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You searched for +publisher:"Brown University" +contributor:("Reichner, Jonathan"). Showing records 1 – 26 of 26 total matches.

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1. Oakes, Patrick William. The Liquid Crystalline Transition of F-actin and Neutrophil Mechanosensing.

Degree: PhD, Physics, 2009, Brown University

 This dissertation explores two independent projects in biophysics. Part one focuses on the liquid crystalline phase transition of F-actin. The cytoskeletal protein actin can be… (more)

Subjects/Keywords: actin

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APA (6th Edition):

Oakes, P. W. (2009). The Liquid Crystalline Transition of F-actin and Neutrophil Mechanosensing. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:147/

Chicago Manual of Style (16th Edition):

Oakes, Patrick William. “The Liquid Crystalline Transition of F-actin and Neutrophil Mechanosensing.” 2009. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:147/.

MLA Handbook (7th Edition):

Oakes, Patrick William. “The Liquid Crystalline Transition of F-actin and Neutrophil Mechanosensing.” 2009. Web. 25 Mar 2019.

Vancouver:

Oakes PW. The Liquid Crystalline Transition of F-actin and Neutrophil Mechanosensing. [Internet] [Doctoral dissertation]. Brown University; 2009. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:147/.

Council of Science Editors:

Oakes PW. The Liquid Crystalline Transition of F-actin and Neutrophil Mechanosensing. [Doctoral Dissertation]. Brown University; 2009. Available from: https://repository.library.brown.edu/studio/item/bdr:147/

2. Loosley, Alexander J. The Mechanics of Cell Motility and a Unifying Theory for Characterizing Directed Motion.

Degree: PhD, Physics, 2015, Brown University

 This dissertation is split into two parts with the first focusing on characterizing cell motility, and the second focusing on linking cell motility to cell… (more)

Subjects/Keywords: cell motility

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APA (6th Edition):

Loosley, A. J. (2015). The Mechanics of Cell Motility and a Unifying Theory for Characterizing Directed Motion. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419363/

Chicago Manual of Style (16th Edition):

Loosley, Alexander J. “The Mechanics of Cell Motility and a Unifying Theory for Characterizing Directed Motion.” 2015. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:419363/.

MLA Handbook (7th Edition):

Loosley, Alexander J. “The Mechanics of Cell Motility and a Unifying Theory for Characterizing Directed Motion.” 2015. Web. 25 Mar 2019.

Vancouver:

Loosley AJ. The Mechanics of Cell Motility and a Unifying Theory for Characterizing Directed Motion. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:419363/.

Council of Science Editors:

Loosley AJ. The Mechanics of Cell Motility and a Unifying Theory for Characterizing Directed Motion. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419363/

3. Riolo, Matthew T. HDAC6 restrict acetylated survivin nuclear export.

Degree: PhD, Pathobiology, 2012, Brown University

 Survivin is an oncofetal protein expressed in most tumors. It is a unique member of the inhibitor of apoptosis family of proteins having a dual… (more)

Subjects/Keywords: HDAC6

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APA (6th Edition):

Riolo, M. T. (2012). HDAC6 restrict acetylated survivin nuclear export. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297559/

Chicago Manual of Style (16th Edition):

Riolo, Matthew T. “HDAC6 restrict acetylated survivin nuclear export.” 2012. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:297559/.

MLA Handbook (7th Edition):

Riolo, Matthew T. “HDAC6 restrict acetylated survivin nuclear export.” 2012. Web. 25 Mar 2019.

Vancouver:

Riolo MT. HDAC6 restrict acetylated survivin nuclear export. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:297559/.

Council of Science Editors:

Riolo MT. HDAC6 restrict acetylated survivin nuclear export. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297559/

4. Heflin, Katie Elizabeth. The effects of extracellular matrix proteins and fungal �glucan on CR3 structure, neutrophil migration and mechanosensing.

Degree: PhD, Pathobiology, 2011, Brown University

 Neutrophils are the body�s first cellular line of defense. Many of their functions are regulated by integrins through interactions with extracellular matrix (ECM). Insufficient activity… (more)

Subjects/Keywords: CR3

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APA (6th Edition):

Heflin, K. E. (2011). The effects of extracellular matrix proteins and fungal �glucan on CR3 structure, neutrophil migration and mechanosensing. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11296/

Chicago Manual of Style (16th Edition):

Heflin, Katie Elizabeth. “The effects of extracellular matrix proteins and fungal �glucan on CR3 structure, neutrophil migration and mechanosensing.” 2011. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:11296/.

MLA Handbook (7th Edition):

Heflin, Katie Elizabeth. “The effects of extracellular matrix proteins and fungal �glucan on CR3 structure, neutrophil migration and mechanosensing.” 2011. Web. 25 Mar 2019.

Vancouver:

Heflin KE. The effects of extracellular matrix proteins and fungal �glucan on CR3 structure, neutrophil migration and mechanosensing. [Internet] [Doctoral dissertation]. Brown University; 2011. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:11296/.

Council of Science Editors:

Heflin KE. The effects of extracellular matrix proteins and fungal �glucan on CR3 structure, neutrophil migration and mechanosensing. [Doctoral Dissertation]. Brown University; 2011. Available from: https://repository.library.brown.edu/studio/item/bdr:11296/

5. Chung, Waihong. MOLECULAR MECHANISMS OF HEPATOCARCINOGENESIS IN ATX/IRS TRANSGENIC MICE.

Degree: PhD, Pathobiology, 2015, Brown University

 Hepatocellular carcinoma (HCC) is the fifth most common cancer in the world and the third leading cause of cancer death in the United States. Chronic… (more)

Subjects/Keywords: Hepatocellular

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APA (6th Edition):

Chung, W. (2015). MOLECULAR MECHANISMS OF HEPATOCARCINOGENESIS IN ATX/IRS TRANSGENIC MICE. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419352/

Chicago Manual of Style (16th Edition):

Chung, Waihong. “MOLECULAR MECHANISMS OF HEPATOCARCINOGENESIS IN ATX/IRS TRANSGENIC MICE.” 2015. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:419352/.

MLA Handbook (7th Edition):

Chung, Waihong. “MOLECULAR MECHANISMS OF HEPATOCARCINOGENESIS IN ATX/IRS TRANSGENIC MICE.” 2015. Web. 25 Mar 2019.

Vancouver:

Chung W. MOLECULAR MECHANISMS OF HEPATOCARCINOGENESIS IN ATX/IRS TRANSGENIC MICE. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:419352/.

Council of Science Editors:

Chung W. MOLECULAR MECHANISMS OF HEPATOCARCINOGENESIS IN ATX/IRS TRANSGENIC MICE. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419352/

6. Magruder, Hilary. Signaling Effectors Required for G Protein-Coupled Estrogen Receptor-, GPER, Induced Events Associated with Breast Cancer Progression.

Degree: PhD, Pathobiology, 2014, Brown University

 Stimulation of estrogen receptor (ER)-negative human breast cancer cells with 17β-estradiol (E2β) results in fibronectin (FN) matrix assembly and transactivation of the epidermal growth factor… (more)

Subjects/Keywords: estrogen receptor (ER)

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APA (6th Edition):

Magruder, H. (2014). Signaling Effectors Required for G Protein-Coupled Estrogen Receptor-, GPER, Induced Events Associated with Breast Cancer Progression. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:386260/

Chicago Manual of Style (16th Edition):

Magruder, Hilary. “Signaling Effectors Required for G Protein-Coupled Estrogen Receptor-, GPER, Induced Events Associated with Breast Cancer Progression.” 2014. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:386260/.

MLA Handbook (7th Edition):

Magruder, Hilary. “Signaling Effectors Required for G Protein-Coupled Estrogen Receptor-, GPER, Induced Events Associated with Breast Cancer Progression.” 2014. Web. 25 Mar 2019.

Vancouver:

Magruder H. Signaling Effectors Required for G Protein-Coupled Estrogen Receptor-, GPER, Induced Events Associated with Breast Cancer Progression. [Internet] [Doctoral dissertation]. Brown University; 2014. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:386260/.

Council of Science Editors:

Magruder H. Signaling Effectors Required for G Protein-Coupled Estrogen Receptor-, GPER, Induced Events Associated with Breast Cancer Progression. [Doctoral Dissertation]. Brown University; 2014. Available from: https://repository.library.brown.edu/studio/item/bdr:386260/

7. Biron, Bethany. The Role of Peptidyl arginine deiminase, type IV (PAD4) in the Pathology of Shock/Sepsis.

Degree: Pathobiology, 2017, Brown University

 Sepsis is a life threatening condition that elicits a dysregulated and damaging immune response, which in turn leads to tissue damage and Multiple Organ Dysfunction… (more)

Subjects/Keywords: Cellular immunity

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APA (6th Edition):

Biron, B. (2017). The Role of Peptidyl arginine deiminase, type IV (PAD4) in the Pathology of Shock/Sepsis. (Thesis). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:733274/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Biron, Bethany. “The Role of Peptidyl arginine deiminase, type IV (PAD4) in the Pathology of Shock/Sepsis.” 2017. Thesis, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:733274/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Biron, Bethany. “The Role of Peptidyl arginine deiminase, type IV (PAD4) in the Pathology of Shock/Sepsis.” 2017. Web. 25 Mar 2019.

Vancouver:

Biron B. The Role of Peptidyl arginine deiminase, type IV (PAD4) in the Pathology of Shock/Sepsis. [Internet] [Thesis]. Brown University; 2017. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:733274/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Biron B. The Role of Peptidyl arginine deiminase, type IV (PAD4) in the Pathology of Shock/Sepsis. [Thesis]. Brown University; 2017. Available from: https://repository.library.brown.edu/studio/item/bdr:733274/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Cao, Cong. The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome.

Degree: PhD, Pathobiology, 2011, Brown University

 The Epidermal growth factor (EGF) receptor (EGFR), tropomyosin-receptor kinase (Trk) and other receptor tyrosine kinase (RTK) family members play pivotal roles in regulating the normal… (more)

Subjects/Keywords: receptor tyrosine kinase signaling

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APA (6th Edition):

Cao, C. (2011). The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11272/

Chicago Manual of Style (16th Edition):

Cao, Cong. “The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome.” 2011. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:11272/.

MLA Handbook (7th Edition):

Cao, Cong. “The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome.” 2011. Web. 25 Mar 2019.

Vancouver:

Cao C. The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome. [Internet] [Doctoral dissertation]. Brown University; 2011. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:11272/.

Council of Science Editors:

Cao C. The role of inhibitory heterotrimeric G proteins in Receptor Tyrosine Kinase function and identification of defective TrkB signaling in Angelman Syndrome. [Doctoral Dissertation]. Brown University; 2011. Available from: https://repository.library.brown.edu/studio/item/bdr:11272/

9. Linden, Jennifer R. Galectin-3 and Candidiasis: Its Role in Human Neutrophil Phagocytosis and in Disseminated Disease.

Degree: PhD, Pathobiology, 2012, Brown University

 Candida albicans causes the majority of invasive candidiasis in immunocompromised adults while Candida parapsilosis is a leading cause of neonatal candidiasis. While considerable study has… (more)

Subjects/Keywords: Candida parapsilosis

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APA (6th Edition):

Linden, J. R. (2012). Galectin-3 and Candidiasis: Its Role in Human Neutrophil Phagocytosis and in Disseminated Disease. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297569/

Chicago Manual of Style (16th Edition):

Linden, Jennifer R. “Galectin-3 and Candidiasis: Its Role in Human Neutrophil Phagocytosis and in Disseminated Disease.” 2012. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:297569/.

MLA Handbook (7th Edition):

Linden, Jennifer R. “Galectin-3 and Candidiasis: Its Role in Human Neutrophil Phagocytosis and in Disseminated Disease.” 2012. Web. 25 Mar 2019.

Vancouver:

Linden JR. Galectin-3 and Candidiasis: Its Role in Human Neutrophil Phagocytosis and in Disseminated Disease. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:297569/.

Council of Science Editors:

Linden JR. Galectin-3 and Candidiasis: Its Role in Human Neutrophil Phagocytosis and in Disseminated Disease. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297569/

10. Toyjanova, Jennet. The Effect of Confinement on Neutrophil Force Generation.

Degree: PhD, Mechanics of Solids, 2015, Brown University

 Generation of forces is essential for the migration of neutrophils to the sites of infection or injury. The mechanical properties and local confinement of the… (more)

Subjects/Keywords: cell mechanics

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APA (6th Edition):

Toyjanova, J. (2015). The Effect of Confinement on Neutrophil Force Generation. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419365/

Chicago Manual of Style (16th Edition):

Toyjanova, Jennet. “The Effect of Confinement on Neutrophil Force Generation.” 2015. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:419365/.

MLA Handbook (7th Edition):

Toyjanova, Jennet. “The Effect of Confinement on Neutrophil Force Generation.” 2015. Web. 25 Mar 2019.

Vancouver:

Toyjanova J. The Effect of Confinement on Neutrophil Force Generation. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:419365/.

Council of Science Editors:

Toyjanova J. The Effect of Confinement on Neutrophil Force Generation. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419365/

11. Newsome, Courtni Takiyah. Effects of Beta-Glucan on Sepsis and Endotoxemia.

Degree: PhD, Pathobiology, 2010, Brown University

 Beta-glucans are glucose polymers found of the fungal cell wall. Beta-glucans have immunological effects and can stimulate innate immune cells. Priming immune cell function can… (more)

Subjects/Keywords: beta-glucan

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APA (6th Edition):

Newsome, C. T. (2010). Effects of Beta-Glucan on Sepsis and Endotoxemia. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11086/

Chicago Manual of Style (16th Edition):

Newsome, Courtni Takiyah. “Effects of Beta-Glucan on Sepsis and Endotoxemia.” 2010. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:11086/.

MLA Handbook (7th Edition):

Newsome, Courtni Takiyah. “Effects of Beta-Glucan on Sepsis and Endotoxemia.” 2010. Web. 25 Mar 2019.

Vancouver:

Newsome CT. Effects of Beta-Glucan on Sepsis and Endotoxemia. [Internet] [Doctoral dissertation]. Brown University; 2010. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:11086/.

Council of Science Editors:

Newsome CT. Effects of Beta-Glucan on Sepsis and Endotoxemia. [Doctoral Dissertation]. Brown University; 2010. Available from: https://repository.library.brown.edu/studio/item/bdr:11086/

12. Nevers, Tania A. Inflammatory and dysfunctional regulatory T cells in adverse pregnancy outcomes.

Degree: PhD, Pathobiology, 2012, Brown University

 The etiologies for adverse pregnancy outcomes such as recurrent spontaneous abortion, preeclampsia, preterm birth and gestational diabetes mellitus (GDM) are multi-factorial and remain poorly understood.… (more)

Subjects/Keywords: Regulatory T cells

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APA (6th Edition):

Nevers, T. A. (2012). Inflammatory and dysfunctional regulatory T cells in adverse pregnancy outcomes. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297562/

Chicago Manual of Style (16th Edition):

Nevers, Tania A. “Inflammatory and dysfunctional regulatory T cells in adverse pregnancy outcomes.” 2012. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:297562/.

MLA Handbook (7th Edition):

Nevers, Tania A. “Inflammatory and dysfunctional regulatory T cells in adverse pregnancy outcomes.” 2012. Web. 25 Mar 2019.

Vancouver:

Nevers TA. Inflammatory and dysfunctional regulatory T cells in adverse pregnancy outcomes. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:297562/.

Council of Science Editors:

Nevers TA. Inflammatory and dysfunctional regulatory T cells in adverse pregnancy outcomes. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297562/

13. Valm, Alex Mihkel. Combinatorial Labeling and Spectral Imaging (CLASI): A novel microscopy method for the systems-level analysis of biological structure and its application to the study of human oral microbial community organization.

Degree: PhD, Pathobiology, 2012, Brown University

 Just as the phenotypically different cells that make up multicellular organisms are distributed in tissues with structures that embody specific functions, microbial cells with different… (more)

Subjects/Keywords: fluorescence imaging

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APA (6th Edition):

Valm, A. M. (2012). Combinatorial Labeling and Spectral Imaging (CLASI): A novel microscopy method for the systems-level analysis of biological structure and its application to the study of human oral microbial community organization. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297560/

Chicago Manual of Style (16th Edition):

Valm, Alex Mihkel. “Combinatorial Labeling and Spectral Imaging (CLASI): A novel microscopy method for the systems-level analysis of biological structure and its application to the study of human oral microbial community organization.” 2012. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:297560/.

MLA Handbook (7th Edition):

Valm, Alex Mihkel. “Combinatorial Labeling and Spectral Imaging (CLASI): A novel microscopy method for the systems-level analysis of biological structure and its application to the study of human oral microbial community organization.” 2012. Web. 25 Mar 2019.

Vancouver:

Valm AM. Combinatorial Labeling and Spectral Imaging (CLASI): A novel microscopy method for the systems-level analysis of biological structure and its application to the study of human oral microbial community organization. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:297560/.

Council of Science Editors:

Valm AM. Combinatorial Labeling and Spectral Imaging (CLASI): A novel microscopy method for the systems-level analysis of biological structure and its application to the study of human oral microbial community organization. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297560/

14. Jiz, Mario Antonio II L. Vaccine Development in Schistosomiasis japonica: Paramyosin As a Leading Candidate.

Degree: PhD, Division of Biology and Medicine. Pathobiology, 2009, Brown University

 Schistosomiasis is a chronic debilitating disease caused by helminths of the genus Schistosoma, and currently affecting millions in the developing world. Treatment with Praziquantel has… (more)

Subjects/Keywords: vaccine

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APA (6th Edition):

Jiz, M. A. I. L. (2009). Vaccine Development in Schistosomiasis japonica: Paramyosin As a Leading Candidate. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:175/

Chicago Manual of Style (16th Edition):

Jiz, Mario Antonio II L. “Vaccine Development in Schistosomiasis japonica: Paramyosin As a Leading Candidate.” 2009. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:175/.

MLA Handbook (7th Edition):

Jiz, Mario Antonio II L. “Vaccine Development in Schistosomiasis japonica: Paramyosin As a Leading Candidate.” 2009. Web. 25 Mar 2019.

Vancouver:

Jiz MAIL. Vaccine Development in Schistosomiasis japonica: Paramyosin As a Leading Candidate. [Internet] [Doctoral dissertation]. Brown University; 2009. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:175/.

Council of Science Editors:

Jiz MAIL. Vaccine Development in Schistosomiasis japonica: Paramyosin As a Leading Candidate. [Doctoral Dissertation]. Brown University; 2009. Available from: https://repository.library.brown.edu/studio/item/bdr:175/

15. Brown, Caitlin Westberg. Notch3 promotes anoikis resistance via expression of col4α2 in epithelial ovarian cancer.

Degree: PhD, Pathobiology, 2014, Brown University

 Epithelial ovarian cancer (EOC) is the most lethal gynecologic malignancy. The five-year survival rate of women diagnosed with stage IV high-grade serous epithelial ovarian cancer… (more)

Subjects/Keywords: epithelial ovarian cancer

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APA (6th Edition):

Brown, C. W. (2014). Notch3 promotes anoikis resistance via expression of col4α2 in epithelial ovarian cancer. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:386266/

Chicago Manual of Style (16th Edition):

Brown, Caitlin Westberg. “Notch3 promotes anoikis resistance via expression of col4α2 in epithelial ovarian cancer.” 2014. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:386266/.

MLA Handbook (7th Edition):

Brown, Caitlin Westberg. “Notch3 promotes anoikis resistance via expression of col4α2 in epithelial ovarian cancer.” 2014. Web. 25 Mar 2019.

Vancouver:

Brown CW. Notch3 promotes anoikis resistance via expression of col4α2 in epithelial ovarian cancer. [Internet] [Doctoral dissertation]. Brown University; 2014. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:386266/.

Council of Science Editors:

Brown CW. Notch3 promotes anoikis resistance via expression of col4α2 in epithelial ovarian cancer. [Doctoral Dissertation]. Brown University; 2014. Available from: https://repository.library.brown.edu/studio/item/bdr:386266/

16. Hutchins, Noelle A. The Multifaceted Roles of Fas and PD-L1 on Liver Sinusoidal Endothelial Cell Dysfunction in Sepsis.

Degree: PhD, Pathobiology, 2013, Brown University

 This dissertation focuses on the response of liver sinusoidal endothelial cells (LSECs) to experimental sepsis in order to better understand the pathologic processes contributing to… (more)

Subjects/Keywords: sepsis

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APA (6th Edition):

Hutchins, N. A. (2013). The Multifaceted Roles of Fas and PD-L1 on Liver Sinusoidal Endothelial Cell Dysfunction in Sepsis. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:320516/

Chicago Manual of Style (16th Edition):

Hutchins, Noelle A. “The Multifaceted Roles of Fas and PD-L1 on Liver Sinusoidal Endothelial Cell Dysfunction in Sepsis.” 2013. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:320516/.

MLA Handbook (7th Edition):

Hutchins, Noelle A. “The Multifaceted Roles of Fas and PD-L1 on Liver Sinusoidal Endothelial Cell Dysfunction in Sepsis.” 2013. Web. 25 Mar 2019.

Vancouver:

Hutchins NA. The Multifaceted Roles of Fas and PD-L1 on Liver Sinusoidal Endothelial Cell Dysfunction in Sepsis. [Internet] [Doctoral dissertation]. Brown University; 2013. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:320516/.

Council of Science Editors:

Hutchins NA. The Multifaceted Roles of Fas and PD-L1 on Liver Sinusoidal Endothelial Cell Dysfunction in Sepsis. [Doctoral Dissertation]. Brown University; 2013. Available from: https://repository.library.brown.edu/studio/item/bdr:320516/

17. Stout, David Andrew. Analysis of nanostructuredve vs Lipopolysaccharide (LPS) Activated Neutrophil Chemotaxis in 3D Collagen Matrices Using Traction Force Microscopy.

Degree: PhD, Biomedical Engineering, 2014, Brown University

 Understanding the fundamental mechanisms, and forces, underlying cell migration holds the promise of effective approaches for treating diseases and promoting cellular transplantation. One such disease… (more)

Subjects/Keywords: Traction Force Microscopy

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APA (6th Edition):

Stout, D. A. (2014). Analysis of nanostructuredve vs Lipopolysaccharide (LPS) Activated Neutrophil Chemotaxis in 3D Collagen Matrices Using Traction Force Microscopy. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:386196/

Chicago Manual of Style (16th Edition):

Stout, David Andrew. “Analysis of nanostructuredve vs Lipopolysaccharide (LPS) Activated Neutrophil Chemotaxis in 3D Collagen Matrices Using Traction Force Microscopy.” 2014. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:386196/.

MLA Handbook (7th Edition):

Stout, David Andrew. “Analysis of nanostructuredve vs Lipopolysaccharide (LPS) Activated Neutrophil Chemotaxis in 3D Collagen Matrices Using Traction Force Microscopy.” 2014. Web. 25 Mar 2019.

Vancouver:

Stout DA. Analysis of nanostructuredve vs Lipopolysaccharide (LPS) Activated Neutrophil Chemotaxis in 3D Collagen Matrices Using Traction Force Microscopy. [Internet] [Doctoral dissertation]. Brown University; 2014. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:386196/.

Council of Science Editors:

Stout DA. Analysis of nanostructuredve vs Lipopolysaccharide (LPS) Activated Neutrophil Chemotaxis in 3D Collagen Matrices Using Traction Force Microscopy. [Doctoral Dissertation]. Brown University; 2014. Available from: https://repository.library.brown.edu/studio/item/bdr:386196/

18. Byrd, Angel Shree'. Abstract of, “Regulation of Human Neutrophil Functions by the Integrin, CR3 – An Extracellular Matrix-Based Mechanism of Rapid Neutrophil Extracellular Trap Formation” by Angel S. Byrd, Ph.D., Brown University, May 2014.

Degree: PhD, Pathobiology, 2014, Brown University

 Neutrophil-mediated host defense against pathogens includes the extrusion of a lattice of DNA and microbicidal enzymes known as Neutrophil Extracellular Traps (NETs). The receptor:ligand interactions… (more)

Subjects/Keywords: NEONATES

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APA (6th Edition):

Byrd, A. S. (2014). Abstract of, “Regulation of Human Neutrophil Functions by the Integrin, CR3 – An Extracellular Matrix-Based Mechanism of Rapid Neutrophil Extracellular Trap Formation” by Angel S. Byrd, Ph.D., Brown University, May 2014. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:386316/

Chicago Manual of Style (16th Edition):

Byrd, Angel Shree'. “Abstract of, “Regulation of Human Neutrophil Functions by the Integrin, CR3 – An Extracellular Matrix-Based Mechanism of Rapid Neutrophil Extracellular Trap Formation” by Angel S. Byrd, Ph.D., Brown University, May 2014.” 2014. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:386316/.

MLA Handbook (7th Edition):

Byrd, Angel Shree'. “Abstract of, “Regulation of Human Neutrophil Functions by the Integrin, CR3 – An Extracellular Matrix-Based Mechanism of Rapid Neutrophil Extracellular Trap Formation” by Angel S. Byrd, Ph.D., Brown University, May 2014.” 2014. Web. 25 Mar 2019.

Vancouver:

Byrd AS. Abstract of, “Regulation of Human Neutrophil Functions by the Integrin, CR3 – An Extracellular Matrix-Based Mechanism of Rapid Neutrophil Extracellular Trap Formation” by Angel S. Byrd, Ph.D., Brown University, May 2014. [Internet] [Doctoral dissertation]. Brown University; 2014. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:386316/.

Council of Science Editors:

Byrd AS. Abstract of, “Regulation of Human Neutrophil Functions by the Integrin, CR3 – An Extracellular Matrix-Based Mechanism of Rapid Neutrophil Extracellular Trap Formation” by Angel S. Byrd, Ph.D., Brown University, May 2014. [Doctoral Dissertation]. Brown University; 2014. Available from: https://repository.library.brown.edu/studio/item/bdr:386316/

19. Toussaint, Leon E. Invariant Natural Killer T Cell Development is Uniquely Regulated by Y-Chromosome Genes and SHIP-1.

Degree: PhD, Pathobiology, 2012, Brown University

 The Src homology 2 (SH2) domain-containing inositol-5-phosphatase 1 (SHIP-1) plays a critical role in regulating the phosphoinositide 3-kinases (PI3Ks) signaling pathway. PI3Ks are important for… (more)

Subjects/Keywords: Y-linked genes

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APA (6th Edition):

Toussaint, L. E. (2012). Invariant Natural Killer T Cell Development is Uniquely Regulated by Y-Chromosome Genes and SHIP-1. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297571/

Chicago Manual of Style (16th Edition):

Toussaint, Leon E. “Invariant Natural Killer T Cell Development is Uniquely Regulated by Y-Chromosome Genes and SHIP-1.” 2012. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:297571/.

MLA Handbook (7th Edition):

Toussaint, Leon E. “Invariant Natural Killer T Cell Development is Uniquely Regulated by Y-Chromosome Genes and SHIP-1.” 2012. Web. 25 Mar 2019.

Vancouver:

Toussaint LE. Invariant Natural Killer T Cell Development is Uniquely Regulated by Y-Chromosome Genes and SHIP-1. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:297571/.

Council of Science Editors:

Toussaint LE. Invariant Natural Killer T Cell Development is Uniquely Regulated by Y-Chromosome Genes and SHIP-1. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297571/

20. Andrews, Christina. A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment.

Degree: Biomedical Engineering, 2018, Brown University

 Neutrophils are the most abundant circulating white blood cell in the human body, and play a crucial role in the innate immune response to infection… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Andrews, C. (2018). A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment. (Thesis). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:792823/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andrews, Christina. “A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment.” 2018. Thesis, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:792823/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andrews, Christina. “A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment.” 2018. Web. 25 Mar 2019.

Vancouver:

Andrews C. A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment. [Internet] [Thesis]. Brown University; 2018. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:792823/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andrews C. A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment. [Thesis]. Brown University; 2018. Available from: https://repository.library.brown.edu/studio/item/bdr:792823/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. O'Brien, Xian Marie. Innate Immune Functions of Human Polymorphonuclear Leukocytes As Mediated by the β2 Integrin, CR3, and Modulated by β1 Integrin Engagement and β-glucan, a Fungal Pathogen Associated Molecular Pattern.

Degree: PhD, Pathobiology, 2010, Brown University

 Invasive fungal infections are emerging as a significant cause of morbidity and mortality, especially among the increasing immunosuppressed patient populations. Transition to a filamentous hyphal… (more)

Subjects/Keywords: respiratory burst

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APA (6th Edition):

O'Brien, X. M. (2010). Innate Immune Functions of Human Polymorphonuclear Leukocytes As Mediated by the β2 Integrin, CR3, and Modulated by β1 Integrin Engagement and β-glucan, a Fungal Pathogen Associated Molecular Pattern. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11088/

Chicago Manual of Style (16th Edition):

O'Brien, Xian Marie. “Innate Immune Functions of Human Polymorphonuclear Leukocytes As Mediated by the β2 Integrin, CR3, and Modulated by β1 Integrin Engagement and β-glucan, a Fungal Pathogen Associated Molecular Pattern.” 2010. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:11088/.

MLA Handbook (7th Edition):

O'Brien, Xian Marie. “Innate Immune Functions of Human Polymorphonuclear Leukocytes As Mediated by the β2 Integrin, CR3, and Modulated by β1 Integrin Engagement and β-glucan, a Fungal Pathogen Associated Molecular Pattern.” 2010. Web. 25 Mar 2019.

Vancouver:

O'Brien XM. Innate Immune Functions of Human Polymorphonuclear Leukocytes As Mediated by the β2 Integrin, CR3, and Modulated by β1 Integrin Engagement and β-glucan, a Fungal Pathogen Associated Molecular Pattern. [Internet] [Doctoral dissertation]. Brown University; 2010. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:11088/.

Council of Science Editors:

O'Brien XM. Innate Immune Functions of Human Polymorphonuclear Leukocytes As Mediated by the β2 Integrin, CR3, and Modulated by β1 Integrin Engagement and β-glucan, a Fungal Pathogen Associated Molecular Pattern. [Doctoral Dissertation]. Brown University; 2010. Available from: https://repository.library.brown.edu/studio/item/bdr:11088/

22. Toorie, Anika M. The role of hypothalamic Sirt1 in the regulation of the hypophysiotropic adrenal and thyroid axes.

Degree: PhD, Pathobiology, 2015, Brown University

 Sirtuin 1 is a metabolic sensor that has a role in metabolic homeostasis in the periphery and at the central level. Its role in the… (more)

Subjects/Keywords: diet induced obesity

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APA (6th Edition):

Toorie, A. M. (2015). The role of hypothalamic Sirt1 in the regulation of the hypophysiotropic adrenal and thyroid axes. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419479/

Chicago Manual of Style (16th Edition):

Toorie, Anika M. “The role of hypothalamic Sirt1 in the regulation of the hypophysiotropic adrenal and thyroid axes.” 2015. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:419479/.

MLA Handbook (7th Edition):

Toorie, Anika M. “The role of hypothalamic Sirt1 in the regulation of the hypophysiotropic adrenal and thyroid axes.” 2015. Web. 25 Mar 2019.

Vancouver:

Toorie AM. The role of hypothalamic Sirt1 in the regulation of the hypophysiotropic adrenal and thyroid axes. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:419479/.

Council of Science Editors:

Toorie AM. The role of hypothalamic Sirt1 in the regulation of the hypophysiotropic adrenal and thyroid axes. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419479/

23. Johnson, Courtney Michele. Molecular Mechanisms Underlying The Human Neutrophil Response To Fungal Beta-Glucan In The Context Of The Extracellular Matrix Protein Fibronectin.

Degree: PhD, Pathobiology, 2015, Brown University

 Complement Receptor 3 (CR3), a β2 integrin found on neutrophils, plays an important role in fungal recognition and immune response. Co-ligation of CR3 with fibronectin… (more)

Subjects/Keywords: Complement Receptor 3

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APA (6th Edition):

Johnson, C. M. (2015). Molecular Mechanisms Underlying The Human Neutrophil Response To Fungal Beta-Glucan In The Context Of The Extracellular Matrix Protein Fibronectin. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419450/

Chicago Manual of Style (16th Edition):

Johnson, Courtney Michele. “Molecular Mechanisms Underlying The Human Neutrophil Response To Fungal Beta-Glucan In The Context Of The Extracellular Matrix Protein Fibronectin.” 2015. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:419450/.

MLA Handbook (7th Edition):

Johnson, Courtney Michele. “Molecular Mechanisms Underlying The Human Neutrophil Response To Fungal Beta-Glucan In The Context Of The Extracellular Matrix Protein Fibronectin.” 2015. Web. 25 Mar 2019.

Vancouver:

Johnson CM. Molecular Mechanisms Underlying The Human Neutrophil Response To Fungal Beta-Glucan In The Context Of The Extracellular Matrix Protein Fibronectin. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:419450/.

Council of Science Editors:

Johnson CM. Molecular Mechanisms Underlying The Human Neutrophil Response To Fungal Beta-Glucan In The Context Of The Extracellular Matrix Protein Fibronectin. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419450/

24. Crane, Meredith J. Innate Immune Events Promoting Antiviral Defense during MCMV Infection.

Degree: PhD, Pathobiology, 2012, Brown University

 The studies presented in this thesis investigated innate immune events in the liver during acute infection with murine cytomegalovirus (MCMV). While many responses that occur… (more)

Subjects/Keywords: MCMV

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APA (6th Edition):

Crane, M. J. (2012). Innate Immune Events Promoting Antiviral Defense during MCMV Infection. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297563/

Chicago Manual of Style (16th Edition):

Crane, Meredith J. “Innate Immune Events Promoting Antiviral Defense during MCMV Infection.” 2012. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:297563/.

MLA Handbook (7th Edition):

Crane, Meredith J. “Innate Immune Events Promoting Antiviral Defense during MCMV Infection.” 2012. Web. 25 Mar 2019.

Vancouver:

Crane MJ. Innate Immune Events Promoting Antiviral Defense during MCMV Infection. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:297563/.

Council of Science Editors:

Crane MJ. Innate Immune Events Promoting Antiviral Defense during MCMV Infection. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297563/

25. Ramirez, Teresa. Contributions of Insulin Resistance, Endoplasmic Reticulum Stress, and Ceramides in Alcoholic Liver Disease.

Degree: PhD, Molecular Pharmacology, Physiology, and Biotechnology, 2014, Brown University

 Background: Chronic alcoholic liver disease is associated with hepatic insulin resistance, inflammation, oxidative and ER stress, mitochondrial dysfunction, and DNA damage. Peroxisome proliferator activated receptor… (more)

Subjects/Keywords: Alcoholic liver disease; Experimental model; PPAR agonists; Steato-hepatitis; Insulin Resistance; Histopathology; Electron microscopy; ER stress

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APA (6th Edition):

Ramirez, T. (2014). Contributions of Insulin Resistance, Endoplasmic Reticulum Stress, and Ceramides in Alcoholic Liver Disease. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:386183/

Chicago Manual of Style (16th Edition):

Ramirez, Teresa. “Contributions of Insulin Resistance, Endoplasmic Reticulum Stress, and Ceramides in Alcoholic Liver Disease.” 2014. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:386183/.

MLA Handbook (7th Edition):

Ramirez, Teresa. “Contributions of Insulin Resistance, Endoplasmic Reticulum Stress, and Ceramides in Alcoholic Liver Disease.” 2014. Web. 25 Mar 2019.

Vancouver:

Ramirez T. Contributions of Insulin Resistance, Endoplasmic Reticulum Stress, and Ceramides in Alcoholic Liver Disease. [Internet] [Doctoral dissertation]. Brown University; 2014. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:386183/.

Council of Science Editors:

Ramirez T. Contributions of Insulin Resistance, Endoplasmic Reticulum Stress, and Ceramides in Alcoholic Liver Disease. [Doctoral Dissertation]. Brown University; 2014. Available from: https://repository.library.brown.edu/studio/item/bdr:386183/

26. Longato, Lisa. Pathogenic mechanisms and consequences of steatohepatitis.

Degree: PhD, Pathobiology, 2012, Brown University

 The mechanisms by which steatohepatitis in alcoholic (ALD) or non-alcoholic (NAFLD) fatty liver disease becomes progressive and advances to stages of fibrosis, cirrhosis, and liver… (more)

Subjects/Keywords: NAFLD

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APA (6th Edition):

Longato, L. (2012). Pathogenic mechanisms and consequences of steatohepatitis. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297566/

Chicago Manual of Style (16th Edition):

Longato, Lisa. “Pathogenic mechanisms and consequences of steatohepatitis.” 2012. Doctoral Dissertation, Brown University. Accessed March 25, 2019. https://repository.library.brown.edu/studio/item/bdr:297566/.

MLA Handbook (7th Edition):

Longato, Lisa. “Pathogenic mechanisms and consequences of steatohepatitis.” 2012. Web. 25 Mar 2019.

Vancouver:

Longato L. Pathogenic mechanisms and consequences of steatohepatitis. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Mar 25]. Available from: https://repository.library.brown.edu/studio/item/bdr:297566/.

Council of Science Editors:

Longato L. Pathogenic mechanisms and consequences of steatohepatitis. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297566/

.