual of Style (16th Edition):

Hilchie, Ashley. “Studies on the anticancer properties of pleurocidins: a preclinical evaluation.” 2012. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/15713.

MLA Handbook (7th Edition):

Hilchie, Ashley. “Studies on the anticancer properties of pleurocidins: a preclinical evaluation.” 2012. Web. 22 Jan 2021.

Vancouver:

Hilchie A. Studies on the anticancer properties of pleurocidins: a preclinical evaluation. [Internet] [Doctoral dissertation]. Dalhousie University; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/15713.

Council of Science Editors:

Hilchie A. Studies on the anticancer properties of pleurocidins: a preclinical evaluation. [Doctoral Dissertation]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15713

17. Pan, Luzhe. THE P53-NMNAT2 FEEDBACK REGULATORY LOOP: MECHANISMS UNDERLYING P53 ACTIVATION.

Degree: PhD, Department of Microbiology & Immunology, 2012, Dalhousie University

 The tumor suppressor p53 acts as a master transcription factor that controls hundreds of effecter genes in response to various cellular stresses. The flexibility of… (more)

Subjects/Keywords: P53; NMNAT2; FEEDBACK; ACTIVATION

…grateful to my supervisory committee: Dr. Roy Duncan, Dr. Rick Singer, Dr. David Waisman and Dr… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pan, L. (2012). THE P53-NMNAT2 FEEDBACK REGULATORY LOOP: MECHANISMS UNDERLYING P53 ACTIVATION. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15717

Chicago Manual of Style (16th Edition):

Pan, Luzhe. “THE P53-NMNAT2 FEEDBACK REGULATORY LOOP: MECHANISMS UNDERLYING P53 ACTIVATION.” 2012. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/15717.

MLA Handbook (7th Edition):

Pan, Luzhe. “THE P53-NMNAT2 FEEDBACK REGULATORY LOOP: MECHANISMS UNDERLYING P53 ACTIVATION.” 2012. Web. 22 Jan 2021.

Vancouver:

Pan L. THE P53-NMNAT2 FEEDBACK REGULATORY LOOP: MECHANISMS UNDERLYING P53 ACTIVATION. [Internet] [Doctoral dissertation]. Dalhousie University; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/15717.

Council of Science Editors:

Pan L. THE P53-NMNAT2 FEEDBACK REGULATORY LOOP: MECHANISMS UNDERLYING P53 ACTIVATION. [Doctoral Dissertation]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15717

18. Nasrallah, Gheyath K. Potential roles of the chaperonin (HtpB), polyamines, and the polyamine binding protein (PotD) in Legionella pneumophila pathogenesis.

Degree: PhD, Department of Microbiology & Immunology, 2011, Dalhousie University

 The intracellular pathogen Legionella pneumophila replicates in a membrane-bound compartment known as the Legionella-containing vacuole (LCV) where it abundantly releases its chaperonin HtpB, suggesting that… (more)

Subjects/Keywords: Legionella; chaperonin; polyamines; intracellular growth

Dr. Roy Duncan, for their invaluable help and guidance over the past few years. I would… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nasrallah, G. K. (2011). Potential roles of the chaperonin (HtpB), polyamines, and the polyamine binding protein (PotD) in Legionella pneumophila pathogenesis. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14200

Chicago Manual of Style (16th Edition):

Nasrallah, Gheyath K. “Potential roles of the chaperonin (HtpB), polyamines, and the polyamine binding protein (PotD) in Legionella pneumophila pathogenesis.” 2011. Doctoral Dissertation, Dalhousie University. Accessed January 22, 2021. http://hdl.handle.net/10222/14200.

MLA Handbook (7th Edition):

Nasrallah, Gheyath K. “Potential roles of the chaperonin (HtpB), polyamines, and the polyamine binding protein (PotD) in Legionella pneumophila pathogenesis.” 2011. Web. 22 Jan 2021.

Vancouver:

Nasrallah GK. Potential roles of the chaperonin (HtpB), polyamines, and the polyamine binding protein (PotD) in Legionella pneumophila pathogenesis. [Internet] [Doctoral dissertation]. Dalhousie University; 2011. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10222/14200.

Council of Science Editors:

Nasrallah GK. Potential roles of the chaperonin (HtpB), polyamines, and the polyamine binding protein (PotD) in Legionella pneumophila pathogenesis. [Doctoral Dissertation]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14200

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