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You searched for +publisher:"Cornell University" +contributor:("August, Avery"). Showing records 1 – 14 of 14 total matches.

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Cornell University

1. Solouki, Sabrina. T CELL RECEPTOR SIGNAL STRENGTH AND ANTIGEN AFFINITY DIFFERENTIALLY REGULATE CD8+ MEMORY T CELL DEVELOPMENT.

Degree: PhD, Biomedical and Biological Sciences, 2020, Cornell University

 CD8+ T cells play a critical role in adaptive immunity by maintaining the ability to differentiate into CD8+ memory T cells, which provide the basis… (more)

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APA (6th Edition):

Solouki, S. (2020). T CELL RECEPTOR SIGNAL STRENGTH AND ANTIGEN AFFINITY DIFFERENTIALLY REGULATE CD8+ MEMORY T CELL DEVELOPMENT. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/70427

Chicago Manual of Style (16th Edition):

Solouki, Sabrina. “T CELL RECEPTOR SIGNAL STRENGTH AND ANTIGEN AFFINITY DIFFERENTIALLY REGULATE CD8+ MEMORY T CELL DEVELOPMENT.” 2020. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/70427.

MLA Handbook (7th Edition):

Solouki, Sabrina. “T CELL RECEPTOR SIGNAL STRENGTH AND ANTIGEN AFFINITY DIFFERENTIALLY REGULATE CD8+ MEMORY T CELL DEVELOPMENT.” 2020. Web. 28 Feb 2021.

Vancouver:

Solouki S. T CELL RECEPTOR SIGNAL STRENGTH AND ANTIGEN AFFINITY DIFFERENTIALLY REGULATE CD8+ MEMORY T CELL DEVELOPMENT. [Internet] [Doctoral dissertation]. Cornell University; 2020. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/70427.

Council of Science Editors:

Solouki S. T CELL RECEPTOR SIGNAL STRENGTH AND ANTIGEN AFFINITY DIFFERENTIALLY REGULATE CD8+ MEMORY T CELL DEVELOPMENT. [Doctoral Dissertation]. Cornell University; 2020. Available from: http://hdl.handle.net/1813/70427


Cornell University

2. Mahamed, Deeqa. The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology.

Degree: PhD, Immunology, 2013, Cornell University

 The protozoan pathogen Toxoplasma gondii is a highly successful parasite that infects up to a third of the world's population, causing morbidity and mortality in… (more)

Subjects/Keywords: Toxoplasma gondii; Extracellular Adenosine; CD73

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APA (6th Edition):

Mahamed, D. (2013). The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33839

Chicago Manual of Style (16th Edition):

Mahamed, Deeqa. “The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology.” 2013. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/33839.

MLA Handbook (7th Edition):

Mahamed, Deeqa. “The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology.” 2013. Web. 28 Feb 2021.

Vancouver:

Mahamed D. The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/33839.

Council of Science Editors:

Mahamed D. The 5'-Ectoenzyme Cd73 Promotes Toxoplasma Gondii Persistence In The Cns While Limiting Systemic Immunopathology. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33839


Cornell University

3. Mohinta, Sonia. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.

Degree: PhD, Immunology, 2014, Cornell University

 A number of autoimmune and chronic inflammatory diseases are related to environmental stress. Aryl hydrocarbon receptor (AHR) is regarded as an environmental sensor integrating immune… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor; Th17; Tcell differentiation

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APA (6th Edition):

Mohinta, S. (2014). Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/36177

Chicago Manual of Style (16th Edition):

Mohinta, Sonia. “Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.” 2014. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/36177.

MLA Handbook (7th Edition):

Mohinta, Sonia. “Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function.” 2014. Web. 28 Feb 2021.

Vancouver:

Mohinta S. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/36177.

Council of Science Editors:

Mohinta S. Role Of Aryl Hydrocarbon Receptor (Ahr) And The Effect Of Selective Ahr Modulators (Sahrms) On T Cell Differentiation And Effector Function. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36177


Cornell University

4. Elmore, Sara. Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection.

Degree: PhD, Immunology, 2014, Cornell University

 The immune response to an invading pathogen must be tightly regulated so as to combat the infection while avoiding immune-mediated pathologies. This requires the coordinated… (more)

Subjects/Keywords: Toxoplasma gondii; Immunity; Infection

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APA (6th Edition):

Elmore, S. (2014). Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/38921

Chicago Manual of Style (16th Edition):

Elmore, Sara. “Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection.” 2014. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/38921.

MLA Handbook (7th Edition):

Elmore, Sara. “Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection.” 2014. Web. 28 Feb 2021.

Vancouver:

Elmore S. Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/38921.

Council of Science Editors:

Elmore S. Immunity And Cell Signaling In The Host Response To Toxoplasma Gondii Infection. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/38921


Cornell University

5. Gebreselassie, Nebiat. The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis.

Degree: PhD, Immunology, 2012, Cornell University

 Parasitic diseases pose a significant burden on the health of millions of people. Chronic helminth infections are typified by enhanced Th2 responses. Understanding how Th2… (more)

Subjects/Keywords: Trichinella; Nematode; Th2 immune response; stat6; Eosinophils; Nitric oxide; Glucose homeostasis

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APA (6th Edition):

Gebreselassie, N. (2012). The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/31113

Chicago Manual of Style (16th Edition):

Gebreselassie, Nebiat. “The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis.” 2012. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/31113.

MLA Handbook (7th Edition):

Gebreselassie, Nebiat. “The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis.” 2012. Web. 28 Feb 2021.

Vancouver:

Gebreselassie N. The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/31113.

Council of Science Editors:

Gebreselassie N. The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/31113


Cornell University

6. Ahn, Sung Ji. NON-LINEAR OPTICAL METHODS TO UNDERSTAND PATHOPHYSIOLOGY OF SMALL BLEEDS.

Degree: PhD, Biomedical Engineering, 2017, Cornell University

 Cerebral microhemorrhages (CMBs) are small hemorrhagic strokes found in the brain, also known as silent stroke since they do not illicit noticeable symptoms. Recently, due… (more)

Subjects/Keywords: cerebral micro bleeds; microgila; third harmonic generation; Biomedical engineering; Blood Flow; Multiphoton microscopy

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APA (6th Edition):

Ahn, S. J. (2017). NON-LINEAR OPTICAL METHODS TO UNDERSTAND PATHOPHYSIOLOGY OF SMALL BLEEDS. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/59064

Chicago Manual of Style (16th Edition):

Ahn, Sung Ji. “NON-LINEAR OPTICAL METHODS TO UNDERSTAND PATHOPHYSIOLOGY OF SMALL BLEEDS.” 2017. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/59064.

MLA Handbook (7th Edition):

Ahn, Sung Ji. “NON-LINEAR OPTICAL METHODS TO UNDERSTAND PATHOPHYSIOLOGY OF SMALL BLEEDS.” 2017. Web. 28 Feb 2021.

Vancouver:

Ahn SJ. NON-LINEAR OPTICAL METHODS TO UNDERSTAND PATHOPHYSIOLOGY OF SMALL BLEEDS. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/59064.

Council of Science Editors:

Ahn SJ. NON-LINEAR OPTICAL METHODS TO UNDERSTAND PATHOPHYSIOLOGY OF SMALL BLEEDS. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/59064


Cornell University

7. Kim, Do-Geun. The Role Of Extracellular Adenosine In Regulation Of Paracellular And Transcellular Permeability Of Blood Brain Barrier.

Degree: PhD, Veterinary Medicine, 2015, Cornell University

 The brain is the center of cognitive function and also regulates the physiology of the body. Due to its importance, it requires special vascular structure… (more)

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APA (6th Edition):

Kim, D. (2015). The Role Of Extracellular Adenosine In Regulation Of Paracellular And Transcellular Permeability Of Blood Brain Barrier. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/40912

Chicago Manual of Style (16th Edition):

Kim, Do-Geun. “The Role Of Extracellular Adenosine In Regulation Of Paracellular And Transcellular Permeability Of Blood Brain Barrier.” 2015. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/40912.

MLA Handbook (7th Edition):

Kim, Do-Geun. “The Role Of Extracellular Adenosine In Regulation Of Paracellular And Transcellular Permeability Of Blood Brain Barrier.” 2015. Web. 28 Feb 2021.

Vancouver:

Kim D. The Role Of Extracellular Adenosine In Regulation Of Paracellular And Transcellular Permeability Of Blood Brain Barrier. [Internet] [Doctoral dissertation]. Cornell University; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/40912.

Council of Science Editors:

Kim D. The Role Of Extracellular Adenosine In Regulation Of Paracellular And Transcellular Permeability Of Blood Brain Barrier. [Doctoral Dissertation]. Cornell University; 2015. Available from: http://hdl.handle.net/1813/40912


Cornell University

8. Carter, Chavez. Role Of Itk In Th17 Mediated Inflammation Model Hypersensitivity Pneumonitis.

Degree: PhD, Immunology, 2015, Cornell University

 Hypersensitivity Pneumonitis (HP) is a lung disease caused by repeated inhalation of environmental antigens leading to inflammation, tissue scarring, and some loss of lung function.… (more)

Subjects/Keywords: Itk; Hypersensitivity Pneumonitis; T cells

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APA (6th Edition):

Carter, C. (2015). Role Of Itk In Th17 Mediated Inflammation Model Hypersensitivity Pneumonitis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/41075

Chicago Manual of Style (16th Edition):

Carter, Chavez. “Role Of Itk In Th17 Mediated Inflammation Model Hypersensitivity Pneumonitis.” 2015. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/41075.

MLA Handbook (7th Edition):

Carter, Chavez. “Role Of Itk In Th17 Mediated Inflammation Model Hypersensitivity Pneumonitis.” 2015. Web. 28 Feb 2021.

Vancouver:

Carter C. Role Of Itk In Th17 Mediated Inflammation Model Hypersensitivity Pneumonitis. [Internet] [Doctoral dissertation]. Cornell University; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/41075.

Council of Science Editors:

Carter C. Role Of Itk In Th17 Mediated Inflammation Model Hypersensitivity Pneumonitis. [Doctoral Dissertation]. Cornell University; 2015. Available from: http://hdl.handle.net/1813/41075


Cornell University

9. Mohanan Nair Padmini, Sunish. Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation.

Degree: PhD, Veterinary Medicine, 2014, Cornell University

 Numerous recent studies have shown that epigenetic modifications play a significant role in cancer pathogenesis. The PADs are a family of epigenetic enzymes that catalyze… (more)

Subjects/Keywords: Peptidylarginine deiminase 2 (PAD2); Cancer progression; Extracellular chromatin traps (ETosis)

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APA (6th Edition):

Mohanan Nair Padmini, S. (2014). Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/36176

Chicago Manual of Style (16th Edition):

Mohanan Nair Padmini, Sunish. “Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation.” 2014. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/36176.

MLA Handbook (7th Edition):

Mohanan Nair Padmini, Sunish. “Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation.” 2014. Web. 28 Feb 2021.

Vancouver:

Mohanan Nair Padmini S. Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/36176.

Council of Science Editors:

Mohanan Nair Padmini S. Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36176


Cornell University

10. Huang, Weishan. Function Of Il-2-Inducible T Cell Kinase (Itk) In Innate T Cells And Mast Cells.

Degree: PhD, Pharmacology, 2014, Cornell University

 IL-2-inducible T cell kinase (ITK) is expressed in T lymphocytes and mast cells (MC), and functions as a critical signaling mediator downstream of numerous cell… (more)

Subjects/Keywords: IL-2-inducible T cell kinase (ITK); Innate T cells; Mast cells

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APA (6th Edition):

Huang, W. (2014). Function Of Il-2-Inducible T Cell Kinase (Itk) In Innate T Cells And Mast Cells. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/36191

Chicago Manual of Style (16th Edition):

Huang, Weishan. “Function Of Il-2-Inducible T Cell Kinase (Itk) In Innate T Cells And Mast Cells.” 2014. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/36191.

MLA Handbook (7th Edition):

Huang, Weishan. “Function Of Il-2-Inducible T Cell Kinase (Itk) In Innate T Cells And Mast Cells.” 2014. Web. 28 Feb 2021.

Vancouver:

Huang W. Function Of Il-2-Inducible T Cell Kinase (Itk) In Innate T Cells And Mast Cells. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/36191.

Council of Science Editors:

Huang W. Function Of Il-2-Inducible T Cell Kinase (Itk) In Innate T Cells And Mast Cells. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36191


Cornell University

11. Wang, Jie. MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES.

Degree: PhD, Immunology and Infectious Disease, 2017, Cornell University

 Neonatal infection is a major cause of morbidity and mortality worldwide. While adults generate robust immunity to most intracellular pathogens, neonates have an impaired ability… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Wang, J. (2017). MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/59007

Chicago Manual of Style (16th Edition):

Wang, Jie. “MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES.” 2017. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/59007.

MLA Handbook (7th Edition):

Wang, Jie. “MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES.” 2017. Web. 28 Feb 2021.

Vancouver:

Wang J. MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/59007.

Council of Science Editors:

Wang J. MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/59007

12. Yan, Angela. THE ROLE OF CD73 IN GLIOBLASTOMA PATHOGENESIS AND ANTI-GLIOBLASTOMA IMMUNE RESPONSE.

Degree: PhD, Biomedical and Biological Sciences, 2019, Cornell University

 CD73 is a key enzyme in extracellular ATP metabolism; it converts AMP to extracellular adenosine. CD73 is involved in many cellular functions, including cell adhesion,… (more)

Subjects/Keywords: Immunology; A2B adenosine receptor; cancer immunology; CD73; extracellular adenosine; glioblastoma; glioma chemoresistance; Oncology

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APA (6th Edition):

Yan, A. (2019). THE ROLE OF CD73 IN GLIOBLASTOMA PATHOGENESIS AND ANTI-GLIOBLASTOMA IMMUNE RESPONSE. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/67210

Chicago Manual of Style (16th Edition):

Yan, Angela. “THE ROLE OF CD73 IN GLIOBLASTOMA PATHOGENESIS AND ANTI-GLIOBLASTOMA IMMUNE RESPONSE.” 2019. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/67210.

MLA Handbook (7th Edition):

Yan, Angela. “THE ROLE OF CD73 IN GLIOBLASTOMA PATHOGENESIS AND ANTI-GLIOBLASTOMA IMMUNE RESPONSE.” 2019. Web. 28 Feb 2021.

Vancouver:

Yan A. THE ROLE OF CD73 IN GLIOBLASTOMA PATHOGENESIS AND ANTI-GLIOBLASTOMA IMMUNE RESPONSE. [Internet] [Doctoral dissertation]. Cornell University; 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/67210.

Council of Science Editors:

Yan A. THE ROLE OF CD73 IN GLIOBLASTOMA PATHOGENESIS AND ANTI-GLIOBLASTOMA IMMUNE RESPONSE. [Doctoral Dissertation]. Cornell University; 2019. Available from: http://hdl.handle.net/1813/67210

13. Purwada, Alberto. Bioengineered Immune Organoids for Controlling B Cell Development.

Degree: PhD, Biomedical Engineering, 2017, Cornell University

 The humoral arm of the adaptive immune system works by producing antigen-specific antibody to clear pathogens from the extracellular spaces in the body. The humoral… (more)

Subjects/Keywords: Biomedical engineering; 3D Cell Culture; B Cells; Germinal Center; Lymphoid Tissues; Tissue Engineering

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APA (6th Edition):

Purwada, A. (2017). Bioengineered Immune Organoids for Controlling B Cell Development. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/56816

Chicago Manual of Style (16th Edition):

Purwada, Alberto. “Bioengineered Immune Organoids for Controlling B Cell Development.” 2017. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/56816.

MLA Handbook (7th Edition):

Purwada, Alberto. “Bioengineered Immune Organoids for Controlling B Cell Development.” 2017. Web. 28 Feb 2021.

Vancouver:

Purwada A. Bioengineered Immune Organoids for Controlling B Cell Development. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/56816.

Council of Science Editors:

Purwada A. Bioengineered Immune Organoids for Controlling B Cell Development. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/56816

14. Pomeroy, Brianna Joy. LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS.

Degree: PhD, Veterinary Medicine, 2017, Cornell University

 Cows are susceptible to new Escherichia coli intramammary infections (IMI) in the non-lactating period of late gestation (known as the ‘dry period’). These IMI often… (more)

Subjects/Keywords: Bovine; E. coli; Late gestation; Mastitis; Monocyte; Monocyte-derived dendritic cell; Veterinary science; Immunology; Epidemiology

…to the exciting world of bovine research when I was an undergraduate at Cornell University… 

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APA (6th Edition):

Pomeroy, B. J. (2017). LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/47803

Chicago Manual of Style (16th Edition):

Pomeroy, Brianna Joy. “LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS.” 2017. Doctoral Dissertation, Cornell University. Accessed February 28, 2021. http://hdl.handle.net/1813/47803.

MLA Handbook (7th Edition):

Pomeroy, Brianna Joy. “LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS.” 2017. Web. 28 Feb 2021.

Vancouver:

Pomeroy BJ. LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1813/47803.

Council of Science Editors:

Pomeroy BJ. LATE GESTATION MATERNAL IMMUNE FUNCTION AND CONCURRENT ESCHERICHIA COLI INTRAMAMMARY INFECTION DYNAMICS. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/47803

.