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You searched for +publisher:"Cornell University" +contributor:("Denkers, Eric Young"). Showing records 1 – 12 of 12 total matches.

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Cornell University

1. Mahamed, Deeqa. The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology.

Degree: PhD, Immunology, 2013, Cornell University

 The protozoan pathogen Toxoplasma gondii is a highly successful parasite that infects up to a third of the world's population, causing morbidity and mortality in… (more)

Subjects/Keywords: Toxoplasma gondii; Extracellular Adenosine; CD73

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APA (6th Edition):

Mahamed, D. (2013). The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33839

Chicago Manual of Style (16th Edition):

Mahamed, Deeqa. “The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology.” 2013. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/33839.

MLA Handbook (7th Edition):

Mahamed, Deeqa. “The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology.” 2013. Web. 06 Mar 2021.

Vancouver:

Mahamed D. The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/33839.

Council of Science Editors:

Mahamed D. The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33839


Cornell University

2. Mohinta, Sonia. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.

Degree: PhD, Immunology, 2014, Cornell University

 A number of autoimmune and chronic inflammatory diseases are related to environmental stress. Aryl hydrocarbon receptor (AHR) is regarded as an environmental sensor integrating immune… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Th17; Tcell differentiation

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APA (6th Edition):

Mohinta, S. (2014). Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/36177

Chicago Manual of Style (16th Edition):

Mohinta, Sonia. “Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.” 2014. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/36177.

MLA Handbook (7th Edition):

Mohinta, Sonia. “Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.” 2014. Web. 06 Mar 2021.

Vancouver:

Mohinta S. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/36177.

Council of Science Editors:

Mohinta S. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36177


Cornell University

3. Elmore, Sara. Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection.

Degree: PhD, Immunology, 2014, Cornell University

 The immune response to an invading pathogen must be tightly regulated so as to combat the infection while avoiding immune-mediated pathologies. This requires the coordinated… (more)

Subjects/Keywords: Toxoplasma gondii; Immunity; Infection

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APA (6th Edition):

Elmore, S. (2014). Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/38921

Chicago Manual of Style (16th Edition):

Elmore, Sara. “Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection.” 2014. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/38921.

MLA Handbook (7th Edition):

Elmore, Sara. “Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection.” 2014. Web. 06 Mar 2021.

Vancouver:

Elmore S. Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/38921.

Council of Science Editors:

Elmore S. Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/38921


Cornell University

4. Abi Abdallah, Delbert. Exploring The Multiple Functions Of Neutrophils.

Degree: PhD, Immunology, 2012, Cornell University

 Neutrophils are one of the most effective antimicrobial cells in the innate immune system. They arrive at a site of infection or inflammation in response… (more)

Subjects/Keywords: Neutrophils; Toxoplasma gondii; Neutrophil extracellular traps; Neutrophil antigen presentation

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APA (6th Edition):

Abi Abdallah, D. (2012). Exploring The Multiple Functions Of Neutrophils. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/29282

Chicago Manual of Style (16th Edition):

Abi Abdallah, Delbert. “Exploring The Multiple Functions Of Neutrophils.” 2012. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/29282.

MLA Handbook (7th Edition):

Abi Abdallah, Delbert. “Exploring The Multiple Functions Of Neutrophils.” 2012. Web. 06 Mar 2021.

Vancouver:

Abi Abdallah D. Exploring The Multiple Functions Of Neutrophils. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/29282.

Council of Science Editors:

Abi Abdallah D. Exploring The Multiple Functions Of Neutrophils. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29282


Cornell University

5. Blum, Lisa. Induction Of The Intestinal Th2 Immune Response To The Parasitic Nematode Trichinella Spiralis: Rapid Expulsion, Distinct Regional Immunity, And The Role Of Innate Mediators.

Degree: PhD, Immunology, 2012, Cornell University

 Intestinal helminths infect over 2 billion people worldwide, and the mechanisms by which immunity to these pathogens is induced are not well-understood. Trichinella spiralis exhibits… (more)

Subjects/Keywords: Trichinella spiralis; mast cells; interleukin-33

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APA (6th Edition):

Blum, L. (2012). Induction Of The Intestinal Th2 Immune Response To The Parasitic Nematode Trichinella Spiralis: Rapid Expulsion, Distinct Regional Immunity, And The Role Of Innate Mediators. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/29395

Chicago Manual of Style (16th Edition):

Blum, Lisa. “Induction Of The Intestinal Th2 Immune Response To The Parasitic Nematode Trichinella Spiralis: Rapid Expulsion, Distinct Regional Immunity, And The Role Of Innate Mediators.” 2012. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/29395.

MLA Handbook (7th Edition):

Blum, Lisa. “Induction Of The Intestinal Th2 Immune Response To The Parasitic Nematode Trichinella Spiralis: Rapid Expulsion, Distinct Regional Immunity, And The Role Of Innate Mediators.” 2012. Web. 06 Mar 2021.

Vancouver:

Blum L. Induction Of The Intestinal Th2 Immune Response To The Parasitic Nematode Trichinella Spiralis: Rapid Expulsion, Distinct Regional Immunity, And The Role Of Innate Mediators. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/29395.

Council of Science Editors:

Blum L. Induction Of The Intestinal Th2 Immune Response To The Parasitic Nematode Trichinella Spiralis: Rapid Expulsion, Distinct Regional Immunity, And The Role Of Innate Mediators. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29395


Cornell University

6. Lin, Changyou. Interaction Between Cytolethal Distending Toxin Produced By Campylobacter Jejuni And Helicobacter Hepaticus And Host Innate Immune Defense.

Degree: PhD, Immunology, 2014, Cornell University

 Cytolethal distending toxin (CDT) of C. jejuni, a leading cause of intestinal illness worldwide, and H. hepaticus, a laboratory mice pathogen causes cell cycle arrest… (more)

Subjects/Keywords: Cytolethal Distending Toxin; Neutrophil Extracellular Trap; Cellular senescence

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APA (6th Edition):

Lin, C. (2014). Interaction Between Cytolethal Distending Toxin Produced By Campylobacter Jejuni And Helicobacter Hepaticus And Host Innate Immune Defense. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/36062

Chicago Manual of Style (16th Edition):

Lin, Changyou. “Interaction Between Cytolethal Distending Toxin Produced By Campylobacter Jejuni And Helicobacter Hepaticus And Host Innate Immune Defense.” 2014. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/36062.

MLA Handbook (7th Edition):

Lin, Changyou. “Interaction Between Cytolethal Distending Toxin Produced By Campylobacter Jejuni And Helicobacter Hepaticus And Host Innate Immune Defense.” 2014. Web. 06 Mar 2021.

Vancouver:

Lin C. Interaction Between Cytolethal Distending Toxin Produced By Campylobacter Jejuni And Helicobacter Hepaticus And Host Innate Immune Defense. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/36062.

Council of Science Editors:

Lin C. Interaction Between Cytolethal Distending Toxin Produced By Campylobacter Jejuni And Helicobacter Hepaticus And Host Innate Immune Defense. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36062


Cornell University

7. Cullender, Tyler. Tlr5 And Anti-Flagellin Iga Are Critical Elements To Quenching Bacterial Motility In The Mammalian Gut.

Degree: PhD, Microbiology, 2013, Cornell University

 The mammalian gut contains an incredibly diverse and abundant bacterial community that makes essential contributions to host metabolism and immune system priming. The close proximity… (more)

Subjects/Keywords: TLR5; IgA; flagellin

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APA (6th Edition):

Cullender, T. (2013). Tlr5 And Anti-Flagellin Iga Are Critical Elements To Quenching Bacterial Motility In The Mammalian Gut. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/34086

Chicago Manual of Style (16th Edition):

Cullender, Tyler. “Tlr5 And Anti-Flagellin Iga Are Critical Elements To Quenching Bacterial Motility In The Mammalian Gut.” 2013. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/34086.

MLA Handbook (7th Edition):

Cullender, Tyler. “Tlr5 And Anti-Flagellin Iga Are Critical Elements To Quenching Bacterial Motility In The Mammalian Gut.” 2013. Web. 06 Mar 2021.

Vancouver:

Cullender T. Tlr5 And Anti-Flagellin Iga Are Critical Elements To Quenching Bacterial Motility In The Mammalian Gut. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/34086.

Council of Science Editors:

Cullender T. Tlr5 And Anti-Flagellin Iga Are Critical Elements To Quenching Bacterial Motility In The Mammalian Gut. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/34086


Cornell University

8. Craven, Melanie. Comparative Analysis Of Host-Bacterial Interactions In The Gastrointestinal Tract.

Degree: PhD, Veterinary Medicine, 2011, Cornell University

 Technological advances in culture-independent methods using high throughput DNA sequencing have revolutionized our understanding of gastrointestinal (GI) microbiology. Our ability to now characterize the intestinal… (more)

Subjects/Keywords: Dysbiosis; inflammatory bowel disease

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APA (6th Edition):

Craven, M. (2011). Comparative Analysis Of Host-Bacterial Interactions In The Gastrointestinal Tract. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33572

Chicago Manual of Style (16th Edition):

Craven, Melanie. “Comparative Analysis Of Host-Bacterial Interactions In The Gastrointestinal Tract.” 2011. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/33572.

MLA Handbook (7th Edition):

Craven, Melanie. “Comparative Analysis Of Host-Bacterial Interactions In The Gastrointestinal Tract.” 2011. Web. 06 Mar 2021.

Vancouver:

Craven M. Comparative Analysis Of Host-Bacterial Interactions In The Gastrointestinal Tract. [Internet] [Doctoral dissertation]. Cornell University; 2011. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/33572.

Council of Science Editors:

Craven M. Comparative Analysis Of Host-Bacterial Interactions In The Gastrointestinal Tract. [Doctoral Dissertation]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/33572


Cornell University

9. Huang, Lu. Versatility Of Eosinophils In Immunity To The Parasitic Nematode, Trichinella Spiralis.

Degree: PhD, Immunology, 2014, Cornell University

 Helminth infections continue to threaten human and animal health worldwide. Dissection of the interactions between helminths and helminth-induced Th2 immunity is necessary for the development… (more)

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APA (6th Edition):

Huang, L. (2014). Versatility Of Eosinophils In Immunity To The Parasitic Nematode, Trichinella Spiralis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/37094

Chicago Manual of Style (16th Edition):

Huang, Lu. “Versatility Of Eosinophils In Immunity To The Parasitic Nematode, Trichinella Spiralis.” 2014. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/37094.

MLA Handbook (7th Edition):

Huang, Lu. “Versatility Of Eosinophils In Immunity To The Parasitic Nematode, Trichinella Spiralis.” 2014. Web. 06 Mar 2021.

Vancouver:

Huang L. Versatility Of Eosinophils In Immunity To The Parasitic Nematode, Trichinella Spiralis. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/37094.

Council of Science Editors:

Huang L. Versatility Of Eosinophils In Immunity To The Parasitic Nematode, Trichinella Spiralis. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/37094


Cornell University

10. Schneider, Anne. Unraveling The Paradoxical Impacts Of Toxoplasma Gondii On Dendritic Cell Stat1 Signaling.

Degree: PhD, Veterinary Medicine, 2013, Cornell University

 Toxoplasma gondii is a protozoan parasite of wide geographic and host range. Although many infections in humans are asymptomatic, the potential exists for severe disease.… (more)

Subjects/Keywords: toxoplasma; dendritic cell; stat1

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APA (6th Edition):

Schneider, A. (2013). Unraveling The Paradoxical Impacts Of Toxoplasma Gondii On Dendritic Cell Stat1 Signaling. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33973

Chicago Manual of Style (16th Edition):

Schneider, Anne. “Unraveling The Paradoxical Impacts Of Toxoplasma Gondii On Dendritic Cell Stat1 Signaling.” 2013. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/33973.

MLA Handbook (7th Edition):

Schneider, Anne. “Unraveling The Paradoxical Impacts Of Toxoplasma Gondii On Dendritic Cell Stat1 Signaling.” 2013. Web. 06 Mar 2021.

Vancouver:

Schneider A. Unraveling The Paradoxical Impacts Of Toxoplasma Gondii On Dendritic Cell Stat1 Signaling. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/33973.

Council of Science Editors:

Schneider A. Unraveling The Paradoxical Impacts Of Toxoplasma Gondii On Dendritic Cell Stat1 Signaling. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33973


Cornell University

11. Ju, Chia-Hsin. Innate Immunity Of The Intestinal Epithelium During Toxoplasma Gondii Infection.

Degree: PhD, Veterinary Medicine, 2011, Cornell University

 The single layer of epithelial cells that line the intestinal tract provides both a physiologic and immunologic barrier for microbes and foreign antigens. Intestinal epithelial… (more)

Subjects/Keywords: Innate immunity; Intestinal epithelial cells; Toxoplasma gondii

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APA (6th Edition):

Ju, C. (2011). Innate Immunity Of The Intestinal Epithelium During Toxoplasma Gondii Infection. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33498

Chicago Manual of Style (16th Edition):

Ju, Chia-Hsin. “Innate Immunity Of The Intestinal Epithelium During Toxoplasma Gondii Infection.” 2011. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/33498.

MLA Handbook (7th Edition):

Ju, Chia-Hsin. “Innate Immunity Of The Intestinal Epithelium During Toxoplasma Gondii Infection.” 2011. Web. 06 Mar 2021.

Vancouver:

Ju C. Innate Immunity Of The Intestinal Epithelium During Toxoplasma Gondii Infection. [Internet] [Doctoral dissertation]. Cornell University; 2011. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/33498.

Council of Science Editors:

Ju C. Innate Immunity Of The Intestinal Epithelium During Toxoplasma Gondii Infection. [Doctoral Dissertation]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/33498


Cornell University

12. Harbison, Carole. Receptor Binding And Early Steps Of Cellular Infection By Parvoviruses.

Degree: PhD, Veterinary Medicine, 2011, Cornell University

 Parvoviruses are small, non-enveloped, single stranded DNA viruses. The specific attributes of the capsid and genome dictate how the virus interacts with the environment and… (more)

Subjects/Keywords: Virus Entry; Virus Evolution; Anti-viral Immunity

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APA (6th Edition):

Harbison, C. (2011). Receptor Binding And Early Steps Of Cellular Infection By Parvoviruses. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/29255

Chicago Manual of Style (16th Edition):

Harbison, Carole. “Receptor Binding And Early Steps Of Cellular Infection By Parvoviruses.” 2011. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/29255.

MLA Handbook (7th Edition):

Harbison, Carole. “Receptor Binding And Early Steps Of Cellular Infection By Parvoviruses.” 2011. Web. 06 Mar 2021.

Vancouver:

Harbison C. Receptor Binding And Early Steps Of Cellular Infection By Parvoviruses. [Internet] [Doctoral dissertation]. Cornell University; 2011. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/29255.

Council of Science Editors:

Harbison C. Receptor Binding And Early Steps Of Cellular Infection By Parvoviruses. [Doctoral Dissertation]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/29255

.