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You searched for +publisher:"Central Connecticut State University" +contributor:("King, Thomas R."). Showing records 1 – 24 of 24 total matches.

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Central Connecticut State University

1. Ramirez, Francisco. Making a chromosomal assignment for the juvenile alopecia (jal) mutation in mice.

Degree: Department of Biomolecular Sciences, 2012, Central Connecticut State University

The juvenile alopecia (jal) mutation in mice arose on the standard C3H/HeJ inbred background. This mutation controls a poorly-understood recessive disorder in which affected subjects… (more)

Subjects/Keywords: Mutation (Biology); Gene mapping.

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APA (6th Edition):

Ramirez, F. (2012). Making a chromosomal assignment for the juvenile alopecia (jal) mutation in mice. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2081

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramirez, Francisco. “Making a chromosomal assignment for the juvenile alopecia (jal) mutation in mice.” 2012. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2081.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramirez, Francisco. “Making a chromosomal assignment for the juvenile alopecia (jal) mutation in mice.” 2012. Web. 16 Apr 2021.

Vancouver:

Ramirez F. Making a chromosomal assignment for the juvenile alopecia (jal) mutation in mice. [Internet] [Thesis]. Central Connecticut State University; 2012. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2081.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramirez F. Making a chromosomal assignment for the juvenile alopecia (jal) mutation in mice. [Thesis]. Central Connecticut State University; 2012. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2081

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

2. Feliciano, Aaron M. Does the retarded hair growth (rhg) mutation control altered amino acid metabolism and retinal degeneration in mice?.

Degree: Department of Biomolecular Sciences, 2015, Central Connecticut State University

Mice with two copies of the retarded hair growth mutation (abbreviated rhg) display stunted growth, and delayed hair development. Genetic studies have recently located rhg… (more)

Subjects/Keywords: Hair – Growth – Molecular aspects.; Mutation (Biology); Amino acids – Metabolism.; Retinal degeneration.; Mice.

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APA (6th Edition):

Feliciano, A. M. (2015). Does the retarded hair growth (rhg) mutation control altered amino acid metabolism and retinal degeneration in mice?. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2085

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Feliciano, Aaron M. “Does the retarded hair growth (rhg) mutation control altered amino acid metabolism and retinal degeneration in mice?.” 2015. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2085.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Feliciano, Aaron M. “Does the retarded hair growth (rhg) mutation control altered amino acid metabolism and retinal degeneration in mice?.” 2015. Web. 16 Apr 2021.

Vancouver:

Feliciano AM. Does the retarded hair growth (rhg) mutation control altered amino acid metabolism and retinal degeneration in mice?. [Internet] [Thesis]. Central Connecticut State University; 2015. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2085.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Feliciano AM. Does the retarded hair growth (rhg) mutation control altered amino acid metabolism and retinal degeneration in mice?. [Thesis]. Central Connecticut State University; 2015. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2085

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

3. Radden, Legairre A. II. The rough fur (ruf) mutation in mice is an allele of the myelin protein zero-like 3 gene (Mpzl3).

Degree: Department of Biomolecular Sciences, 2016, Central Connecticut State University

The spontaneous, autosomal recessive mutation known as rough fur (abbreviated ruf) is characterized in homozygotes by an unkempt, wet-looking coat, mild hyperkeratosis of the skin… (more)

Subjects/Keywords: Mice – Morphology.

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APA (6th Edition):

Radden, L. A. I. (2016). The rough fur (ruf) mutation in mice is an allele of the myelin protein zero-like 3 gene (Mpzl3). (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radden, Legairre A II. “The rough fur (ruf) mutation in mice is an allele of the myelin protein zero-like 3 gene (Mpzl3).” 2016. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radden, Legairre A II. “The rough fur (ruf) mutation in mice is an allele of the myelin protein zero-like 3 gene (Mpzl3).” 2016. Web. 16 Apr 2021.

Vancouver:

Radden LAI. The rough fur (ruf) mutation in mice is an allele of the myelin protein zero-like 3 gene (Mpzl3). [Internet] [Thesis]. Central Connecticut State University; 2016. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radden LAI. The rough fur (ruf) mutation in mice is an allele of the myelin protein zero-like 3 gene (Mpzl3). [Thesis]. Central Connecticut State University; 2016. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Connor, Malcolm E. (Malcolm Ellington). Are the mouse jal and ruf mutations associated with de novo integration of an endogenous retroviral (ERV) element?.

Degree: Department of Biomolecular Sciences, 2017, Central Connecticut State University

It has previously been shown that the juvenile alopecia (jal) mutation resides in the GATA binding protein 3 (Gata3) gene in mice. However, no DNA… (more)

Subjects/Keywords: Mice – Genetics.

…shown in Figure 1. These mice were maintained at Central Connecticut State University (… 

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APA (6th Edition):

Connor, M. E. (. E. (2017). Are the mouse jal and ruf mutations associated with de novo integration of an endogenous retroviral (ERV) element?. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2564

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Connor, Malcolm E (Malcolm Ellington). “Are the mouse jal and ruf mutations associated with de novo integration of an endogenous retroviral (ERV) element?.” 2017. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2564.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Connor, Malcolm E (Malcolm Ellington). “Are the mouse jal and ruf mutations associated with de novo integration of an endogenous retroviral (ERV) element?.” 2017. Web. 16 Apr 2021.

Vancouver:

Connor ME(E. Are the mouse jal and ruf mutations associated with de novo integration of an endogenous retroviral (ERV) element?. [Internet] [Thesis]. Central Connecticut State University; 2017. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2564.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Connor ME(E. Are the mouse jal and ruf mutations associated with de novo integration of an endogenous retroviral (ERV) element?. [Thesis]. Central Connecticut State University; 2017. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2564

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Bigiarelli, Krista J. Using SNP analysis to refine a genetic map for the "curly whiskers" mutation in mice.

Degree: Department of Biomolecular Sciences, 2019, Central Connecticut State University

The "curly whiskers" mutation in mice (abbreviated cw) causes recessive hair curling and may also generate a single-gene barrier to tissue-graft acceptance, a combination of… (more)

Subjects/Keywords: Single nucleotide polymorphisms.; Mice.

…housed and fed in accordance with Federal and institutional guidelines. The Central Connecticut… …State University Institutional Animal Care and Use Committee (IACUC) approved all… 

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APA (6th Edition):

Bigiarelli, K. J. (2019). Using SNP analysis to refine a genetic map for the "curly whiskers" mutation in mice. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2929

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bigiarelli, Krista J. “Using SNP analysis to refine a genetic map for the "curly whiskers" mutation in mice.” 2019. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2929.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bigiarelli, Krista J. “Using SNP analysis to refine a genetic map for the "curly whiskers" mutation in mice.” 2019. Web. 16 Apr 2021.

Vancouver:

Bigiarelli KJ. Using SNP analysis to refine a genetic map for the "curly whiskers" mutation in mice. [Internet] [Thesis]. Central Connecticut State University; 2019. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2929.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bigiarelli KJ. Using SNP analysis to refine a genetic map for the "curly whiskers" mutation in mice. [Thesis]. Central Connecticut State University; 2019. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2929

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Palanza, Kenneth M. Fine Mapping of the Rough Fur (ruf) Mutation in Mice / Kenneth M. Palanza.

Degree: Department of Biomolecular Sciences, 2017, Central Connecticut State University

The autosomal recessive mouse mutation called rough fur (abbreviated ruf) first appeared at The Jackcon Laboratory (Bar Harbor, ME) in an animal stock colony comprised… (more)

Subjects/Keywords: Mice – Morphology.

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APA (6th Edition):

Palanza, K. M. (2017). Fine Mapping of the Rough Fur (ruf) Mutation in Mice / Kenneth M. Palanza. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Palanza, Kenneth M. “Fine Mapping of the Rough Fur (ruf) Mutation in Mice / Kenneth M. Palanza.” 2017. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Palanza, Kenneth M. “Fine Mapping of the Rough Fur (ruf) Mutation in Mice / Kenneth M. Palanza.” 2017. Web. 16 Apr 2021.

Vancouver:

Palanza KM. Fine Mapping of the Rough Fur (ruf) Mutation in Mice / Kenneth M. Palanza. [Internet] [Thesis]. Central Connecticut State University; 2017. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Palanza KM. Fine Mapping of the Rough Fur (ruf) Mutation in Mice / Kenneth M. Palanza. [Thesis]. Central Connecticut State University; 2017. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

7. Rule, Michael C. High resolution genetic mapping of the mouse male sterility and histoincompatibility (mshi) mutation on proximal chromosome 10.

Degree: Department of Biological Sciences, 1998, Central Connecticut State University

The male sterility and histoincompatibility mutation (mshi) is an autosomal recessive mutation that produces apparently pleiotropic effects in mice. Mice homozygous for mshi manifest two… (more)

Subjects/Keywords: Mutation (Biology); Gene mapping.

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APA (6th Edition):

Rule, M. C. (1998). High resolution genetic mapping of the mouse male sterility and histoincompatibility (mshi) mutation on proximal chromosome 10. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rule, Michael C. “High resolution genetic mapping of the mouse male sterility and histoincompatibility (mshi) mutation on proximal chromosome 10.” 1998. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rule, Michael C. “High resolution genetic mapping of the mouse male sterility and histoincompatibility (mshi) mutation on proximal chromosome 10.” 1998. Web. 16 Apr 2021.

Vancouver:

Rule MC. High resolution genetic mapping of the mouse male sterility and histoincompatibility (mshi) mutation on proximal chromosome 10. [Internet] [Thesis]. Central Connecticut State University; 1998. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rule MC. High resolution genetic mapping of the mouse male sterility and histoincompatibility (mshi) mutation on proximal chromosome 10. [Thesis]. Central Connecticut State University; 1998. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

8. Walton, Cherie. Incorporation of Listeriolysin O into a ligand-based carrier system resulting in enhancement of gene expression.

Degree: Department of Biological Sciences, 1998, Central Connecticut State University

Receptor-mediated endocytosis as a route of gene delivery shows promise as it is a natural cellular process and allows targeting of ligands to specific cells.… (more)

Subjects/Keywords: Proteins – Physiological aspects.; DNA-binding proteins.

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APA (6th Edition):

Walton, C. (1998). Incorporation of Listeriolysin O into a ligand-based carrier system resulting in enhancement of gene expression. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walton, Cherie. “Incorporation of Listeriolysin O into a ligand-based carrier system resulting in enhancement of gene expression.” 1998. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walton, Cherie. “Incorporation of Listeriolysin O into a ligand-based carrier system resulting in enhancement of gene expression.” 1998. Web. 16 Apr 2021.

Vancouver:

Walton C. Incorporation of Listeriolysin O into a ligand-based carrier system resulting in enhancement of gene expression. [Internet] [Thesis]. Central Connecticut State University; 1998. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2177.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walton C. Incorporation of Listeriolysin O into a ligand-based carrier system resulting in enhancement of gene expression. [Thesis]. Central Connecticut State University; 1998. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2177

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

9. Hildebrandt, Audrey. Conservation of the H-mshi antigen between the standard BALB/By, C57BL/6J and wild-derived CAST/Ei inbred strains of mice.

Degree: Department of Biological Sciences, 1997, Central Connecticut State University

Discusses twentieth century expansion in the use of organ transplantation as a tool for maintaining human health. Bone marrow transplants for leukemia patients, heart transplants… (more)

Subjects/Keywords: Transplantation of organs, tissues, etc.; Transplantation immunology.; Major histocompatibility complex.; MHC antibodies.

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APA (6th Edition):

Hildebrandt, A. (1997). Conservation of the H-mshi antigen between the standard BALB/By, C57BL/6J and wild-derived CAST/Ei inbred strains of mice. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hildebrandt, Audrey. “Conservation of the H-mshi antigen between the standard BALB/By, C57BL/6J and wild-derived CAST/Ei inbred strains of mice.” 1997. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hildebrandt, Audrey. “Conservation of the H-mshi antigen between the standard BALB/By, C57BL/6J and wild-derived CAST/Ei inbred strains of mice.” 1997. Web. 16 Apr 2021.

Vancouver:

Hildebrandt A. Conservation of the H-mshi antigen between the standard BALB/By, C57BL/6J and wild-derived CAST/Ei inbred strains of mice. [Internet] [Thesis]. Central Connecticut State University; 1997. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hildebrandt A. Conservation of the H-mshi antigen between the standard BALB/By, C57BL/6J and wild-derived CAST/Ei inbred strains of mice. [Thesis]. Central Connecticut State University; 1997. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

10. Turner, Joseph P. High resolution genetic mapping of the mouse male sterility (mste) mutation on proximal choromsome 10.

Degree: Department of Biological Sciences, 1996, Central Connecticut State University

The male mouse sterility mutation (mste) is an autosomal recessive mutation discovered in BALB / cBy mice which causes male sterility due to apparent spermatogonial… (more)

Subjects/Keywords: Gene mapping.; Chromosomes – Analysis.; Crossing over (Genetics); Polymerase chain reaction – Diagnostic use.

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APA (6th Edition):

Turner, J. P. (1996). High resolution genetic mapping of the mouse male sterility (mste) mutation on proximal choromsome 10. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Turner, Joseph P. “High resolution genetic mapping of the mouse male sterility (mste) mutation on proximal choromsome 10.” 1996. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Turner, Joseph P. “High resolution genetic mapping of the mouse male sterility (mste) mutation on proximal choromsome 10.” 1996. Web. 16 Apr 2021.

Vancouver:

Turner JP. High resolution genetic mapping of the mouse male sterility (mste) mutation on proximal choromsome 10. [Internet] [Thesis]. Central Connecticut State University; 1996. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Turner JP. High resolution genetic mapping of the mouse male sterility (mste) mutation on proximal choromsome 10. [Thesis]. Central Connecticut State University; 1996. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

11. Amador-Medina, Ivette. Adrenal gland influence on the host TNF-a response to lipopolysaccharide (LPS) and monophosphoryl lipid A (MPLA) : evaluation of TNF-a mRNA transcription.

Degree: Department of Biological Sciences, 1995, Central Connecticut State University

Lipopolysaccharide (LPS) and the non-toxic derivative of lipid A, Monophosphoryl Lipid A (MPLA) were utilized to evaluate the influence of the adrenal gland on the… (more)

Subjects/Keywords: Endotoxins.; Tumor necrosis factor.; Adrenocortical hormones.

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APA (6th Edition):

Amador-Medina, I. (1995). Adrenal gland influence on the host TNF-a response to lipopolysaccharide (LPS) and monophosphoryl lipid A (MPLA) : evaluation of TNF-a mRNA transcription. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amador-Medina, Ivette. “Adrenal gland influence on the host TNF-a response to lipopolysaccharide (LPS) and monophosphoryl lipid A (MPLA) : evaluation of TNF-a mRNA transcription.” 1995. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amador-Medina, Ivette. “Adrenal gland influence on the host TNF-a response to lipopolysaccharide (LPS) and monophosphoryl lipid A (MPLA) : evaluation of TNF-a mRNA transcription.” 1995. Web. 16 Apr 2021.

Vancouver:

Amador-Medina I. Adrenal gland influence on the host TNF-a response to lipopolysaccharide (LPS) and monophosphoryl lipid A (MPLA) : evaluation of TNF-a mRNA transcription. [Internet] [Thesis]. Central Connecticut State University; 1995. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amador-Medina I. Adrenal gland influence on the host TNF-a response to lipopolysaccharide (LPS) and monophosphoryl lipid A (MPLA) : evaluation of TNF-a mRNA transcription. [Thesis]. Central Connecticut State University; 1995. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

12. Myrie, Kenute A. Investigation of the basis of male sterility in the mshi/mshi mutant mouse.

Degree: Department of Biological Sciences, 1997, Central Connecticut State University

Male BALB/cBy mice that are homozygous for the coisogenic mshi mutation are sterile and aggressively reject grafts from wildtype BALB/cBy donors. Since mutant testes are… (more)

Subjects/Keywords: Reproduction.; Mammals – Physiology.; Transgenic mice.

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APA (6th Edition):

Myrie, K. A. (1997). Investigation of the basis of male sterility in the mshi/mshi mutant mouse. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Myrie, Kenute A. “Investigation of the basis of male sterility in the mshi/mshi mutant mouse.” 1997. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,2528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Myrie, Kenute A. “Investigation of the basis of male sterility in the mshi/mshi mutant mouse.” 1997. Web. 16 Apr 2021.

Vancouver:

Myrie KA. Investigation of the basis of male sterility in the mshi/mshi mutant mouse. [Internet] [Thesis]. Central Connecticut State University; 1997. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Myrie KA. Investigation of the basis of male sterility in the mshi/mshi mutant mouse. [Thesis]. Central Connecticut State University; 1997. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

13. Paul, Emily L. Defining the location of murine frizzy in relation to Fgfr2.

Degree: Department of Biomolecular Sciences, 2006, Central Connecticut State University

 Ahearn et al. (J. Heredity 93:210-213) have previously shown that the rat fuzzy and Charles River "hairless" mutations are defects in the same gene on… (more)

Subjects/Keywords: Mice  – Genetics; Mice  – Variation

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APA (6th Edition):

Paul, E. L. (2006). Defining the location of murine frizzy in relation to Fgfr2. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paul, Emily L. “Defining the location of murine frizzy in relation to Fgfr2.” 2006. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paul, Emily L. “Defining the location of murine frizzy in relation to Fgfr2.” 2006. Web. 16 Apr 2021.

Vancouver:

Paul EL. Defining the location of murine frizzy in relation to Fgfr2. [Internet] [Thesis]. Central Connecticut State University; 2006. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paul EL. Defining the location of murine frizzy in relation to Fgfr2. [Thesis]. Central Connecticut State University; 2006. Available from: http://content.library.ccsu.edu/u?/ccsutheses,905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

14. Palaski, Kathleen M. Development of a protocol for identifying DNA markers from the pseudoautosomal region in a backcross generation of rats.

Degree: Department of Biological Sciences, 2003, Central Connecticut State University

 The pairing and recombination of homologous regions of the X and Y chromosomes, in an area known as the pseudoautosomal region (PAR), has been a… (more)

Subjects/Keywords: Genetic markers; Cytology

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APA (6th Edition):

Palaski, K. M. (2003). Development of a protocol for identifying DNA markers from the pseudoautosomal region in a backcross generation of rats. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Palaski, Kathleen M. “Development of a protocol for identifying DNA markers from the pseudoautosomal region in a backcross generation of rats.” 2003. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Palaski, Kathleen M. “Development of a protocol for identifying DNA markers from the pseudoautosomal region in a backcross generation of rats.” 2003. Web. 16 Apr 2021.

Vancouver:

Palaski KM. Development of a protocol for identifying DNA markers from the pseudoautosomal region in a backcross generation of rats. [Internet] [Thesis]. Central Connecticut State University; 2003. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Palaski KM. Development of a protocol for identifying DNA markers from the pseudoautosomal region in a backcross generation of rats. [Thesis]. Central Connecticut State University; 2003. Available from: http://content.library.ccsu.edu/u?/ccsutheses,935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

15. Ye, Naiqing. Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation.

Degree: Department of Biomolecular Sciences, 2006, Central Connecticut State University

 The male sterility and histoincompatibility mutant, mshi, results from a recessive mutation that arose spontaneously in the BALB/cBy inbred mouse strain. Using a 402-member BALB/cBy-mshi/mshi… (more)

Subjects/Keywords: Mice  – Infertility; Transgenic mice

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APA (6th Edition):

Ye, N. (2006). Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,976

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ye, Naiqing. “Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation.” 2006. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,976.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ye, Naiqing. “Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation.” 2006. Web. 16 Apr 2021.

Vancouver:

Ye N. Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation. [Internet] [Thesis]. Central Connecticut State University; 2006. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,976.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ye N. Interleukin 20 receptor a (il20ra) is not the molecular basis of the mouse male sterility and histoincompatibility (mshi) mutation. [Thesis]. Central Connecticut State University; 2006. Available from: http://content.library.ccsu.edu/u?/ccsutheses,976

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

16. Haughton, Richard A. (Richard Alexander), 1981-. Mapping the Mouse frizzy Mutation on Chromosome 7 with a (BALB x FS )F1 x FS Backcross.

Degree: Department of Biomolecular Sciences, 2007, Central Connecticut State University

 The mouse frizzy (fr) mutation arose on a genetically mixed background and was first described by G.D. Snell of The Jackson Laboratory in 1951. Mice… (more)

Subjects/Keywords: Chromosome abnormalities; Animal mutation

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APA (6th Edition):

Haughton, Richard A. (Richard Alexander), 1. (2007). Mapping the Mouse frizzy Mutation on Chromosome 7 with a (BALB x FS )F1 x FS Backcross. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1010

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haughton, Richard A. (Richard Alexander), 1981-. “Mapping the Mouse frizzy Mutation on Chromosome 7 with a (BALB x FS )F1 x FS Backcross.” 2007. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1010.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haughton, Richard A. (Richard Alexander), 1981-. “Mapping the Mouse frizzy Mutation on Chromosome 7 with a (BALB x FS )F1 x FS Backcross.” 2007. Web. 16 Apr 2021.

Vancouver:

Haughton, Richard A. (Richard Alexander) 1. Mapping the Mouse frizzy Mutation on Chromosome 7 with a (BALB x FS )F1 x FS Backcross. [Internet] [Thesis]. Central Connecticut State University; 2007. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1010.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haughton, Richard A. (Richard Alexander) 1. Mapping the Mouse frizzy Mutation on Chromosome 7 with a (BALB x FS )F1 x FS Backcross. [Thesis]. Central Connecticut State University; 2007. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1010

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

17. Lu, Yi-Chien. Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice.

Degree: Department of Biomolecular Sciences, 2004, Central Connecticut State University

 The recessive male sterility and histoincompatibility mutation (mshi) in the mouse generates pleiotropic effects on graft transplantation and male reproduction. Previous analysis of backcross mice… (more)

Subjects/Keywords: Mice  – Infertility; Transgenic mice

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APA (6th Edition):

Lu, Y. (2004). Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1026

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lu, Yi-Chien. “Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice.” 2004. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1026.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lu, Yi-Chien. “Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice.” 2004. Web. 16 Apr 2021.

Vancouver:

Lu Y. Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice. [Internet] [Thesis]. Central Connecticut State University; 2004. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1026.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lu Y. Interleukin 20 receptor (IL20RA) is not the gene disrupted by the male sterility and histoincompatibility (mshi) mutation in mice. [Thesis]. Central Connecticut State University; 2004. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1026

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

18. Thompson, David S. Refining the Location of the frizzy (fr) Mutation on Mus musculus Chromosome 7.

Degree: Department of Biomolecular Sciences, 2006, Central Connecticut State University

 The frizzy (fr) mutation in Mus musculus affects hair structure and formation. Early studies demonstrated that the gene responsible for frizzy assorts to mouse Chromosome… (more)

Subjects/Keywords: Mice  – Genetics

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APA (6th Edition):

Thompson, D. S. (2006). Refining the Location of the frizzy (fr) Mutation on Mus musculus Chromosome 7. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thompson, David S. “Refining the Location of the frizzy (fr) Mutation on Mus musculus Chromosome 7.” 2006. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thompson, David S. “Refining the Location of the frizzy (fr) Mutation on Mus musculus Chromosome 7.” 2006. Web. 16 Apr 2021.

Vancouver:

Thompson DS. Refining the Location of the frizzy (fr) Mutation on Mus musculus Chromosome 7. [Internet] [Thesis]. Central Connecticut State University; 2006. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thompson DS. Refining the Location of the frizzy (fr) Mutation on Mus musculus Chromosome 7. [Thesis]. Central Connecticut State University; 2006. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

19. LaMonica, Kristi April. Re-derivation of the B6.C-H2d/bBy inbred mouse strain to remove an unwanted spontaneously arising new barrier to histocompatibility.

Degree: Department of Biological Sciences, 2003, Central Connecticut State University

 Minor histocompatibility (H) antigens are typically identified as barriers to transplantation in graft exchange experiments between major histocompatibility complex (MHC)-matched hosts (Bailey 1975; Snell and… (more)

Subjects/Keywords: Histocompatibility; X chromosome

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APA (6th Edition):

LaMonica, K. A. (2003). Re-derivation of the B6.C-H2d/bBy inbred mouse strain to remove an unwanted spontaneously arising new barrier to histocompatibility. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LaMonica, Kristi April. “Re-derivation of the B6.C-H2d/bBy inbred mouse strain to remove an unwanted spontaneously arising new barrier to histocompatibility.” 2003. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LaMonica, Kristi April. “Re-derivation of the B6.C-H2d/bBy inbred mouse strain to remove an unwanted spontaneously arising new barrier to histocompatibility.” 2003. Web. 16 Apr 2021.

Vancouver:

LaMonica KA. Re-derivation of the B6.C-H2d/bBy inbred mouse strain to remove an unwanted spontaneously arising new barrier to histocompatibility. [Internet] [Thesis]. Central Connecticut State University; 2003. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LaMonica KA. Re-derivation of the B6.C-H2d/bBy inbred mouse strain to remove an unwanted spontaneously arising new barrier to histocompatibility. [Thesis]. Central Connecticut State University; 2003. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

20. Khan, Izaz M. The frizzy (fr) mutation is located between Izaz2.4 and D7Mit165 on Mus musculus Chromosome 7.

Degree: Department of Biomolecular Sciences, 2008, Central Connecticut State University

 The mouse frizzy (fr) mutation is a Mendelian recessive trait that affects hair structure and formation in mice. The mutation arose in a mouse of… (more)

Subjects/Keywords: Chromosome abnormalities; Mice  – Genetics

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APA (6th Edition):

Khan, I. M. (2008). The frizzy (fr) mutation is located between Izaz2.4 and D7Mit165 on Mus musculus Chromosome 7. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Khan, Izaz M. “The frizzy (fr) mutation is located between Izaz2.4 and D7Mit165 on Mus musculus Chromosome 7.” 2008. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Khan, Izaz M. “The frizzy (fr) mutation is located between Izaz2.4 and D7Mit165 on Mus musculus Chromosome 7.” 2008. Web. 16 Apr 2021.

Vancouver:

Khan IM. The frizzy (fr) mutation is located between Izaz2.4 and D7Mit165 on Mus musculus Chromosome 7. [Internet] [Thesis]. Central Connecticut State University; 2008. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Khan IM. The frizzy (fr) mutation is located between Izaz2.4 and D7Mit165 on Mus musculus Chromosome 7. [Thesis]. Central Connecticut State University; 2008. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

21. Alner, Kadie-Ann. Is H2d required for optimum presentation of the H-hix b -encoded antigan? / Kadie-Ann Alner.

Degree: Department of Biology, 2002, Central Connecticut State University

 Investigation of X-linked histocompatibility may offer an opportunity to find CD and HD minor H haplotype components which are widely separated on the X chromosome.… (more)

Subjects/Keywords: Immunogenetics; Transplantation immunology

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APA (6th Edition):

Alner, K. (2002). Is H2d required for optimum presentation of the H-hix b -encoded antigan? / Kadie-Ann Alner. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1457

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alner, Kadie-Ann. “Is H2d required for optimum presentation of the H-hix b -encoded antigan? / Kadie-Ann Alner.” 2002. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1457.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alner, Kadie-Ann. “Is H2d required for optimum presentation of the H-hix b -encoded antigan? / Kadie-Ann Alner.” 2002. Web. 16 Apr 2021.

Vancouver:

Alner K. Is H2d required for optimum presentation of the H-hix b -encoded antigan? / Kadie-Ann Alner. [Internet] [Thesis]. Central Connecticut State University; 2002. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1457.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alner K. Is H2d required for optimum presentation of the H-hix b -encoded antigan? / Kadie-Ann Alner. [Thesis]. Central Connecticut State University; 2002. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1457

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

22. Berry, Peter R., 1965-. Maternal insufficiency in rats homozygous for the shorn (SHN) mutation / Peter R. Berry.

Degree: Department of Biomolecular Sciences, 2005, Central Connecticut State University

 Rats that are hypotrichotic (lacking normal hair) due to the autosomal recessive shorn (shn) mutation showed a low survival rate in brother by sister matings… (more)

Subjects/Keywords: Parental behavior in animals; Rats  – Physiology; Animal mutation

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APA (6th Edition):

Berry, Peter R., 1. (2005). Maternal insufficiency in rats homozygous for the shorn (SHN) mutation / Peter R. Berry. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1482

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Berry, Peter R., 1965-. “Maternal insufficiency in rats homozygous for the shorn (SHN) mutation / Peter R. Berry.” 2005. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1482.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Berry, Peter R., 1965-. “Maternal insufficiency in rats homozygous for the shorn (SHN) mutation / Peter R. Berry.” 2005. Web. 16 Apr 2021.

Vancouver:

Berry, Peter R. 1. Maternal insufficiency in rats homozygous for the shorn (SHN) mutation / Peter R. Berry. [Internet] [Thesis]. Central Connecticut State University; 2005. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1482.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Berry, Peter R. 1. Maternal insufficiency in rats homozygous for the shorn (SHN) mutation / Peter R. Berry. [Thesis]. Central Connecticut State University; 2005. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1482

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

23. Reale, Virginia Danielle Hikel. Engineering a heterologous transforming growth factor-alpha promoter that is glucose-responsive in 293T cells / Virginia Danielle Hikel Reale.

Degree: Department of Biological Sciences, 2002, Central Connecticut State University

 Transforming growth factor-alpha (TGFa), a member of the endothelial growth factor (EGF) family of mitogens, was previously thought to exist solely in neoplastic cells and… (more)

Subjects/Keywords: Cytology

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APA (6th Edition):

Reale, V. D. H. (2002). Engineering a heterologous transforming growth factor-alpha promoter that is glucose-responsive in 293T cells / Virginia Danielle Hikel Reale. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reale, Virginia Danielle Hikel. “Engineering a heterologous transforming growth factor-alpha promoter that is glucose-responsive in 293T cells / Virginia Danielle Hikel Reale.” 2002. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reale, Virginia Danielle Hikel. “Engineering a heterologous transforming growth factor-alpha promoter that is glucose-responsive in 293T cells / Virginia Danielle Hikel Reale.” 2002. Web. 16 Apr 2021.

Vancouver:

Reale VDH. Engineering a heterologous transforming growth factor-alpha promoter that is glucose-responsive in 293T cells / Virginia Danielle Hikel Reale. [Internet] [Thesis]. Central Connecticut State University; 2002. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reale VDH. Engineering a heterologous transforming growth factor-alpha promoter that is glucose-responsive in 293T cells / Virginia Danielle Hikel Reale. [Thesis]. Central Connecticut State University; 2002. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

24. Wu, Lillya Kaun-Lin. Frizzy (fr), a Prss8 variant, generates defective skin and hair development in mice.

Degree: Department of Biomolecular Sciences, 2009, Central Connecticut State University

 Frizzy (fr) is a spontaneous recessive mutation in mice characterized by curly vibrissae, a disheveled coat and short hair (Snell 1951). Previous linkage analysis located… (more)

Subjects/Keywords: Mice  – Genetics; Mice  – Variation

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APA (6th Edition):

Wu, L. K. (2009). Frizzy (fr), a Prss8 variant, generates defective skin and hair development in mice. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Lillya Kaun-Lin. “Frizzy (fr), a Prss8 variant, generates defective skin and hair development in mice.” 2009. Thesis, Central Connecticut State University. Accessed April 16, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Lillya Kaun-Lin. “Frizzy (fr), a Prss8 variant, generates defective skin and hair development in mice.” 2009. Web. 16 Apr 2021.

Vancouver:

Wu LK. Frizzy (fr), a Prss8 variant, generates defective skin and hair development in mice. [Internet] [Thesis]. Central Connecticut State University; 2009. [cited 2021 Apr 16]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu LK. Frizzy (fr), a Prss8 variant, generates defective skin and hair development in mice. [Thesis]. Central Connecticut State University; 2009. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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